Cases reported "Maxillary Diseases"

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1/33. An aggressive peripheral giant cell granuloma in a child.

    An 11-year-old boy was evaluated for a maxillary lesion that had been increasing in size over a six-month period. Intraoral examination revealed a firm ulcerated lesion on the maxillary posterior alveolus, which was displacing teeth. The lesion was painful and easily bled during mastication. An incisional biopsy of the lesion resulted in the diagnosis of peripheral giant cell granuloma. The patient was taken to the operating room for excision of the lesion and extraction of involved teeth. The patient was followed for a period of six months with no recurrence.
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2/33. Recurrent giant cell reparative granuloma of hard palate: role of Tc-99m-MDP three-phase bone scan.

    A 42-year-old female presented with pain in the hard palate following surgical curettage for giant cell reparative granuloma. Radiographs of the face at this time did not reveal any abnormality. The surgeons' query was to differentiate fibrosis from recurrence before considering her for recurettage. The three-phase bone scan helped in establishing the diagnosis of recurrence, which was confirmed after histological examination of the curettaged tissue.
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3/33. Central giant cell granuloma: a troublesome lesion.

    This case reports a central giant cell granuloma, an uncommon but potentially troublesome idiopathic benign lesion. Its radiographic similarity to a routine inflammatory periradicular lesion led to not only delay in diagnosis, but also further delay in successful treatment.
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4/33. Highly aggressive brown tumour of the maxilla as first manifestation of primary hyperparathyroidism.

    A case is presented of a 62-year-old man with a right maxillary swelling for the previous three months. The lesion was expansive and osteolytic, with invasion of the adjacent maxillary sinus, nasal and pterygomaxillary fossae and floor of the orbit. histology revealed the presence of an intrabony giant cell lesion. blood tests demonstrated elevations in calcium (16.2 mg/dl) and parathyroid hormone (PTH) concentrations (841 pg/ml). This suggested the diagnosis of hyperparathyroidism initially manifesting as a brown tumour of the maxilla. Posterior explorations confirmed the existence of an underlying ectopic parathyroid adenoma as the cause of the condition.
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5/33. An endosseous, implant-retained obturator for the rehabilitation of a recurrent central giant cell granuloma: a clinical report.

    The prosthodontic rehabilitation of a patient with a recurrent maxillary CGCG has been described. The patient's immense defect originally was rehabilitated with a scapular microvascular free flap, endosseous dental implants, and an ISP that became obsolete by virtue of the resection of recurrent disease and the subsequent need for velopharyngeal obturation. The patient was provided with a surgical obturator at the time of the resection of the recurrent CGCG. The surgical obturator thereafter was modified into an interim obturator to provide velopharyngeal competence while the soft tissues around the palatal defect healed. Finally, the construction of a bar-retained definitive obturator markedly improved the patient's speech, mastication, and deglutition.
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6/33. A central giant cell granuloma in a patient seeking orthodontic treatment.

    BACKGROUND: This case report illustrates the need to consider central giant cell granuloma, or CGCG, in the differential diagnosis in cases of greatly displaced teeth and facial deformity. The authors review the literature regarding CGCG and emphasize the importance of early diagnosis and treatment. CASE DESCRIPTION: A 9-year-old boy was seen in an orthodontic office with the chief complaint of overlapping front teeth. On radiographic examination, the authors noted a large maxillary midline radiolucency. The child was referred to an oral and maxillofacial surgeon for definitive diagnosis and treatment. CLINICAL IMPLICATIONS: patients may come to dental offices for treatment of malaligned teeth. The dentist needs to be aware of possible oral pathology when malaligned teeth are present. CGCG should be included in the differential diagnosis for patients with greatly displaced teeth and facial deformity.
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7/33. Rapid growth of giant cell granuloma in pregnancy treated with calcitonin.

    The giant cell granuloma of the jaws is a benign osteolytic lesion that may be treated by curettage, intralesional corticosteroids, or calcitonin. These medical treatments may be particularly useful when lesions arise in the immature facial skeleton, recur, or enlarge very rapidly-the last two situations being recognized complications of pregnancy. In this study, a patient is presented with a central giant cell lesion of the maxilla that switched from a relatively indolent growth pattern to become a rapidly enlarging and destructive lesion in the maxilla almost immediately after the patient became pregnant. Although calcitonin treatment is normally avoided in pregnancy, it proved highly effective, caused no obstetric or fetal side effects, and was not contraindicated by renal failure due to lupus nephritis. Histologically, the lesion was converted to a fibro-osseous lesion-like appearance. On the basis of the results of this case, calcitonin appears to be a safe, effective, and conservative treatment for giant cell granulomas that enlarge rapidly during pregnancy.
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8/33. Central giant cell granuloma of the palate; unusual localisation in a five year old child.

    The central giant cell granuloma of the maxillo-mandibular region is a relatively uncommon lesion (3.5%-0.1%). Essentially, it occurs in the second decade of life and it is mainly located in the mandibular region. The female/male ratio is 3:1. The authors present an unusual localisation of central giant cell granuloma in a five year old child's maxillary bone. After a surgical curettage of the lesion, an eighteen months follow-up examination did not show any recurrence.
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9/33. oral manifestations of renal osteodystrophy: case report and review of the literature.

    renal osteodystrophy, characterized by uneven bone growth and demineralization, is described. oral manifestations of the disorder are described, and the value of dental radiographs in early detection of renal osteodystrophy is noted. A case report of a patient with severe oral complications, which resulted from long-standing end-stage renal disease and secondary hyperparathyroidism, is presented. Giant cell lesions of hyperparathyroidism, referred to as brown tumors (which may be associated with pain and swelling), are the key clinical oral manifestations and are the most dramatic dental radiographic finding in patients with renal osteodystrophy. Bone changes may include loss of lamina dura, giant cell lesions of hyperparathyroidism, and bone demineralization. The dentist's role in detection, assessment, and treatment is stressed.
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10/33. Giant cell granuloma of the maxilla: case report.

    Giant cell granuloma is an uncommon bony lesion in the head and neck region, most commonly affecting the maxilla and mandible. Although it is a benign disease process, it can also be locally destructive. Surgery is the traditional and still the most accepted treatment for giant cell granuloma. The case described here involved the maxilla which was treated with surgical excision, followed by local injection of steroids.
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