1+ Tricuspid/Pulmonic valve insufficiency?
just picked up my test results from cardiologist and I see it says:
Assessment: 1+ tricuspid valve insufficiency and 1+ pulmonic valve insufficiency
Aim 19 years old this test is a year old. (Never went back to the doc to get them until now) the reason I went back is because I feel a slight discomfort in my chest from time to time. Is not pain just discomfort that I get from walking moderate distance or lifting 20+ LB things. Please help
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The pulmonic valve is normally a thin tricuspid structure that prevents blood from regurgitating into the right ventricle once ejected into the low-pressure pulmonary circulation. Pulmonic regurgitation refers to retrograde flow from the pulmonary artery into the right ventricle during diastole. Physiologic (trace-to-mild) pulmonic regurgitation is present in nearly all individuals, particularly in those with advanced age. However, pathologic conditions that produce excessive and clinically significant regurgitation can result in impairment of right ventricular function and eventual clinical manifestations of right-sided volume overload and heart failure. Often, pulmonic regurgitation is not the primary process but a finding secondary to an underlying process such as pulmonary hypertension or dilated cardiomyopathy.
http://emedicine.medscape.com/article/157639-overview (
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what is mild tricuspid regurgitation,trace pulmonary valve insufficiency,atrial septrum is aneurysmal,?
also patent foramen ovale is suspected.have chest pains,high blood pressure140/100 pulse 110-116 i have passed out twice in two years very tired alot not over weight.i am taking blood pressure med, 325mg aspirin a day and a beta-blocker(blood pressure)and hydrochlorot(diuretic) DR TELLS ME NOTHING TO WORRY ABOUT ,well im concerned because my gram died aneurysm in brain aunt has lot of problems w/heart her 2girls heart murmurs grandad died of something with his heart (idiopathic pulmonary embolism) or fibrosis of heart something like that,uncle and gram died from congestive heart failure my mom no problems dad has emphazema. will i suddenly die from CHF.i've read that CHF is from some underlying problem. im 36yrs old and have other health issues to name couple asthma,(from smoking) dr hears wheezing, i think its something else and fibromyalgia (im also on the pain patch fentanyl(50mg every 2 days) and had radiation treatment once. thanks very much in advance for all your help
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minldan - Oh my, I'm sure Sandy A meant well but a lot of what she wrote is not quite correct, so DON'T PANIC! In truth, the echocardiographic findings that you mention are all fairly benign, but I will go through them one by one for you.
1) Mild tricuspid regurgitation - Regurgitation just means blood leaking backwards through a valve. Having read hundreds of echocardiograms, I can tell you that it is rare for an echo NOT to have some trace to mild regurgitation. Most valves do not form a 100% perfect seal, but that does not mean they are abnorma. The tricuspid valve separates the top and bottom chambers on the right side of your heart. The fact is that the VAST majority of people with mild tricuspid regurgitation will NEVER have any problems that require medication or surgery. I would not say "it isn't closing as it should" because that implies that the valve is abnormal which is not necessarily the case.
2) Trace pulmonary valve insufficiency - The pulmonary valve prevents blood from leaking back into your heart after it is pumped into the lungs. "Trace" means that the doctor was able to see the teeniest squirt of blood back through the valve. Again, this is NOT abnormal and I would NOT say that your valve "isn't working totally as it should" or that it "isn't closing as it should". Also, there IS some blackflow of blood through the valve (that's what insufficiency means) but again, it is so little that we don't even call it "mild".
3) Aneurysmal atrial septum - The atrial septum is the wall of tissue that separates the top left and right chambers of your heart. "Aneurysmal" means that the wall is a little floppy and bounces back and forth. It is not "weak" - it's more like there is extra material there, like a size 4 woman wearing a size 8 dress. Now DON'T PANIC. It sounds scary, but some people think that there MIGHT be an increased risk of strokes in people with aneurysmal atrial septums. The key word is MIGHT. Also, remember that being at INCREASED risk doesn't mean that you are at HIGH risk. If something increases your risk from 1 in a million to 2 in a million, for example, your risk is increased, but it's still 2 in a million! That is why cardiologists do not recommend doing anything drastic like surgery for this - the surgery would probably be riskier than not doing anything at all! Taking an aspirin a day is probably a reasonable thing to do but at this point, we don't recommend it for everyone with an aneurysmal atrial septum.
