1/86. Cramping pain and prolonged elevation of serum creatine kinase levels in a patient with guillain-barre syndrome following campylobacter jejuni enteritis.We describe a patient with guillain-barre syndrome (GBS) following Campylobacter jejuni enteritis, accompanied with severe cramping pain and a marked increase in serum creatine kinase (CK) levels. Both conditions became evident three weeks after the onset of GBS and continued for longer than one month. In this patient, it is possible that rapid extensive denervation due to severe axonal degeneration of motor nerve terminals might have caused hyperexcitability in regional muscles, leading to recurrent muscle cramps and persistent release of muscular CK.- - - - - - - - - - ranking = 1keywords = enteritis (Clic here for more details about this article) |
2/86. A case of guillain-barre syndrome following a family outbreak of campylobacter jejuni enteritis.We describe an outbreak of campylobacter jejuni enteritis involving three family members of whom one developed guillain-barre syndrome (GBS). The patients' serum reacted strongly with several gangliosides and with the lipopolysaccharide (LPS) fractions from the C. jejuni strains isolated from his family members. Only low titer anti-ganglioside antibodies were found in his siblings. HLA-typing did not indicate a locus associated with auto-antibody production. Comparing the immune response in GBS patients and C. jejuni enteritis patients can be of great value in determining the additional factors that lead to post-Campylobacter GBS. Ganglioside mimicry alone is necessary but not sufficient for the induction of anti-ganglioside antibodies. Other susceptibility factors are required to induce an anti-neural immune response.- - - - - - - - - - ranking = 1.2keywords = enteritis (Clic here for more details about this article) |
3/86. campylobacter jejuni enteritis.We report the development of campylobacter jejuni enteritis in a patient with preexisting humoral and cellular immune recognition of C. jejuni antigens. This is one of few studies in which the immunologic status of a person with regard to C. jejuni before and after C. jejuni infection is directly compared, and it is the only study of which we are aware that includes measurements of cellular immunity. The findings may be important to Campylobacter vaccine development efforts.- - - - - - - - - - ranking = 1keywords = enteritis (Clic here for more details about this article) |
4/86. myocarditis in a patient with salmonella and campylobacter enteritis.myocarditis associated with bacterial enteritis has only rarely been described and the pathogenesis is unclear. Herein we report a case where a young adult developed myocarditis during the acute stage of an infection with salmonella heidelberg and Campylobacter jejunii/coli. The patient's troponin i value was elevated. We suggest that use of cardiac-sensitive troponins may be a useful tool for diagnosis of acute myocarditis in the context of bacterial enteritis. We also suggest the need for further investigation of the pathogenesis of myocarditis associated with enteric pathogens.- - - - - - - - - - ranking = 1.2keywords = enteritis (Clic here for more details about this article) |
5/86. Emergence of cotrimoxazole- and quinolone-resistant campylobacter infections in bone marrow transplant recipients.Clinical and microbiological data were collected prospectively from 704 patients who underwent bone marrow transplantation (BMT) during an 11-year period (1991-2001), and the first two cases of Campylobacter infection occurring in BMT recipients in the pre-engraftment period were identified. The two cases occurred on days 2 and 3 post-BMT, respectively. Both patients had campylobacter jejuni enteritis, and one case was complicated by bacteraemia. In both cases the presenting symptoms were indistinguishable from hospital-acquired pre-engraftment diarrhoea, which is commonly caused by clostridium difficile. Both of the campylobacter jejuni isolates were resistant to cotrimoxazole and ciprofloxacin. Both patients responded to intravenous meropenem and subsequently had uneventful marrow engraftment.- - - - - - - - - - ranking = 0.2keywords = enteritis (Clic here for more details about this article) |
