Cases reported "Pseudotumor Cerebri"

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1/9. Headaches and papilledema secondary to dural arteriovenous malformation.

    A 21-year-old developed progressive headaches and pulsatile tinnitus. He was found to have papilledema and a pulsatile bruit. A dural arteriovenous malformation was not recognized on brain CT or MRI, but was well documented on magnetic resonance angiography and cerebral angiography. Dural malformations draining into the superior sagittal sinus can cause increased intracranial venous pressure and secondarily increase intracranial pressure even without a mass effect.
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keywords = pulsatile tinnitus, tinnitus
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2/9. Long-term evolution of papilledema in idiopathic intracranial hypertension: observations concerning two cases.

    Chronic headaches, associated with papilledema and pulsatile tinnitus without any neuroradiologic, cytobiochemical or cerebrospinal fluid abnormalities are suggestive of idiopathic intracranial hypertension (IIH). However the absence of the papilledema does not rule out this diagnosis. The reason why some patients do not develop papilledema in IIH is ignored, however there are some hypotheses concerning the structure of the optical nerve. In this study we described two female patients that presented diagnosis of IIH with papilledema, with subsequent resolution of papilledema without the due resolution of intracranial hypertension. The long-term behavior of the optic nerve (ON) facing an increased intracranial pressure was evaluated through repeated measurements of the intracranial pressure. We concluded that the ON submitted to high intracranial pressure for a certain length of time can adapt itself with subsequent disappearance of the papilledema. The presence or not of papilledema in IIH can be related to the period in which the diagnosis is accomplished.
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keywords = pulsatile tinnitus, tinnitus
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3/9. dronabinol reduces signs and symptoms of idiopathic intracranial hypertension: a case report.

    A case is presented in which a woman diagnosed with a longstanding history of idiopathic intracranial hypertension reported improvement of frontal headaches, photophobia, transient blindness, enlarged blind spots, and tinnitus after smoking marijuana. All these symptoms and signs were associated with increased intracranial pressure (220-425 mm of water). Treatment with dronabinol at a dose of 10 mg twice a day, then reduced to 5 mg twice a day, relieved all of her symptoms. Previously noted papilledema and enlargement of blind spots also resolved, and this, in the absence of psychoactive effect or weight gain.
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ranking = 0.13030445619042
keywords = tinnitus
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4/9. ventriculoperitoneal shunt as treatment for perilymphatic fistula: a report of six cases.

    We report six cases of perilymphatic fistula in patients who received ventriculoperitoneal shunts as part of their final mode of therapy. The last of our 6 patients actually received a ventriculoperitoneal shunt as her initial mode of therapy. All but one had benign intracranial hypertension. All six felt better (less disequilibrium, tinnitus, and pressure and occasional hearing improvement) after LP with removal of 15-20 ml of cerebrospinal fluid.
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ranking = 0.13030445619042
keywords = tinnitus
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5/9. Otologic manifestations of pseudotumor cerebri.

    Otologic manifestations of pseudotumor cerebri heightened awareness of pseudotumor cerebri and its varied subtle presentations will allow for a more expedient diagnosis by the otolaryngologist. pseudotumor cerebri is defined as increased intracranial pressure and papilledema without a mass lesion or obstruction of the ventricular system. Presenting symptoms most commonly are headache and visual dysfunction. However, patients may present with associated symptoms of tinnitus, dizziness and hearing loss. These patients may be seen first by the otolaryngologist. Fourteen patients with a diagnosis of pseudotumor cerebri are reviewed. Nine of the 14 patients had neurotologic symptoms during the course of their evaluation. pseudotumor cerebri is a diagnostic challenge. Keys to diagnosis are a high index of suspicion, careful history and funduscopic examination in patients with unexplained neuro-otologic symptoms.
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ranking = 0.13030445619042
keywords = tinnitus
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6/9. The minor symptoms of increased intracranial pressure: 101 patients with benign intracranial hypertension.

    Of 101 patients with benign intracranial hypertension not related to vasculitis, neck stiffness occurred in 31, tinnitus in 27, distal extremity paresthesias in 22, joint pains in 13, low back pain in 5, and gait "ataxia" in 4. Symptoms resolved promptly upon lowering the intracranial pressure by lumbar puncture, and were probably directly caused by intracranial hypertension. awareness of these "minor" symptoms of increased intracranial pressure can facilitate diagnosis and management.
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ranking = 0.13030445619042
keywords = tinnitus
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7/9. Otologic symptoms and findings of the pseudotumor cerebri syndrome: a preliminary report.

    pseudotumor cerebri or benign intracranial hypertension is a syndrome characterized by increased intracranial pressure without focal signs of neurologic dysfunction. The clinical manifestations of this syndrome are usually headache and/or disturbance of vision. Although tinnitus, hearing loss, and vertigo have been described in association with intracranial hypertension, otologic symptomatology as the presenting manifestation of this syndrome has not been previously reported. In this article we report the otologic symptoms and findings of two pseudotumor cerebri patients, one of whom presented with pulsatile tinnitus. The pathogenesis of the otologic symptoms, diagnostic workup, and management of these patients are discussed.
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ranking = 1.1303044561904
keywords = pulsatile tinnitus, tinnitus
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8/9. tinnitus from intracranial hypertension.

    Five patients had unilateral tinnitus from increased intracranial pressure of different etiologies. In each case, the tinnitus was produced by a venous bruit and could be decreased by Valsalva's maneuver, head turning to the ipsilateral side, or by light pressure over the ipsilateral jugular vein. Correction of the increased intracranial pressure obliterated the tinnitus. Turbulence, created as blood flows from the hypertensive intracranial portion into the low pressure of the jugular bulb, is proposed as the mechanism producing the tinnitus.
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ranking = 0.5212178247617
keywords = tinnitus
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9/9. Pulsatile tinnitus: recent advances in diagnosis.

    Pulsatile tinnitus can often be a diagnostic problem. This communication reports the authors' experience on 100 patients with pulsatile tinnitus and describes a practical and effective diagnostic approach. Better understanding of the various etiologies of pulsatile tinnitus, coupled with the introduction of magnetic resonance angiography, in conjunction with magnetic resonance imaging, have made a major impact on the evaluation of this symptom in recent years. cerebral angiography, previously performed on the majority of patients, is presently indicated in selected cases only. intracranial hypertension, glomus tumors, and carotid atherosclerosis were the most common diagnoses made in our patients.
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ranking = 2.6515222809521
keywords = pulsatile tinnitus, tinnitus
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