1/6. amelogenesis imperfecta: diagnosis and resolution of a case with hypoplasia and hypocalcification of enamel, dental agenesis, and skeletal open bite.A case of amelogenesis imperfecta with hypoplasia, hypocalcification of the enamel, congenital absence of teeth 12 and 22, delayed eruption of tooth 23, edge-to-edge incisal relationship, open bite, and bilateral posterior cross bite at the level of the first and second premolars is presented. Lateral skull teleradiography indicated a Class III skeletal pattern of maxillary origin associated with a dolichofacial pattern with multiple indicators of facial hyperdivergence. The patient presented a major esthetic abnormality of the face and required orthodontic treatment prior to a prosthetic solution with full-coverage metal-ceramic crowns in both the maxilla and the mandible. The diagnosis of cases such as this one and the therapeutic implications from an orthodontic and prosthetic standpoint are reported.- - - - - - - - - - ranking = 1keywords = open bite, bite, open (Clic here for more details about this article) |
2/6. Improving quality of life with a team approach: a case report.An adolescent female who presented amelogenesis imperfecta with severe anterior open bite, long face, facial asymmetry, high angle, and Class III skeletal pattern was treated with an interdisciplinary (orthodontics, orthognathic surgery, and prosthodontics) treatment approach. Presurgical orthodontic treatment was followed by surgical maxillary posterior impaction with anterior advancement and mandibular setback operation with vertical chin reduction and genioplasty. After the surgery, anterior ceramic laminate veneers and posterior full ceramic onlay-crowns were performed. The results showed that function and esthetics were achieved successfully with interdisciplinary collaboration.- - - - - - - - - - ranking = 0.19978800030692keywords = open bite, bite, open (Clic here for more details about this article) |
3/6. amelogenesis imperfecta: the multidisciplinary approach. A case report.amelogenesis imperfecta is a hereditary developmental disorder of the dental enamel, in both primary and permanent dentition. The main clinical characteristics are extensive loss of tooth tissue, poor esthetics, and tooth sensitivity. Transmission of the gene takes place by either autosomal, dominant X-linked, or recessive modes. This clinical report describes a treatment sequence based on a multidisciplinary approach. A 21-year-old girl with hypoplastic amelogenesis imperfecta was referred to the Ege University School of dentistry clinic. She was concerned about the poor appearance and sensitivity of her teeth. The patient presented with an anterior open bite, although orthodontic treatment had been completed previously. Periodontal gingivectomy of her posterior teeth followed by endodontic treatment where indicated was proposed. The prosthodontic treatment consisted of metal ceramic fixed partial dentures of precious alloy. At the end of treatment, function and esthetics were improved to a level acceptable to both the patient and the dental team.- - - - - - - - - - ranking = 0.19978800030692keywords = open bite, bite, open (Clic here for more details about this article) |
4/6. A case of amelogenesis imperfecta, cleft lip and palate and polycystic kidney disease.OBJECTIVE: amelogenesis imperfecta (AI) is a heterogeneous group of genetic disorders characterized by developmental abnormalities of tooth enamel. The AI is also seen as part of multi-organ abnormalities, e.g. with cone-rod dystrophy, hypothalamo-hypophyseal insufficiency and renal failure. The present patient with AI and nephrocalcinosis exhibited a phenotype different from previous cases with renal failure. To highlight the characteristics of this rare case, extensive analysis that included histological, biochemical and genetic examinations was performed. PATIENT: The present Japanese male patient exhibited dentition with AI and bilateral cleft lip and palate. Ground sections of his extracted tooth showed that it was hypomaturation-type AI, unlike previous cases with nephrocalcinosis were hypoplastic-type. He showed nephrocalcinosis and hematuria at 15 years of age but these symptoms appeared to be secondary to polycystic kidney disease. He showed skeletal Class II pattern with a retrognathic profile and retroclined incisors of both arches. A dolicofacial appearance was seen with an enlarged gonial angle. Biochemical makers including serum alkaline phosphatase, parathyroid hormone, calcitonin, calcium, and phosphate, were all in the normal range. sequence analysis of the genes encoding amelogenin and enamelin, which are known to be responsible for hypoplastic-type AI, did not reveal any mutations. Since mouse null mutant of homeobox transcription factor, Msx2, exhibits a phenotype resembling AI, the human homolog of this gene, MSX2, was sequenced. There was a missense mutation of T447C that resulted in the conversion of methionine to threonine at 129.- - - - - - - - - - ranking = 0.0021199969307888keywords = bite (Clic here for more details about this article) |
5/6. Current treatment modalities in the conservative restoration of amelogenesis imperfecta: a case report.amelogenesis imperfecta is an inherited disorder involving enamel formation that affects the appearance of the teeth to various degrees. Cosmetic rehabilitation of these patients has been open to a variety of treatment options. Complete-coverage restorations are often recommended. patients are occasionally averse to this because of the extensive preparation of tooth structure or lack of financial means. In the past, conservative measures were inadequate because the lack of enamel bonding did not allow a durable restoration. Presently, with the use of glass-ionomer cements and dentinal adhesives, dentin-resin bond strengths are approaching those of etched enamel. This article discusses the use of glass-ionomer cement, dentinal priming agents, and etched enamel to create a strong bond between restorative material and tooth structure that provides not only satisfactory esthetics, but also a durable restoration.- - - - - - - - - - ranking = 8.2190767988075E-5keywords = open (Clic here for more details about this article) |
6/6. Analysis of a kindred with amelogenesis imperfecta.amelogenesis imperfecta (AI) is a group of hereditary disorders whose manifestations are generally considered as being confined to the teeth. pedigree analysis of a kindred revealed 2 siblings affected with AI which was consistent with an autosomal recessive mode of inheritance. Cephalometric evaluation showed both children to have an obtuse gonial angle and steep mandibular plane. These skeletal changes were manifested as a severe anterior open bite. Histological evaluation of the teeth demonstrated numerous enamel changes including altered prism morphology, prism coalescence and disruption, globular inclusions, and irregular crystallite orientation. The clinical and histological data are consistent with autosomal recessive pigmented hypomaturation amelogenesis imperfecta. The enamel defects appear to be caused by the combination of decreased mineral deposition with abnormal crystallite and prism formation. Some areas of enamel hypoplasia seem to exist as well.- - - - - - - - - - ranking = 0.19978800030692keywords = open bite, bite, open (Clic here for more details about this article) |