Cases reported "Hypertrophy"

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1/5. Recurrent left mandibular enlargement.

    A 35-year old African-American female presented with a painful enlargement of the left face in the area of the posterior mandible and ramus extending to the submandibular area. She also complained of some difficulty swallowing along with mobility and supereruption of the mandibular left first molar tooth. See if you can make the diagnosis.
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2/5. natal teeth: a potential impediment to nasoalveolar molding in infants with cleft lip and palate.

    The purpose of this article is to describe two cases of bilateral cleft lip and palate with natal/neonatal teeth. Clinical features, prevalence, possible complications, and treatment modalities are discussed. Two patients with cleft lip and palate and natal/neonatal teeth are described. The first patient was a 4-week-old infant with bilateral cleft lip and palate. On initial inspection an odontogenic vestige was noticed on the right lateral border of the premaxillary segment, bordering the cleft. The second patient was a newborn with a vestige on the right side of the premaxilla. At 20 days, another swelling representing a neonatal tooth on the left side was found. Because the teeth interfered with the fabrication and application of the nasoalveolar molding (NAM) appliance, they were removed from both patients. In the first patient, at 1 week after extraction, the NAM device was placed without difficulty. At 8 months, the infant had adapted well to the NAM device and nursed without problems. The second patient did not follow-up for the placement of the NAM device. In patients with cleft lip and palate with natal/neonatal teeth who require NAM, the tooth must be removed to facilitate the fabrication and placement of the device. Natal/neonatal teeth must be extracted with caution because the tooth buds of neighboring teeth may be damaged and remnants of the dental papillae may be left behind. Although general anesthesia is not always indicated for the removal of these teeth, in cases in which the premaxilla is loose, such as the current cases, general anesthesia is warranted.
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3/5. Primary failure of eruption.

    Failure of eruption may be due to mechanical interference or to a failure in the eruption mechanism of the tooth. The etiology of the primary failure of eruption is unknown, but it could be due to an alteration in either metabolism or blood flow to the periodontal ligament. Therapy of patients with this condition is extremely difficult; orthodontic treatment causes ankylosis and probably the only way to move unerupted teeth into occlusion is to reposition them surgically with a small segment alveolar osteotomy.
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4/5. Evidence that charcot-marie-tooth disease with tremor coincides with the Roussy-Levy syndrome.

    We report data on 3 members of a family affected by a dominantly inherited disorder closely resembling Roussy-Levy syndrome (RLS). Electrophysiological findings showed a marked decrease of motor and sensory conduction velocities and EMG signs of mild neurogenic damage. light and electron microscopy of sural nerve biopsy showed a hypertrophic neuropathy with diffuse onion-bulb formations and marked decrease of large size fibers. Teased fiber preparations evidenced reduced internodal lengths and segmental demyelination. Other data from the literature on RLS are reviewed and discussed. The hypothesis that RLS is not a disease entity but a hypertrophic-type of charcot-marie-tooth disease with essential tremor (HMSN type 1) is strongly supported.
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5/5. Hemifacial hypertrophy affecting the maxillary dentition. Report of a case.

    A case of hemihypertrophy affecting part of the right maxilla was followed for 8 years with sequential radiographs. The effects of this unusual condition on the developing dentition were documented. Advanced root formation prior to active resorption, increased deposition of secondary dentin, and the histopathologic description of a tooth have not been previously recorded.
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