what does high blood pressure causing chronic renal failure mean?
It means that your kidneys are not working as well as they should be and the reason is that they have hypertensive changes in the glomerlus and the blood vessels as the cause (
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why banana is rejected for a patient with chronic renal failure?
When kidneys fail, they can no longer remove potassium. That excess potassium will build up in the body. This can cause a blood disorder called hyperkalemia.
It can lower your pulse, give you an irregular heart beat, it can even lead to heart failure and death. (
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how does accupressure help in treatment of chronic renal failure?
Acupressure do help in expulsion of toxins created in the body by pressing the point of Kidney in both hands and legs which are towards the centre of the palms. But It can help recovery but cannot be a cure in itself. Chronic renal failure do need medicines. Eel Serum (Srum Anguillae) and Zingber Off. are two medicines of value alongwith indicated remedies for the morbid condition causing the Renal failure like Lupus, infections, heart ailments with high blood pressure, iatrogenic and the like. Apis Mel, Cantharis, Thuja, Calcaria Sulph,.Beri Beris Vulgaris, Hydrangea, Pareira Brava etc. used prudently according to symptom picture would surely cure maladies behind renal failure. Acupressure and Homeo is a great combination for diagnosis as well as treatment and cure. With best wishes.
Medicines should be used in consultation with homeo doctor. (
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Is renal insufficiency precipitated by NSAID use, reversible?
Is this for you? I don't know the answer, just wanted to send you a big hug, and wish you the best of health. Take good care, my friend!!
((((((You)))))) :) (
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what is Urea,creatinine, Phosphate and Chronic Renal Failure disease?
Urea and creatinine are measures of kidney function (they are byproducts of protein breakdown), phosphate is an electrolyte found in the blood which is important in muscle function.
Urea, creatinine and phosphate all are elevated in renal failure because the kidneys cannot remove them.
Dialysis will remove these. (
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I have chronic renal failure and am not sure what type of foods are good for me, can anyone help out there?
My mom called me and asked me if i knew exactly what i am supposed to eat and what not to eat. I know that some fruits and vegetables are suppose to be bad for you but why and what other types of food can I eat, be specific as possible......
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from what I know you need to eat things low in protein and salts. Don't intake to many fulids and potassium. Don't drink dark colored soft drinks. The goal is to limit the amount of work your kidneys have to do. The website at the bottom has specific food to avoid. (
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what will happen in red blood cell during chronic renal failure?
Pls. answers my qusetion.....just for my nursing subject cause Im a BSN student need the answer to day. much as possible to day or tommorow
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This is a basic anatomy and physiology question. I'm very concerned that you are in a BSN program and do not know this/cannot easily find this answer on your own! (
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Is there an effective alternative to Sensipar in chronic renal failure/treatment of hyperparathyroidism?
Any natural augmentation for the med; rx of sx nausea, or other meds I read about but not yet proven or approved in US? So far removal seems only treatment. Any info is appreciated.
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Mainly surgery is the only alternative (
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why is it hypertension is one of the factor than can cause chronic renal failure?
The increased blood pressure puts extra pressure on the glomeruli [the ball shaped filtering packets of the inner kidney] and eventually this causes damage.
Sadly, the body responds to failing kidneys by increasing the blood pressure in an effort to make said kidneys more efficient, so this is a vicious circle.
While increased blood pressure does have some effect on the small blood vessels of major organs, that is more an effect of the glycosolated end products associated with diabetes mellitus (
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if a patient has c chronic renal failure is it still possible to reverse the condition?
Chronic renal failure (CRF) is the progressive loss of kidney function. The kidneys attempt to compensate for renal damage by hyperfiltration (excessive straining of the blood) within the remaining functional nephrons (filtering units that consist of a glomerulus and corresponding tubule). Over time, hyperfiltration causes further loss of function.
Chronic loss of function causes generalized wasting (shrinking in size) and progressive scarring within all parts of the kidneys. In time, overall scarring obscures the site of the initial damage. Yet, it is not until over 70% of the normal combined function of both kidneys is lost that most patients begin to experience symptoms of kidney failure.
Control hypertension (high blood pressure)—Target systolic blood pressure (BP) is 120 to 135 mm Hg; target diastolic BP is 70 to 80 mm Hg. Antihypertensive medication from the ACE class is preferable because of protective effects on the kidneys.
Restrict dietary protein—Dietary protein is broken down into amino acids and absorbed from the stomach into the blood. The amino acids are taken from the bloodstream and used to build muscle and perform other essential functions. Excess amino acids are further broken down into carbohydrates and nitrogen-containing waste that is eliminated by the kidneys. Amino acid disposal further burdens the kidneys, and is believed to speed the progression of CRF. This process is like forcing a damaged machine to work harder, causing it to break down sooner than expected.
Affected patients must be cautious not to overdo protein restriction, because it can lead to malnutrition and muscle wasting. Moderate protein restriction for a CRF patient is about 0.6 to 0.8 gm/kg/day, which is effectively achieved by following the advice of a dietician.
Manage pre-end-stage renal disease (pre-ESRD)—Treatment for pre-ESRD should begin once the glomerular filtration rate (GFR) falls below 30 milliliters per minute (< 30 mL/min). Pre-ESRD management includes the identification and treatment of anemia (low red blood cell count). When the GFR drops below 30 mL/min, anemia often develops because the kidneys produce an inadequate amount of erythropoetin (EPO). This hormone is made by the kidneys and travels to the bone marrow, where it stimulates red blood cell production. Anemic patients are candidates for EPO (Procrit®) injections to maintain their hematocrit (volume percent of red blood cells in whole blood) between 30% and 36%.
Identify and Treat Secondary Hyperparathyroidism—With the loss of kidney function, phosphate accumulates in the blood. Excess phosphate in the blood reduces levels of blood calcium, and low blood calcium levels trigger the parathyroid gland (located in the neck) to release more parathyroid hormone (PTH). PTH then dissolves bone tissue to release stored calcium and raise the level of calcium in the blood. This chronic cycle of events is called secondary hyperthyroidism.
The net result of this condition is the development of metabolic bone disease (renal osteodystrophy). These patients are at risk for bone fractures, bone and muscle pain, which can sometimes be accompanied by severe itching, and cardiovascular complications. Severe itching is thought to be in part due to the elevated circulating PTH level itself.
Tin (
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