6/86. Hyperreflexia in axonal guillain-barre syndrome subsequent to campylobacter jejuni enteritis.We describe a patient with the acute motor axonal neuropathy (AMAN) form of guillain-barre syndrome (GBS), who showed generalized hyperreflexia. A 24-year-old man developed acute paralysis following campylobacter jejuni enteritis. He showed exaggerated tendon reflexes with abnormal reflex spread to other segments, and was initially diagnosed as having post-infectious myelitis. Nerve conduction studies showed motor axonal degeneration (the AMAN pattern), and increased soleus h-reflex amplitudes. His serum was positive for IgG antibodies to gangliosides GM1b and GalNAc-GD1a. He was treated with plasmapheresis, resulting in rapid recovery. Hyperreflexia was still present 12 months after onset when muscle strength was completely normal. This case provides further evidence that patients with AMAN can develop increased motor neuron excitability, and possible mechanisms for the hyperreflexia are discussed.- - - - - - - - - - ranking = 1keywords = enteritis (Clic here for more details about this article) |
7/86. Transmission of campylobacter hyointestinalis from a pig to a human.We report on a case of human gastroenteritis caused by the pathogen campylobacter hyointestinalis. Recurrent watery diarrhea and intermittent vomiting were the most significant symptoms of the previously healthy patient. Whole-cell protein electrophoresis and 16S rRNA gene sequencing were used to identify this Campylobacter species. Investigation of the patient's surroundings led to the recovery of a second C. hyointestinalis strain originating from porcine feces. Subsequent typing of the human and the porcine isolates by pulsed-field gel electrophoresis revealed similar macrorestriction profiles, indicating transmission of this pathogen.- - - - - - - - - - ranking = 0.2keywords = enteritis (Clic here for more details about this article) |
8/86. Acute isolated bulbar palsy with anti-GT1a IgG antibody subsequent to campylobacter jejuni enteritis.We describe a patient with acute isolated bulbar palsy following enteritis. A 29-year-old man developed dysphagia and nasal voice without limb weakness, ataxia, or areflexia. High titres of serum anti-GT1a and anti-campylobacter jejuni IgG antibodies were detected. He was treated with plasmapheresis, resulting in rapid clinical improvement. This case suggests that an acute isolated bulbar palsy may be caused by a pathology relating to guillain-barre syndrome (GBS), in which anti-GT1a IgG antibody may have a role.- - - - - - - - - - ranking = 1keywords = enteritis (Clic here for more details about this article) |
9/86. Fulminant guillain-barre syndrome after campylobacter jejuni enteritis and monospecific anti-GT1a IgG antibody.A 21-year-old man developed rapid progression of tetraplegia, bulbar palsy, and respiratory paralysis after Campylobacterjejuni enteritis. Based on the diagnosis of guillain-barre syndrome, he received plasmapheresis and intravenous immunoglobulin. serum anti-GT1a IgG antibody which lacked cross-reactivity with GQ1b was detected. Four months after the onset, the patient still had severe muscle weakness of the lower limbs. This case suggests that anti-GT1a IgG antibody can be associated with severe paralysis in guillain-barre syndrome after C. jejuni enteritis.- - - - - - - - - - ranking = 1.2keywords = enteritis (Clic here for more details about this article) |
10/86. myocarditis related to campylobacter jejuni infection: a case report.BACKGROUND: myocarditis can develop as a complication of various infections and is most commonly linked to enterovirus infections. myocarditis is rarely associated with bacterial infections; salmonellosis and shigellosis have been the most frequently reported bacterial cause. We report a case of myocarditis related to campylobacter jejuni enteritis. CASE PRESENTATION: A 30-year-old previously healthy man presented with a history of prolonged chest pain radiating to the jaw and the left arm. Five days prior to the onset of chest pain, he developed bloody diarrhea, fever and chills. creatine kinase (CK) and CK-MB were elevated to 289 U/L and 28.7 microg/L. troponin i was 30.2 microg/L. The electrocardiogram (ECG) showed T wave inversion in the lateral and inferior leads. The chest pain resolved within 24 hours of admission. The patient had a completely normal ECG stress test. The patient was initiated on ciprofloxacin 500 mg po bid when campylobacter jejuni was isolated from the stool. diarrhea resolved within 48 hours of initiation of ciprofloxacin. The diagnosis of Campylobacter enteritis and related myocarditis was made based on the clinical and laboratory results and the patient was discharged from the hospital in stable condition. CONCLUSION: myocarditis can be a rare but severe complication of infectious disease and should be considered as a diagnosis in patients presenting with chest pain and elevated cardiac enzymes in the absence of underlying coronary disease. It can lead to cardiomyopathy and congestive heart failure. There are only a few reported cases of myocarditis associated with Campylobacter infection.- - - - - - - - - - ranking = 0.4keywords = enteritis (Clic here for more details about this article) |
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