1/16. Acute renal failure following ingestion of wild mushrooms.We describe three cases of acute renal failure in young men who ingested wild mushrooms with the intent of producing hallucinations. Two cases remained dialysis dependent and, in these cases, renal biopsy revealed tubulointerstitial nephritis and fibrosis. Similar cases have been reported in other countries, but not in australia. The most recognized mushroom nephrotoxin is orellanine, however the causative mushroom species and the actual toxin involved in these cases are unknown.- - - - - - - - - - ranking = 1keywords = wild (Clic here for more details about this article) |
2/16. Acute poisoning with tricholoma equestre.Two cases, mother and her son, suffering from acute poisoning with tricholoma equestre were described. They had eaten 100-300 grams of this wild mushroom during nine consecutive meals. About 48 hours after the last meal containing the mushroom they developed fatigue, muscle weakness and myalgia, loss of appetite, mild nausea, profuse sweating. Maximal serum creatine kinase activity was 18,150 U/L in the mother and 48,136 U/L in the son. Maximal serum levels of aspartate aminotransferase and alanine aminotransferase were 802 U/L and 446 U/L, respectively, in the mother and 2002 U/L and 454 U/L, respectively, in the son. All routine biochemical tests were within normal range. No other causes of rhabdomyolysis such as parasitic, viral, immune diseases, trauma or exposure to medications were found. All the above mentioned symptoms and biochemical abnormalities disappeared within 23 days of hospitalization. Our observation confirms the results of Bedry and co-workers that tricholoma equestre contains a toxin, which can cause rhabdomyolysis.- - - - - - - - - - ranking = 0.2keywords = wild (Clic here for more details about this article) |
3/16. mushroom poisoning by Chlorophyllum molybdites in the Midwest united states. Cases and a review of the syndrome.The paper describes two incidents of poisoning by the mushroom Chlorophyllum molybdites and reviews the literature covering this organism, a common agent of mushroom poisoning in many countries and the most common cause of mushroom poisoning in north america. Both poisoning incidents occurred in adults and were associated with severe gastrointestinal symptoms including profuse diarrhea, vomiting and intestinal pain. In each case, hospitalization was required. An unusual aspect of one case was the development of signs and symptoms suggestive of muscarine poisoning. The review includes a description of the mushroom, the geographic distribution of cases, the signs and symptoms of poisoning and its treatment, the toxic principles, particularly susceptible populations, and the variations in response associated with cooking C. molybdites and with individual idiosyncrasies. For identification of C. molybdites, the reader is alerted to the inappropriateness of some books, including many written in europe, and is warned of the occasional finding of sterile mushrooms that lack its characteristic green basidiospores. spores, as allergens, are discussed and simple rules are given for eaters of wild mushrooms.- - - - - - - - - - ranking = 0.2keywords = wild (Clic here for more details about this article) |
4/16. Acute poisoning with tricholoma equestre of five-year old child.This report presents the first case in pediatric literature of the acute intoxication of a five-year old male child who ate 300-400 grams of tricholoma equestre daily for four consecutive days before the onset of poisoning. The symptoms included acute respiratory failure with the need of respiratorotherapy, muscle weakness concerning especially the pelvic girdle and the urinary bladder. The boy could not sit or stand up without help and the bladder had to be catheterised, several times daily, to avoid urine retention. The biochemical tests showed only minor muscle injury with maximal activity of creatine kinase 306 U/L, aspartate aminotransferase 39 U/L, alanine aminotransferase 56 U/L. No other causes responsible for the signs mentioned above such as trauma, viral, bacterial, neurologic and immune diseases or exposure to medications were found. All the symptoms and biochemical abnormalities disappeared within 12 days of hospitalisation. We believe that the clinical picture of poisoning with this wild mushroom might be different in children and adults.- - - - - - - - - - ranking = 0.2keywords = wild (Clic here for more details about this article) |
5/16. Fatal muscarinic syndrome after eating wild mushrooms.death from mushroom poisoning in australia is rare and usually due to liver failure produced by amanita phalloides. We report a 53-year-old woman in queensland who died from an acute muscarinic syndrome 10 hours after eating mushrooms belonging to the genus Rubinoboletus. To our knowledge, this is the first death in australia caused by non-amatoxin-producing mushrooms. It highlights the need for awareness of non-amatoxin-producing mushrooms as potentially lethal.- - - - - - - - - - ranking = 0.8keywords = wild (Clic here for more details about this article) |
