Would pleurisy be classified as a respiratory disease/disorder or pleural disease/disorder?

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I've had pleurisy forever and know the in's and out other than how to classify it. is it a disease or disorder or is that too extreme? is it just a never-ending never-going away never-getting treated infection or a lung virus?! ALSO, i know pleurisy can cause complications with other viruses and diseases but can one just die from having pleurisy...can it be fatal?
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Which is the best hospital in the world to treat pleural effusion?

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Hi, my girl is suffering from pleural effusion and this happened at a time when we were planning to tie the knot. I am extremely worried, is completely curable? Which is the best hospital in the world to treat this disease? Please reply.
depends on the CAUSE of the effusion  (+ info)

My sister & I would like to set up a pleural mesothelioma blog. How do we go about doing this? ?

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Our father has been diagnosed with this rare disease.
If you want to start free then you can go to blogger.com or wordpress.com  (+ info)

Does anyone know about cirrohssis particularly accumulating ascites/pleural effusion after 5 yrs of TIPS work?

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My father had the TIPS procedure done 5 years ago because he had ascites accumulation and it went away after TIPS but now ascites is back plus pleural effusion. My dad has cirrohssis of the liver and according to the doctor his TIPS is working well so why does he have ascites and pleural effusion then?
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what are the 3 priority nursing diagnoses for pleural effusion secondary to coronary artery disease?

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the patient was diagnosed to have congestive heart failure. and also, undergone PTCA.
Sounds like this question should be under the "Homework" section for nursing school..!

At least it sounds exactly like my old questions...LOL  (+ info)

When a person has a pleural effusion, what is that? And what is happening to the person on a cellular level?

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I know that pleural effusion refers to an abnormal collection of fluids in the pleural cavity, but how does that affect respiration? What exactly is going on?
Background: Pleural effusion is defined as an abnormal accumulation of fluid in the pleural space. Excess fluid results from the disruption of the equilibrium that exists across pleural membranes.
In terms of anatomy, the pleural space is bordered by parietal and visceral pleura. Parietal pleurae cover the inner surface of the thoracic cavity, including the mediastinum, diaphragm, and ribs. Visceral pleurae envelop all surfaces of the lungs, including the interlobar fissures. This lining is absent at the hilus, where pulmonary vessels, bronchi, and nerves enter the lung tissue. The mediastinum completely separates the right and left pleural spaces.

Both parietal and visceral membranes are smooth, glistening, and semitransparent. Despite these similarities, the two membranes have unique differences in anatomic architecture, innervation, pain fibers, blood supply, lymphatic drainage, and function. For example, the visceral pleurae contain no pain fibers and have a dual blood supply (bronchial and pulmonary).


Pathophysiology: Pleural effusion is an indicator of a pathologic process that may be of primary pulmonary origin or of an origin related to another organ system or to systemic disease. It may occur in the setting of acute or chronic disease and is not a diagnosis in itself.

Normal pleural fluid has the following characteristics: clear ultrafiltrate of plasma, pH 7.60-7.64, protein content less than 2% (1-2 g/dL), fewer than 1000 WBCs per cubic millimeter, glucose content similar to that of plasma, lactate dehydrogenase (LDH) level less than 50% of plasma and sodium, and potassium and calcium concentration similar to that of the interstitial fluid.

The principal function of pleural fluid is to provide a frictionless surface between the two pleurae in response to changes in lung volume with respiration. The following mechanisms play a role in the formation of pleural effusion:


Altered permeability of the pleural membranes (eg, inflammatory process, neoplastic disease, pulmonary embolus)

Reduction in intravascular oncotic pressure (eg, hypoalbuminemia, hepatic cirrhosis)

Increased capillary permeability or vascular disruption (eg, trauma, neoplastic disease, inflammatory process, infection, pulmonary infarction, drug hypersensitivity, uremia, pancreatitis)

Increased capillary hydrostatic pressure in the systemic and/or pulmonary circulation (eg, congestive heart failure, superior vena caval syndrome)

Reduction of pressure in pleural space; lung unable to expand (eg, extensive atelectasis, mesothelioma)

Inability of the lung to expand (eg, extensive atelectasis, mesothelioma)

Decreased lymphatic drainage or complete blockage, including thoracic duct obstruction or rupture (eg, malignancy, trauma)

Increased fluid in peritoneal cavity, with migration across the diaphragm via the lymphatics (eg, hepatic cirrhosis, peritoneal dialysis)

Movement of fluid from pulmonary edema across the visceral pleura

Persistent increase in pleural fluid oncotic pressure from an existing pleural effusion, causing accumulation of further fluid

Iatrogenic causes (eg, central line misplacement)  (+ info)

Is it dangerous to give water to pet/doggy when she/he had a pleural effusion or a heart condition?

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Is it dangerous to give water to pet when she/he had a pleural effusion or a heart condition or something that has to do with rapid heart beat and rapid breathing with accompanied fluid build up that can be seen like a fat hanging in the ribs or chest part of the doggy?
Ask the vet who diagnosed this condition. You should be following up with the vet for this anyway; it does affect the pumping of the heart and circulation.
If it's a temporary condition (as in, after accident with broken ribs) - hang in there.
If chronic illness, the important thing is to make your dog comfortable esp if pet is seriously ill. I have been thru this decision a few times and still it's hard to decide whether the dog is still happy and comfortable enough to continue...  (+ info)

What diseases would have the symptom of coughing up blood or blood in the phlegm?

