Cases reported "Cholera"

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1/20. vibrio cholerae non-o1 facial cellulitis in a North queensland, Australian child.

    Vibrio cholerae is an uncommon cause of cellulitis in australia. Most reported cases worldwide have involved marine or brackish water contact. A recognized risk factor for acquiring this infection is chronic liver disease secondary to hepatitis b. We describe a case of extensive facial cellulitis caused by vibrio cholerae non-o1, non-0139, in an 11-year-old indigenous girl from North queensland, australia, who was hepatitis b surface antigen-negative. Treatment consisted of extensive debridement, antibiotics, hyperbaric oxygen and facial reconstructive surgery. Early microbiologic diagnosis and a combined therapeutic approach are important in the management of this condition.
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2/20. Vibrio cholerae diarrhoea in a three-day-old breastfed neonate.

    A confirmed case of cholera in a 3-day-old neonate is being reported. Possible source of infection could be by holy water (Chamamrit) given to the baby, which is common ritual in india. Mother's milk has never been reported to transmit cholera, even though mother herself may be suffering from cholera. Contaminated water can transmit the disease as there is no maternally transmitted immunity.
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3/20. Acute secretory travellers' diarrhoea caused by Vibrio cholerae non-01 which does not produce cholera-like or heat-stable enterotoxins.

    An Australian tourist suffering from severe acute watery diarrhoea and dehydration due to Vibrio cholerae non-01 was studied. The V. cholerae strain isolated from the patient belonged to serovar 05. The organism did not produce any of the conventional enterotoxins including cholera-toxin (CT) or heat-stable toxins (NAG-ST) that are known to be associated with intestinal secretion. This report suggests that toxin(s) other than CT-like or NAG-ST may be involved in the pathogenesis of diarrhoea by some V. cholerae non-01 strains.
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4/20. Isolation of vibriostatic agent O/129-resistant vibrio cholerae non-o1 from a patient with gastroenteritis.

    A strain of vibrio cholerae non-o1 was isolated from a 48-year-old male in california who presented with a chief complaint of watery diarrhea. Laboratory investigations of this strain revealed the bacterium to be resistant to trimethoprim-sulfamethoxazole and to the vibriostatic agent O/129 (2,4-diamino-6,7-diisopropylpteridine).
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5/20. Territorial waters of the Baltic Sea as a source of infections caused by vibrio cholerae non-o1, non-O139: report of 3 hospitalized cases.

    A fatal infection with temporal relation to 2 other febrile infections caused by vibrio cholerae non-o1, non-O139 (NCV) occurred in finland in 2003. All infections were associated with contact with seawater. The patient who died had also eaten home-salted whitefish, tested positive for NCV, preceding his symptoms. All patients had compromising factors, and all strains were distinguishable by pulsed-field gel electrophoresis and negative for the ctx gene. These 3 cases illustrate that, despite being uncommon in finland, NCVs can cause clinically significant and even fatal infections.
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6/20. Pulmonary cholera due to infection with a non-O1 Vibrio cholerae strain.

    We present 2 cases of primary pulmonary non-O1 Vibrio cholerae infection. We believe that these are the first documented cases of primary pulmonary infection due to this organism from a freshwater source.
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7/20. Cholera on the louisiana Gulf Coast: historical notes and case report.

    A 67-year old woman was brought by ambulance to the hospital because of syncope and collapse. Forty-eight hours prior to the admission she ate some crabs with her husband. The morning of admission she awakened with massive, watery diarrhea followed by vomiting and shock. She was admitted to the intensive care unit with hypotension and bradycardia. She was resuscitated after a large volume of fluid was administered. Approximately 22 liters of fluids were administered in 24 hours. Stool cultures grew out Vibrio cholerae 01 biotype El Tor, serotype Inaba. She was treated with intravenous doxycycline. Her recovery was uneventful. The patient's husband had mild diarrhea, and Vibrio cholerae 01 biotype El Tor, serotype Inaba was cultured from his stool. Vibrio cholerae is endemic on the louisiana Gulf Coast. The first cholera epidemic occurred in 1832. After a hiatus of about 100 years, cholera reappeared in louisiana in 1978.
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8/20. Cholera in louisiana. Widening spectrum of seafood vehicles.

    The largest cholera outbreak in the united states in over a century occurred in louisiana from August through October 1986. Eighteen persons in 12 family clusters had stool culture or serologic evidence of infection with toxigenic Vibrio cholerae 0-group 1. Thirteen of these persons had severe diarrhea, and 4 required intensive care unit treatment. Although all 18 survived, 1 96-year-old woman with suspected cholera died shortly after hospital admission. A case-control study showed that case-patients were more likely than neighborhood control subjects to have eaten cooked crabs or cooked or raw shrimp during the week before illness. Case-patients who ate crabs were more likely than control subjects who ate crabs to have undercooked and mishandled the crabs after cooking. A third vehicle from the Gulf waters, raw oysters, caused V cholerae 01 infection in two persons residing in florida and georgia. All three seafood vehicles came from multiple sources. Stool isolates from the louisiana case-patients were genetically identical to other North American strains isolated since 1973, but differ from African and Asian isolates. While crabs are the most important vehicle for V cholerae 01 infection in the united states, shrimp and oysters from the Gulf coast can also be vehicles of transmission. A persisting reservoir of V cholerae 01 along the Gulf coast may continue to cause sporadic cases and outbreaks of cholera in Gulf states and in states importing Gulf seafood.
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9/20. Cholera after the consumption of raw oysters. A case report.

    In August 1986, a 76-year-old woman in Miami, florida, developed profuse watery diarrhea and abdominal cramps. Two and four days before the onset of her illness, she had eaten six raw oysters at each of two restaurants in Miami. A stool specimen yielded toxigenic vibrio cholerae o1 biotype El Tor, serotype Inaba. The results of toxin gene probing of the organism recovered from the patient differed significantly from those of other V. cholerae O1 isolates from the Gulf Coast and elsewhere in the world. A program of active surveillance identified no other cases of cholera in Miami. The source of the raw oysters eaten by the patient was traced to louisiana. Her case represents the first reported case of cholera associated with eating raw oysters.
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10/20. Vibrio cholerae non-01 cellulitis.

    A 46-year-old man presented with pain and fever and a postphlebitic ulcer on his left leg. The wound was suppurative and open at the margins, but there was little underlying fasciitis and no apparent muscle or blood vessel involvement. Three separate wound cultures were obtained at two-day intervals, and all showed only Vibrio cholerae non-01. The patient was successfully treated with cefazolin sodium. This marks the second documented case of V cholerae non-01 type alone as a causative agent of cellulitis, and the first case where no saltwater origin could be demonstrated.
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