4) Patent foramen ovale - OK, this not a birth defect or anything. We all have a foramen ovale (small hole) between the top chambers of our hearts while we are in our mothers' bellies. This is necessary to allow our moms to oxygenate our blood since we can't breathe when we're in the womb. Most will close within the first month of life BUT up to 25% of us will have incomplete closure and we call this a patent foramen ovale. 25%!!! That means if you have 20 people in a room together, 4 or 5 of you will have a PFO! That's not so bad, right? Are we going to do surgery to close a hole on 25% of the population? Of course not! In a TINY fraction of people with a PFO, sometimes a blood clot can sneak through and cause a stroke. If someone has a stroke and we think this has happened, we put people on either a blood thinner or an aspirin. We are doing studies now to see if closing the hole NOT WITH SURGERY but with special catheters will reduce the risk of a second stroke compared with just medicines. Again, up to ONE QUARTER OF THE EARTH will have a PFO and almost all of them will go through life without a single problem related to it. Unless you've had an unexplained stroke, when we see a PFO, we don't really think twice about it.
OK, so that's out of the way. Aside from that stuff, Sandy A makes some good points about your long-term health risks. Your high blood pressure is MUCH more likely to cause problems than the PFO or the aneurysmal atrial septum, so you should be focused on that and not on your pretty unremarkable echo! You need to get that under better control which may mean higher doses of medicines or additional medicines. The higher your blood pressure, the higher your long-term risk of heart failure, strokes, kidney diseaes, and heart attacks. Again, DON'T PANIC! This doesn't need to be brought under control TODAY or THIS WEEK or THIS MONTH even. Your blood pressure isn't high to the point of being an emergency, but if left that high for years and years and years, you are more likely to develop the problems mentioned above.
That fact that your gram died of a brain aneurysm isn't really alarming, but it means your blood pressure needs to be well-controlled. I wouldn't worry about your cousins having murmurs, especially since your echo didn't show any real valve problems. If your granddad had idiopathic pulmonary fibrosis (there's really nothing called idiopathic pulmonary embolism) that doesn't increase your risk of heart disease - it's similar to cancer. Uncle and gram dying from congestive heart failure doesn't necessarily mean you are in great danger either unless they were younger than 55 when they developed heart problems. Still, it would suggest that you need to get your blood pressure under control. Emphysema is almost always related to smoking so as long as you don't smoke, that should not affect you.
Don't worry about what Sandy A is saying about your renal arteries. A very small minority of people with high blood pressure have renal artery stenosis and we don't look for it unless you have difficult to control high blood pressure. Right now, with only two blood pressure medicines, you still have a lot of room for improvement.
I know that's a lot of information, but I wanted to make sure that Sandy A doesn't scare you too much. See your doctor and make sure your blood pressure is brought under control. Don't freak out about the echo results, they are truly nothing that you should worry about. I know she means well, but cardiology nurses don't go through years of intense training in interpreting echocardiograms; cardiologists do. If you have any other questions, feel free to send me an email. Hope that helps. Good luck! (
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i have trivial tricuspid insufficiency, is this something i need to see a Dr. for?
conclusion to echocradiogram says
1.normal left ventricular function.
2.no mitral valve prolapse or pericardial effusion.
3.trivial tricuspid insufficiency.
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1) You've got a doctor, the one who ordered the echo
2) trivial means just that, very little.
So, don't worry. (
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Which biocompatible material would be best suited for replacing a tricuspid valve?
We've made a design for a new tricuspid valve for the heart and need to know which biocompatible material would be sturdy yet flexible and also durable....
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A culture of the patients endothelial and fibrous tissues would be most bio compatible for replacing a tricuspid valve. All other materials would encounter rejection problems. (
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I have a leak in the mitral valve and a leak in tricuspid valve, can someone tell me something about this?
I just found out that I have a leak in two valves in my heart, the mitral valve and the tricuspid valve, can someone tell me about this, what does this mean? Is it dangerous? And tell the what questions I should ask my doc as I have an appointment with him.
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Too bad you're not a car, I can do a valve job on a car. If you were a car, I would say you were probably running lean and the ignition timing was off. That's a sure way to crap out a valve.