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This is not a symptom of my own so please don't tell me to go to the doctor, it is for a piece of work I have to do for College.

If you could tell me the name of a disease/ diseases that cause this, preferably not consumption or TB, a little about it and other symptoms it would be a massive help!
If you could also include treatment options and how serious a disease it is that would also be fantastic!



Hope you can help! Thanks!
First: spitting up blood is clinically known as: HEMOPTYSIS (bloody sputum, spit)
Yes, pneumonia is the most likely, but......
The following is from my medical e-book (I'm a nursing student)

"Blood in the sputum (hemoptysis) is most often seen in clients with chronic bronchitis or lung cancer. Clients with tuberculosis, pulmonary infarction, bronchial adenoma, or lung abscess may have grossly bloody sputum."
Also the end stage of cycstic fibrosis will present with hemoptysis.

a biggie in the hospital is:
PULMONARY EMBOLISM
PATHOPHYSIOLOGY
A pulmonary embolism (PE) is a collection of particulate matter (solids, liquids, or gaseous substances) that enters venous circulation and lodges in the pulmonary vessels. Large emboli obstruct pulmonary blood flow, leading to decreased systemic oxygenation, pulmonary tissue hypoxia, and potential death. Any substance can cause an embolism, but a blood clot is the most common.

Pulmonary embolism is the most common acute pulmonary disease (90%) among hospitalized clients. In most people with PE, a blood clot from a deep vein thrombosis (DVT) breaks loose from one of the veins in the legs or the pelvis. The thrombus breaks off, travels through the vena cava and right side of the heart, and then lodges in a smaller blood vessel in the lung. Platelets collect with the embolus, triggering the release of substances that cause blood vessel constriction. Widespread pulmonary vessel constriction and pulmonary hypertension impair gas exchange. Deoxygenated blood shunts into the arterial circulation, causing hypoxemia. About 12% of clients with PE do not have hypoxemia.

Pulmonary embolism affects at least 500,000 people a year in the United States, about 10% of whom die. Many die within 1 hour of the onset of symptoms or before the diagnosis has even been suspected.

For clients with a known risk for PE, small doses of prophylactic subcutaneous heparin may be prescribed every 8 to 12 hours. Heparin prevents excessive coagulation in clients immobilized for a prolonged period, after trauma or surgery, or when restricted to bedrest. Occasionally, a drug to reduce platelet aggregation, such as clopidogrel (Plavix), is used in place of heparin.



A smaller one that popped up in the book:
GOODPASTURE'S SYNDROME
PATHOPHYSIOLOGY
Goodpasture's syndrome is an autoimmune disorder in which autoantibodies are made against the glomerular basement membrane and neutrophils. The two organs with the most damage are the lungs and the kidney. Lung damage is manifested as pulmonary hemorrhage. Kidney damage manifests as glomerulonephritis that may rapidly progress to complete renal failure (see Chapters 74 and 75). Unlike other autoimmune disorders, Goodpasture's syndrome occurs most often in adolescent or young adult men. The exact cause or triggering agent is unknown.

COLLABORATIVE MANAGEMENT
Goodpasture's syndrome usually is not diagnosed until serious lung and/or kidney problems are present. Manifestations include shortness of breath, hemoptysis (bloody sputum), decreased urine output, weight gain, generalized nondependent edema, hypertension, and tachycardia. Chest x-rays show areas of consolidation. The most common cause of death is uremia as a result of renal failure.

Spontaneous resolution of Goodpasture's syndrome has occurred but is rare. Interventions focus on reducing the immune-mediated damage and performing some type of renal supportive therapy.

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What diseases can you get from cutting yourself with a rusty knife?

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This is a question from a growing nurse. I've always wondered if you really can get a disease from a rusty knife or any rust and what kind of diseases you can get.
staph aureus and staph epidermidis are commonly found on the skin and are responsible for
most infected wounds. methicillin resistant staph aureus (MRSA) is becoming a serious
problem. tetanus (clostridium tetani) is also a possibility but is usually not a problem with superficial
cuts that bleed a lot. infected wounds not treated properly can become gangrenous (clostridium
perfringens). clostridium bacteria are anaerobic which means that require a lack of oxygen to
grow. poor circulation or elevating an infected foot may lead to gangrene due to the lack of oxygen
in the infected area. if a person touches the cut with unclean hands, e. coli could infect the wound.  (+ info)

How were these diseases prevented or cured in the 1600 to early 1700s?

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Measles
Smallpox
Chickenpox
Malaria

How were some of these diseases dealt with in the 1600s? If there was no cure or anything to prevent the diseases to happen can you explain why and what resulted in these situations? Thanks!
Inoculation was sometimes used to prevent smallpox but basically either you lived or you died. Most survived chickenpox & measles but there were those who died or were left scarred or with damage to the vision or nervous system. Malaria was a disease of the tropics and is found in parts of Africa, Asia, the Middle East, Central and South America, Hispaniola, and Oceania. Mostly people died.

The 1600s were in the 17th century & the 1700s were in the 18th century. Do some online research.  (+ info)

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