6/16. Successful treatment of an adult with amanita phalloides-induced fulminant liver failure with molecular adsorbent recirculating system (mars).Despite significant advances in intensive care management of amanita phalloides-induced fulminant liver failure (FLF), patients with this condition still have a high mortality rate in the absence of orthotopic liver transplantation. Molecular Adsorbent Recirculating System (mars) is a new, cell-free, extracorporeal liver assistance method utilizing an albumin dialysate for the removal of albumin-bound toxins, and a highly effective depurative therapy in adults with wild mushroom-induced FLF. We report the case of a 39 year old woman with amanita phalloides-induced FLF, admitted to our intensive care unit (ICU) and treated with mars. Our patient had severe hepatic dysfunction: hepatic encephalopathy (grade II), ALT = 5022 (2475-10098) IU/L, bilirubin = 7.18 (4.8-10.1) mg/dL, prothrombin time (PT) = 90.4 (29.3-140.4) s. mars sessions had an immediate impact on liver tests: statistically significant decrease in ammonia, ALT and PT. hepatic encephalopathy was successfully reduced. The patient survived and the hepatic function completely recovered. mars appears to be a safe and highly effective depurative therapy in adults with amanita phalloides-induced FLF.- - - - - - - - - - ranking = 0.2keywords = wild (Clic here for more details about this article) |
7/16. Acute renal failure caused by mushroom poisoning.Despite the abundance of wild mushrooms growing in a variety of habitats in taiwan, mushroom poisoning is rarely reported. Here, we report two cases of mushroom poisoning, possibly due to amanita smithiana or a related species. A 54-year-old man and his 52-year-old wife presented with acute anuric renal failure 1 day after ingesting some white mushrooms they had collected when mountain climbing; they made a full recovery following hemodialysis and supportive treatment. Although rare, mushroom poisoning should be considered in the differential diagnoses of acute renal failure.- - - - - - - - - - ranking = 0.2keywords = wild (Clic here for more details about this article) |
8/16. Successful treatment of a child with fulminant liver failure and coma due to amanita phalloides poisoning using urgent liver transplantation.Intoxication due to eating wild mushrooms presents with a variety of signs, ranging from mild diarrhea to severe organ failure. We present the case of an 11-year-old boy with fulminant liver failure and hepatic coma due to amanita phalloides poisoning treated with an urgent pediatric orthotopic liver transplantation. Successful treatment of patients with fulminant liver failure and hepatic coma caused by amanita phalloides poisoning is possible using urgent orthotopic liver transplantation when conservative medical treatment modalities are ineffective.- - - - - - - - - - ranking = 0.2keywords = wild (Clic here for more details about this article) |
9/16. amanita bisporigera ingestion: mistaken identity, dose-related toxicity, and improvement despite severe hepatotoxicity.Ingestion of wild mushrooms has led to unintentional poisonings caused by mistaken identity. We report 3 cases of exposure to amanita bisporigera, demonstrating dose-related toxicity. The use of nasobiliary drainage as a novel approach to interrupting the enterohepatic circulation of amatoxins is illustrated. Pathophysiology and treatment of amanita poisoning are reviewed.- - - - - - - - - - ranking = 0.2keywords = wild (Clic here for more details about this article) |
10/16. Poisoning due to amatoxin-containing Lepiota species.Twenty-seven consecutive mushroom poisoning cases were followed up over a period of 14 days. Fourteen out of 27 died of liver failure. There were no deaths from renal failure. The mushrooms were identified as the amatoxin-containing Lepiota species. Therapeutic measures included nasogastric lavage, charcoal, vitamin C, vitamin B, penicillin g, corticosteroids, oral streptomycin and, in the case of a few patients, limited amounts of thioctic acid. Of the ten haemodialysed, nine died. Unfortunately charcoal haemoperfusion was not available. It appeared that therapeutic measures were ineffective and it also seemed that the amount of mushroom ingested was the determining factor for the prognosis. An important point to make is that renal failure does not occur and liver failure is always delayed (group II). For this reason all suspected cases of mushroom poisoning, regardless of absence of clinical signs and symptoms, must be hospitalised for a period of at least one week. The poisonous properties of wild mushrooms have been recognized since ancient times. However, despite awareness of their inherent dangers, serious poisoning continues to occur. Fatal intoxications can be attributed almost entirely to the amtoxin-containing species. amanita phalloides have been blamed for over 90% of poisoning deaths in north america. There are reports of intoxications of other amatoxin-containing species in europe, but fatalities due to Lepiota species are reported only rarely. It was previously acknowledged that the interval between ingestion of mushrooms and the onset of symptoms is longer than expected in serious poisonings.(ABSTRACT TRUNCATED AT 250 WORDS)- - - - - - - - - - ranking = 0.2keywords = wild (Clic here for more details about this article) |
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