How would you expect this to affect blood ph and respiratory rate and is this a state of acidosis or alkalosis?

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Patient ingested E Coli and was diagnosed with food poisoning and has had chronic diarrhea for the past 24 hours. The patient has a medical history of Chron's Disease. The diarrhea has been going on nonstop for 24 hours. How would you expect this to affect the blood ph and respiratory rate? Is the patient suffering from a state of acidosis or alkalosis and would you expect this to be related to metabolic or respiratory disorder and why?
To answer this, you need more information. Renal function and pulmonary function will influence the end result. Also the activity of the Crohn's needs to be established, and whether an enteroenteric fistula is present.

However: Assuming he has non-secretory diarrhea (ie not bicarbonate wasting as in a villous adenoma of the colon), then the primary event will be a metabolic alkalosis due to volume contraction. If his kidney function is normal, he will attempt to retain sodium (along with bicarbonate) and lose potassium in the urine. Due to some fairly complex actions in the proximal and distal tubule, he will effectively retain bicarbonate, leading to the alkalosis.

If he has normal pulmonary function, he will probably not have a respiratory compensation (it would have to be hypoventilation, which is hard to do unless other things are going on (such as sedatives, altered level of conciousness etc).

As time goes by, and if the diarrhea persists, in the absence of volume replacement, he will ultimately develop pre-renal azotemia, which will affect the metabolic component (he will develop a metabolic acidosis that is distinct from the metabolic alkalosis he already has).

If he goes on to develop hypeovolemic shock, other things come into play - etc etc etc.

Its not always straighforward. I hope this helps.  (+ info)

How does CNS depression cause respiratory alkalosis?

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Now as far as I knew, CNS depression caused a decreased respiratory rate and would therefore cause hypoventilation and would therefore cause respiratory acidosis, however my sources say that CNS depression causes hyperventilation and therefore is a cause of respiratory alkalosis. Can anyone explain the pathophysiology of this to me so I can understand it?
Yes, quite simply. Your sources are wrong.  (+ info)

How does the renal system compensate for respiratory alkalosis?

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Respiratory Alkalosis is caused by hyperventilation. You are breathing so fast you are losing too much of your body's acid-CO2 and the HCO3 levels (the body's base) build up. The respiratory system governs the CO2 by either speeding up or slowing down your rate of breathing to raise or lower CO2 levels. The renal system governs the bodys base-HCO3 by excretion through the kidneys. They excrete more or less HCO3 when needed. In Respiratory Alkalosis, the renal system increases excretion of HCO3 to try to even out the acid-base balance.  (+ info)

Does respiratory alkalosis have anything to do with urinary incontinence in women?

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no it does not ... respiratory alkalosis is primarily a dysfunction in breathing ... specifically ... hyperventilation  (+ info)

why does rapid breathing at high altitudes results to respiratory alkalosis?

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Lower Po2 at high altitudes increases the respiratory rate and the heart rate. When you increase the respiratory rate you blow off more CO2 resulting in less acid in your blood. This is a respiratory alkalosis. If you bring supplemental oxygen with you this can be prevented.
God bless.
PS I should have told you that as you gain altitude the barometric pressure decreases and if the FIO2 (the fractional concentration of oxygen) remains the same then you are breathing in less oxygen and become hypoxemic.  (+ info)

when a patient is in respiratory alkalosis how do you increase their paco2 levels?

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If they are conscious and breathing spontaneously you get them to breathe into a paper bag. This helps retain CO2 instead of blowing off to much.

If they are unconsious and on a vent then you need to decrease the minute ventilation. You can do this by either lowering their mandatory rate or decreasing their tidal volume.

Have a great day!  (+ info)

What are the preventions against Respiratory Alkalosis?

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Anxiety, fever, stimulant drugs, pain, sepsis(a toxic condition resulting from infection), hypobarism (high altitude), and any other condition in which ventilatory elimination of C02 exceeds it's production. Basically you are hyperventilating! These are all causes of respiratory alkalosis, so if you can manage to avoid all of these you should be okay.  (+ info)

why does prolonged crying results to respiratory alkalosis?

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Because of the prolonged sobbing which is similar to sighing. This increases your minute ventilation and blows off CO2 resulting in the respiratory alkalosis.  (+ info)

In response to respiratory alkalosis, the (what happens)?

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A) respiratory rate increases
B)tidal volume increases
C)kidneys conserve bicarbonate
D)kidneys secrete more hydrogen ions
C  (+ info)

explain how the respiratory and renal system's compensates for metabolic acidosis and alkalosis?

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MMB has it wrong.

When your metabolic system is acidic, the natural response of your respiratory system is to increase your breathing rate. Hyperventilating will blow off CO2 which will raise the pH level in your blood.

To a certain extent the reverse is true with alkalosis.

However, change in the respiratory system is really more of a temporary stop-gap measure employed by the body while it tries to use other means to reset your pH to the normal level of around 7.40  (+ info)

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