1/10. Hypocalcemic tetany in a toddler with undiagnosed rickets.A 17-month-old black girl presented to the pediatric emergency department with sudden onset of her hands and feet "drawing up." A diagnosis of vitamin d deficiency rickets with tetany was made on a history of exclusive breast-feeding, low serum calcium, phosphorus, and 25-hydroxyvitamin D levels, and bone radiographs. Nutritional rickets should be considered in the differential diagnosis of acute hypocalcemia.- - - - - - - - - - ranking = 1keywords = vitamin, deficiency (Clic here for more details about this article) |
2/10. Latent tetany and anxiety, marginal magnesium deficit, and normocalcemia.The identification of marginal magnesium deficit, such as we have detected in a patient with anxiety, depression, and psychomatic complaints, is a difficult diagnostic problem. electromyography of a limb, rendered acutely ischemic either just before or after hyperventilation, can elicit latent tetany in this condition, as well as in calcium deficiency. We have demonstrated iterative electrical activity in our patient, whose magnesium deficit is attributable to renal wasting of magnesium. We have elicited similar patterns in several other patients, who had marginally low serum magnesium and who also exhibited weakness, anxiety, and psychosomatic disorders. This preliminary report suggests the need for further consideration of the possibility that chronic magnesium-deficit may contribute to the syndrome of latent tetany, psychosomatic complaints, and weakness.- - - - - - - - - - ranking = 0.049667965677524keywords = deficiency (Clic here for more details about this article) |
3/10. Variable presentations of rickets in children in the emergency department.Vitamin D-deficient rickets is uncommon but becoming more prevalent in the pediatric population likely related to increases in breast-feeding. It should be considered in many clinical situations. We present 3 cases of rickets presenting acutely to the emergency department. Their presentations included a fracture concerning for child abuse, tetany, and hypocalcemic seizures. In all cases, laboratory and radiographic evaluations were consistent with the diagnosis of nutritional rickets and their symptoms were related to rickets resolved with appropriate treatment. Although uncommon, vitamin D-deficient rickets should be considered in children with the above presentations.- - - - - - - - - - ranking = 0.47516601716124keywords = vitamin (Clic here for more details about this article) |
4/10. Tetanic crisis and antiepileptic drugs. A case report.OBJECTIVE AND methods: We presented a rare case of tetanic crisis in a 23-year old mentally retarded woman with epilepsy after treatment by oxcarbazepine, the new anticonvulsant agent. We reviewed laboratory, radiographic and medical examinations and recommend a proper treatment in such cases. RESULTS AND CONCLUSION: The laboratory tests revealed only severe hypocalcemia. We described the potential role of oxcarbazepine in the induction of activity of cytochrome P 450 system of hepar and increases of less active metabolism of vitamin D. Supplementation of vitamin D and calcium in patients taking antiepileptic drugs is in the same case crucial.- - - - - - - - - - ranking = 0.95033203432248keywords = vitamin (Clic here for more details about this article) |
5/10. keratoconus-tetany-menopause: the new association.This report describes the unusual case of 46-year-old white European woman who presented spontaneous keratoconus. This case suggests a new association: keratoconus-tetany-menopause. This can be explained by Thalasselis' syndrome, a syndrome showing a relation between keratoconus, magnesium (Mg) deficiency, type A behavior, and allergy. Furthermore, Thalasselis' syndrome integrates old and new theories. This report also suggests that the alteration induced by severe but reversible Mg deficiency at the intra- and extracellular levels, associated with metabolic and hormonal imbalances, may trigger the development of keratoconus. Clinical data which support this thesis are reported.- - - - - - - - - - ranking = 0.099335931355048keywords = deficiency (Clic here for more details about this article) |
6/10. central nervous system magnesium deficiency.The central nervous system concentration of magnesium (Mg ) appears to have a critical level below which neurologic dysfunction occurs. Observations presented suggest that the interchange of the Mg ion between the cerebrospinal fluid, extracellular fluid, and bone is more rapid and dynamic than is usually believed. This is especially so when the hypertrophied parathyroid gland is associated with significant skeletal depletion of Mg as judged by history rather than serum level. Magnesium, much like calcium, has a large presence in bone and has a negative feedback relationship with the parathyroid gland. A decline in central nervous system Mg may occur when the skeletal buffer system orchestrated largely by the parathyroid glands is activated by an increase in serum calcium. Observations in veterinary medicine and obstetrics suggest that the transfer of Mg from the extracellular fluid into bone during mineralization processes may be extensive. If the inhibition of the hypertrophied parathyroid gland is prolonged and the skeletal depletion of Mg extreme, serious neurologic symptoms, including seizures, coma, and death, may occur. noise, excitement, and bodily contact appear to precipitate neurologic symptoms in Mg ( )-deficient human subjects as it has been documented to occur in Mg ( )-deficient experimental animals. The similarity of the acute central nervous system demyelinating syndromes with reactive central nervous system Mg deficiency is reviewed.- - - - - - - - - - ranking = 0.24833982838762keywords = deficiency (Clic here for more details about this article) |
7/10. Di-George's syndrome with multiple infections (an autopsy report).Di-George's syndrome is a rare condition of congenital immunodeficiency. An infant presented as hypocalcemic tetany with multiple infections. autopsy confirmed the hypoplasia of thymus and thyroid associated with other anomalies. A brief of the relevant literature is presented.- - - - - - - - - - ranking = 0.049667965677524keywords = deficiency (Clic here for more details about this article) |
8/10. tetany induced by hypokalemia in the absence of alkalosis.A 36-year-old patient developed tetany manifested only by a positive Trousseau's sign and with a negative Chvostek's sign 8 weeks after gastric bypass surgery for obesity. The usual causes of tetany (hypocalcemia, hypomagnesemia and alkalosis) were absent. The only possible etiology found was hypokalemia. Previous cases of hypokalemia induced tetany reported were always accompanied by alkalosis. Its absence in our patient makes this case unique and determines that hypokalemia per se in the absence of alkalosis may be a cause of tetany. It is suggested that the Trousseau's sign should be investigated in patients with severe potassium deficiency.- - - - - - - - - - ranking = 0.049667965677524keywords = deficiency (Clic here for more details about this article) |
9/10. Symptomatic hypoparathyroidism in acquired immunodeficiency syndrome.Involvement of endocrine organs is frequent in patients with hiv infections. We report the first case of symptomatic hypoparathyroidism in a patient in the course of HIV infection. He presented with tetany and hypocalcemia in the presence of decreased levels of parathyroid hormone, which persisted after correction of hypomagnesemia. The family history was negative and none of the autoimmune diseases associated with hypoparathyroidism was present. No local destruction by tumor or infection could be demonstrated apart from HIV infection. We propose a causal role of HIV infection in the development of hypoparathyroidism and discuss the possible mechanisms of parathyroid involvement in AIDS.- - - - - - - - - - ranking = 0.1986718627101keywords = deficiency (Clic here for more details about this article) |
10/10. Neonatal hypocalcaemia after intrauterine exposure to anticonvulsant drugs.Two cases are reported of prolonged hypocalcaemia with tetany in infants born at term, whose mothers had been treated with phenytoin and phenobarbitone in high doses. Both infants presented with jitteriness and tetany in the first and second weeks of life, and in both the hypocalcaemia was resistant to therapy over a longer period. An effect on the fetal vitamin D metabolism by phenytoin and phenobarbitone, resulting in defective bone mineralization and neonatal hypocalcaemia is suggested.- - - - - - - - - - ranking = 0.47516601716124keywords = vitamin (Clic here for more details about this article) |
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