My sister is 24.MRI of her brain showed infarction in the left posterior parietal region...recovery?

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Each part of the brain has its own function, therefore the neurological deficits that result from a stroke depend on the location in the brain it occurred. The left and right parietal lobes contain the primary sensory cortex, which controls sensation (touch and pressure), and a large association area that controls fine sensation (judgment of texture, weight, size, and shape). Damage to the left parietal lobe may disrupt a patient’s ability to understand spoken and/or written language. Her symptoms will depend on the exact location in the parietal lobe and the size of the region involved. If her stroke is truly in the posterior part of the parietal lobe, then her language center should not be affected and she should not have difficulties with speech or writing. If she does have difficulty with speech, she can work with a speech pathologist to improve this. She may have difficulty interpreting sensory information, but with therapy she should be able to learn to compensate for this. She is very young to have a stroke, therefore it is very important to determine the cause of her stroke so that her cause can be modified to reduce her chance of a future stroke. Out of all the areas of the brain to have a stroke the parietal lobe produces the least disabling symptoms. You should of course ask her doctor because he/she will know all the details of her stroke and prognosis for recovery. Good luck to you and your sister.  (+ info)

Which of the following best describes the best location of the femoral artery pressure point.?

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A)At the crease where the thigh meets the trunk. B)Posterior to the medial malleolus of the ankle.C) On top of the foot.D)On the medial aspect of the posterior knee.
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Why is pain felt in a myocardial infarction?

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I think it would be either because there is not enough oxygen being delivered, or narrowing of the arteries. Can someone help me out with the answer to this question?
1. The heart is working far harder than in healthy times, trying to push though the extreme narrowing or occlusion. It is a muscle, not so different than other muscles in your body.
2. Tissue is dying, because oxygen levels are low. This is painful.  (+ info)

I have a 7.5 bi-lateral anterior cerebral artery aneurysm. The Dr. sent me back to work and test later?

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I have another one that 6.4mm and my work is heavy and fast paced and he said there was a 50/50 chance of it rupturing. Shouldn`t I have been hospitalized and tested right away? I had a concussion from being hit on the head at work and kept getting sicker over 2 mo. period. On tues. last wk I had a severe headache was pale, weak and sick at my stomach, sore neck and back and black cicrle under eyes since accident.and sleepy alot. Dr. said this has nothing to do with the aneurysm. Should I get a second opponion?
First of all I will say that no matter what it is your body, and if you are uncomfortable with the prospect of going back to work then by all means you should get a second, third or fourth opinion if that is what it takes. That being said, I would hope that the doctor you are referencing was a neurosurgeon! I would not trust a decision like this to a family doctor or even a regular ER doc. I would request a neurosurgery consult and face to face discussion with them! Hope this helps!  (+ info)

What Does TOPROL Do For Acute Myocardial Infarction And Congestive Heart Failure?

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FATHER HAS DIAGNOSIS ABOVE AND IS HAVING EPISODES OF VOMITING, DIZZINESS AND SLURRING WORD. HE HAS AN EXTENSIVE HISTORY OF HEART DISEASE. TRIPLE BYPASS AT 60, 2 HEART ATTACKS AT 70 EMT'S BROUGHT HIM BACK TWICE THEN HAD ANGIOPLASTY FOLLOWED BY A 99.9% BLOCKED COROTID ARTERY?
Toprol is a beta-blocker. (there are different receptors and binding of molecules causes different effects--binding at the beta receptors increases the heart rate and eventually the blood pressure) It controls the heart rate and also decreases the blood pressure. Controlling the heart rate during a heart attack is important because it decreases the amount of oxygen that the heart needs and during a heart attack, there is less oxygen available in the first place. If your father has just begun to have these symptoms recently, it sounds like he should go to the emergency department. there may be something seriously wrong--slurred words may be a sign of stroke, or they could all be signs of a heart attack with decreased blood flow to the brain.  (+ info)

What is the difference between infarction and gangrene?

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The morphology of both is the same,that is,ischemic necrosis.I know gangrene involves putrefaction which infarction does not.Then,why is repair by fibrosis possible only in infarction,but not in gangrene?What are the exact definitions of infarction and gangrene?Please differentiate them clearly.
Infarction- (1) engorgement or stoppage of a vessel. (2) development or presence of an infarct (a localized area of ischemic necrosis produced by occlusion of the arterial supply or venous drainage of the part.)
Cardiac infarction- myocardial infarction.
cerebral infarction- an ischemic condition of the brain, causing a persistent focal neurological deficit in the area affected.
intestinal infarction- occlusion of an artery or arteriole in the wall of the intestine, resulting in the formation of an area of coagulation necrosis.
myocardial infarction- formation of an infarct in the heart muscle, due to interruption of the blood supply to the area.
pulmonary infarction- infiltration of an airless area of lung with blood cells, due to obstruction of the pulmonary artery by an embolus or thrombus.

gangrene- death of the tissue in considerable mass, usually associated with loss of vascular (nutritive) supply and followed by bacterial invasion and putrefaction.  (+ info)

A blood clot in which artery of the coronary circulation would be more likely to cause sudden death?

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1-right coronary artery
2-posterior intreventricular branches
3-circumflex branch
4-anterior intreventricular branch
The left anterior descending artery is a large artery often called the "widow maker." However a clot in the left main coronary artery would put virtually the entire myocardium in jeopardy.  (+ info)

What is cerebral thrombosis?

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On my grandfather's death certificate, it says he died from cerebral thrombosis and in parenthesis next to it it says vertebral artery. Does this mean he had a stroke?
I am so sorry for your loss.

Cerebral thrombosis is when a blood clot forms in an artery that supplies the brain. When the artery is major and the blood clot is large enough to block the blood flow, tissue death results. This is called a stroke. In layman's terms, your grandfather died of a massive stroke.

The vertebral artery supplies the brainstem and the back of the brain. Your grandfather's blood clot was found in the vertebral artery. Any problem with the brainstem is life threatening.  (+ info)

How fast does nitroglycerin act when used to treat a myocardial infarction, i.e. when given in tablet form?

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I have been trying to find information online about the speed and efficacy of nitroglycerin in tablet form when used to treat a myocardial infarction. It's for a short story I am writing, and I can't finish until I find this information. I don't want to just make something up.
Nitroglycerin doens't actually do anything to treat a myocardial infarction. It is best for angina- the squeezing pain people get in their chest when the blood vessels around their heart are constricted but not completely closed. For angina, it works in seconds to relieve the pain and relax those blood vessels so the heart gets more blood.
During a heart attack, the coronary vessels close completely, and blood flow to part of the heart is cut off. A person who takes nitroglycerin for angina should worry that they are having a heart attack if they take nitroglycerine and the pain does not go away. They should take an aspirin immediately and go to the ER to be given other meds or have a stent put in to open the artery back up.  (+ info)

Can pericardial effusion lead to myocardial infarction or any other serious consequence?

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I am seeking to find out if pericardial effusion resulting from a viral infection can lead to myocardial infarction and/or any other serious consequence?
if left untreated it can lead to cardiac tamponade, which is compression of the heart due to a large increase of fluid in the pericardium. this condition is life-threatening as it prevents the heart from pumping effectively.  (+ info)

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