Cases reported "Bronchopneumonia"

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1/6. Fulminant tuberculous bronchopneumonia in a young hong kong Chinese woman.

    A 24-yr-old, previously healthy Chinese woman presented with symptoms of acute bronchopneumonia which led to acute respiratory failure and death 6 days after admission to hospital despite intensive antibiotic treatment. autopsy showed acute tuberculous bronchopneumonia of the acinar type. This is a rare presentation and outcome of tuberculous infection in hong kong, where 124 cases per 100,000 population were notified in 1988, and indicates that tuberculosis can mimic acute bronchopneumonia and should be considered in cases so presenting, particularly when there is no growth on routine culture and no response to conventional antibiotics. Acute respiratory failure developing in such cases can cause a delay in the diagnosis of tuberculosis.
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ranking = 1
keywords = tuberculosis
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2/6. Chronic bronchopneumonia with recurrent hemoptyses and resultant severe anemia.

    A female patient, 69 years old, was hospitalized because of a 2-year history of recurrent hemoptyses resulting in severe anemia. X-ray examination of the chest showed a mass lesion in the right lower lung field, which had grown over the preceding 2 years. Bronchographic, arteriographic and CT examinations excluded the possibilities of bronchiectasis, pulmonary A-V fistula or sequestration. Histological examination following right lower lobectomy revealed no evidence of neoplasms or tuberculosis, fungal and parasitic infections but showed a predominant mononuclear cell infiltration and abundant small vessels in the affected small bronchi, and peribronchiolar and adjacent alveolar regions. To our knowledge, no case with chronic bronchopneumonia accompanied by such massive hemoptyses, as seen in this case, has been reported to date.
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ranking = 0.5
keywords = tuberculosis
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3/6. Branhamella catarrhalis as a cause of pneumonia in a patient with miliary tuberculosis.

    Branhamella catarrhalis is increasingly reported as a cause of pneumonia in the immunocompromised host. The authors here report what they believe to be a unique case of B catarrhalis bronchopneumonia in a patient who had previously acquired miliary tuberculosis. The patient initially responded to medication but died suddenly following a brief episode of febrile illness. At autopsy, several lines of evidence implicated B catarrhalis in the findings. The authors review the literature regarding cases of lower respiratory tract infection reportedly caused by B catarrhalis. Their own conclusion is that B catarrhalis infection is not necessarily caused by abnormal immunoglobulin, as some workers have suggested, but rather by damaged lung tissue in general and damaged bronchoalveolar cells in particular.
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ranking = 2.5
keywords = tuberculosis
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4/6. Neonatal tuberculosis.

    tuberculosis rarely presents in the neonatal period. Though treatable, it may be fatal despite modern treatment. The diagnosis of congenital tuberculosis should be considered in any neonate with pneumonia that fails to respond to conventional treatment, particularly in a child from an ethnic or socioeconomic environment where tuberculosis is prevalent.
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ranking = 3
keywords = tuberculosis
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5/6. Neonatal tuberculosis: a case report.

    A 3-week-old baby with neonatally acquired tuberculous bronchopneumonia is presented. The diagnosis was considered because the neonate did not respond to conventional management of bronchopneumonia. The importance of including tuberculosis as a differential diagnosis in respiratory disorders, especially in developing countries, is emphasized.
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ranking = 2.5
keywords = tuberculosis
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6/6. hartmannella vermiformis isolated from the cerebrospinal fluid of a young male patient with meningoencephalitis and bronchopneumonia.

    meningoencephalitis and bronchopneumonia were documented in a patient from Peubla, mexico. The patient began with symptoms and signs of a common flu and 12 days after the onset of his disease he was admitted to the hospital presenting symptoms and signs of meningoencephalitis. The clinical course evolved into an endocraneal hypertension syndrome with bronchopneumonia, coma and death. Wide-spectrum antibiotics, immunosuppressive and anti-tuberculosis therapy were unsuccessfully administered. Important antecedents were degree I malnutrition and repeated contact with polluted water. Post-mortem autopsy was not performed. gram-positive cocci were isolated from the spinal fluid 2 days after admission, and then active amebae were isolated from three different samples of the spinal fluid at days 16, 18 and 19 after admission. Such samples were concentrated and inoculated onto specific culture media. Identification of amebae was based on their morphology and biochemistry. All amebae were hartmannella vermiformis. Amebae were apparently not the cause of the disease and might be considered as an opportunistic colonizer which may have caused the evolution of the disease to become worse.
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ranking = 0.5
keywords = tuberculosis
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