1/68. hypertensive encephalopathy in a patient with neonatal thyrotoxicosis.Neonatal hyperthyroidism may give rise to serious cardiovascular complications. A girl with severe thyrotoxicosis in whom hypertensive encephalopathy developed is described. CONCLUSION: Neonatal thyrotoxicosis can give rise to hypertension and may lead to hypertensive encephalopathy.- - - - - - - - - - ranking = 1keywords = encephalopathy (Clic here for more details about this article) |
2/68. hypertensive encephalopathy in a patient with retroperitoneal fibrosis.A patient presented with retroperitoneal fibrosis but without any ureteric obstruction. The diagnosis was made by an abdominal CT scan and also at laparotomy. Post-operatively, she developed hypertensive encephalopathy. An isotope renogram with captopril was abnormal but not diagnostic of renal artery stenosis. The patient's condition improved with steroid and antihypertensive treatment. A follow-up CT scan showed complete resolution of peri-aortic thickening. A causative link is postulated between retroperitoneal fibrosis, trauma during laparotomy, and onset of acute hypertension.- - - - - - - - - - ranking = 0.83333333333333keywords = encephalopathy (Clic here for more details about this article) |
3/68. hypertensive encephalopathy: complication in children treated for myeloproliferative disorders--report of three cases.We routinely perform echo-planar diffusion-weighted sequences in all brain magnetic resonance (MR) imaging studies. When three children undergoing chemotherapy for acute leukemia presented with seizures, conventional MR images demonstrated what appeared to be acute, posterior, parasagittal infarcts. However, diffusion-weighted images were normal. These MR imaging findings were consistent with those of hypertensive encephalopathy. Early recognition and treatment of minimal hypertension in these patients allows reversal of encephalopathy.- - - - - - - - - - ranking = 1keywords = encephalopathy (Clic here for more details about this article) |
4/68. Functional recovery despite prolonged bilateral loss of somatosensory evoked potentials: report on two patients.A bilateral loss of short latency somatosensory evoked potentials (SSEPs) after head trauma or non-traumatic brain damage is normally associated with a deleterious neurological outcome. An adequate recovery in reported in two deeply comatose patients with head trauma or severe hypertensive encephalopathy despite prolonged bilateral loss of SSEPs over days, found in repeated recordings. Hence, a bilateral loss of SSEPs should not be considered alone for prediction of outcome in cerebral injury.- - - - - - - - - - ranking = 0.16705980708513keywords = encephalopathy, neurologic (Clic here for more details about this article) |
5/68. Low doses of liquorice can induce hypertension encephalopathy.Prolonged ingestion of liquorice is a well-known cause of hypertension due to hypermineralocorticoidism. We describe 2 cases of hypertension encephalopathy (in addition to the classical symptoms of hypertension, hypokalemia and suppression of the renin-aldosterone system) which resulted in pseudohyperaldosteronism syndrome due to the regular daily intake of low doses of liquorice. Glycyrrhizic acid, a component of liquorice, produces both hypermineralocorticism and the onset of encephalopathy through the inhibition of 11beta-hydroxysteroid dehydrogenase. hypertension encephalopathy due to the daily intake of low doses of liquorice, however, has not been previously documented. It is proposed that some people could be susceptible to low doses of glycyrrhizic acid because of a 11beta-hydroxysteroid dehydrogenase deficiency.- - - - - - - - - - ranking = 1.1666666666667keywords = encephalopathy (Clic here for more details about this article) |
6/68. The hyponatraemic hypertensive syndrome in a 2-year-old child with behavioural symptoms.In this case report we present a 2-year-old girl with the classical signs of the hyponatraemic hypertensive syndrome. She initially presented with a history of behavioural abnormalities and hyponatraemia (126 mmol/l) and her blood pressure was as high as 220/160 mmHg. After admission, somnolence developed. Intravenous anti-hypertensive therapy was started immediately. The hyponatraemia was treated with i.v. sodium supplementation. The cause of this syndrome proved to be fibromuscular dysplasia of the left renal artery. Finally, a left nephrectomy was performed. With this therapy, blood pressure and serum sodium normalised and the girl promptly regained normal consciousness and behaviour. CONCLUSION: Behavioural abnormalities in the history of a child without any other neurological symptoms might be one of the first signs of hypertensive encephalopathy. In combination with hyponatraemia, these symptoms should alert the physician to consider the hyponatraemic hypertensive syndrome.- - - - - - - - - - ranking = 0.16705980708513keywords = encephalopathy, neurologic (Clic here for more details about this article) |
7/68. hepatitis b-associated polyarteritis nodosa and hypertensive encephalopathy.The case of a two-year-old boy with hypertensive encephalopathy secondary to hepatitis b-associated polyarteritis nodosa is presented. polyarteritis nodosa is a rare condition in children and its association with hepatitis b viral infection is also rare.- - - - - - - - - - ranking = 0.83333333333333keywords = encephalopathy (Clic here for more details about this article) |
8/68. hypertensive encephalopathy presenting with thunderclap headache.A 68-year-old woman presented with thunderclap headache, which led to a search for subarachnoid hemorrhage. Both computerized tomography of the head and cerebrospinal fluid examination were normal. magnetic resonance imaging revealed abnormalities in the white matter in the parieto-occipital regions. There was no aneurysm on magnetic resonance angiography. Treatment of hypertension led to resolution of the posterior leukoencephalopathy. hypertensive encephalopathy with reversible posterior leukoencephalopathy can present as a thunderclap headache.- - - - - - - - - - ranking = 1.1666666666667keywords = encephalopathy (Clic here for more details about this article) |
9/68. Reversible posterior leukoencephalopathy syndrome in a patient with hypertensive encephalopathy--case report.A 58-year-old male presented with reversible posterior leukoencephalopathy syndrome (RPLS) manifesting as headache, papilledema, and renal hypertension. T2-weighted magnetic resonance (MR) imaging showed hyperintensity lesions in the medulla, pons, bilateral thalami, and bilateral deep white matter of the parieto-occipital lobes. The pons was swollen. diffusion-weighted MR imaging did not show increased intensity in these lesions. The lesions disappeared with improvement of clinical symptoms after treatment for hypertension. These findings suggested the lesions were vasogenic edema and the diagnosis was RPLS. T2-weighted and diffusion-weighted MR imaging are useful modalities to differentiate RPLS from other central nervous system abnormalities such as infarction, multiple sclerosis, and central pontine myelinolysis. The clinical and neuroradiological findings of RPLS can be reversed by timely initiation of treatment for the causative factor.- - - - - - - - - - ranking = 1.5keywords = encephalopathy (Clic here for more details about this article) |
10/68. Reversible posterior leukoencephalopathy syndrome: a report of 2 cases.Reversible posterior leukoencephalopathy syndrome (RPLE) is an increasingly recognised disorder, most commonly associated with malignant hypertension, toxaemia of pregnancy or the use of immunosuppressive agents. Two cases of RPLE syndrome occurring in the setting of accelerated hypertension and eclampsia are described. Both patients had seizures, altered sensorium and typical findings on neuroimaging. They had complete clinical and radiological recovery. The clinical course, pathophysiology and neuroimaging features of RPLE syndrome are discussed.- - - - - - - - - - ranking = 0.83333333333333keywords = encephalopathy (Clic here for more details about this article) |
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