I'm a mechanic, Jim, not a doctor.
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Poor Star Trek joke. (
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My daughter has HLHS and she has had her final surgery the Fontan. mild leaking of the tricuspid valve ?
After her echo they refer to mild leaking of the tricuspid valve has anyone with hlhs experienced this?
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Hypoplastic Left Heart Syndrome (HLHS) is a condition in which the left side of the heart is underdeveloped. Usually, the left ventricle, the left atrium, the mitral valve and the aorta are affected. It is called a syndrome because it can encompass several different variations and varying degrees of development of these parts of the heart.
To understand HLHS, it is helpful to understand how a normal baby's heart works. The heart is comprised of four chambers: the upper chambers are called the left and right atria, and the lower chambers are called the left and right ventricles.
Very simply, in a healthy heart blood flows from the right atrium to the right ventricle where it is then pumped through the pulmonary artery to the lungs to be oxygenated. Blood then flows back to the heart via the left atrium to the left ventricle, which pumps this oxygenated blood through the aorta out to the body. This is how the body's organs and tissue receive oxygen, which is vital. When a baby has HLHS, the left side of the heart is underdeveloped so it cannot sufficiently pump the oxygenated blood out to the body.
Babies with HLHS do not have problems while in the womb - it is only after birth that the heart fails to work properly. This is because all babies receive oxygen from the placenta while in the womb, so blood does not need to go to the lungs. In addition, there is an opening between the pulmonary artery and the aorta, called the patent ductus arteriosus (PDA) that is present in all babies. It allows the blood to go from the right ventricle out to the body, bypassing the left side of the heart.
The PDA usually closes a few days after birth, separating the left and right sides of the heart. It is at this time that babies with undetected HLHS will exhibit problems as they experience a lack of blood flow to the body. They may look blue, have trouble eating, and breathe rapidly. If left untreated, this heart defect is fatal - usually within the first few days or weeks of life.
Once HLHS has been diagnosed, a drug called prostaglandin is given to keep the PDA open until surgery is performed. There are two surgical options for treating HLHS: a Heart Transplant or the 3-Stage Surgical Procedure, the first stage of which is called the Norwood procedure. (For bloodless surgical options, please see the Columbus Children's Hospital
website.)
3-STAGE SURGICAL PROCEDURE:
An alternative to a heart transplant is the 3-Stage Surgical Procedure. The 3-Stage Procedure reconstructs the child's heart allowing it to work using only two of the heart's four chambers.
The first stage is called the Norwood procedure, or the "Modified Norwood" which was developed in 1979 by Dr. William Norwood. Because the left ventricle cannot pump blood adequately out to the body, the Norwood procedure allows the right ventricle to pump blood to both the lungs and the body. This surgery is usually performed during the baby's first week of life. A new version of this first surgery is called the "Sano Shunt." Ask the surgeon about the pros and cons of the traditional Norwood vs. the Sano Shunt, how many of each he has performed, and what the success rates are.
The second stage procedure, called the Hemi-Fontan or Bidirectional Glenn, reduces the work of the right ventricle by allowing it to pump blood only to the body, and allowing most of the blood to flow automatically from the body into the lungs. The second stage is performed when the pressure in the baby's lungs has decreased, no longer requiring the pumping action of the right ventricle to get blood into the lungs. This surgery is usually performed at about 4-6 months of age when the traditional Norwood has been performed. When the Sano Shunt is performed, the second surgery usually needs to be done earlier - at about 3-4 months of age - and the babies need constant monitoring to determine when they are ready for the second surgery.
The third and final stage, the Fontan, allows the rest of the blood coming back from the body to go to the lungs. There are two different ways to perform the Fontan: The Lateral Tunnel Fontan, the traditional approach, and the newer External Conduit Fontan. The Lateral Tunnel Fontan is usually performed at about 18 months - 3 years of age, and the External Conduit Fontan is usually performed at about 3-4 years of age.
The External Conduit Fontan uses an external conduit, or tube, that is placed just outside the heart. It is hoped that by using this conduit that the potential for arrhythmias will be greatly reduced in the future. However, this is a relatively new procedure and the outcomes will not be known for several years. It is usually done later than the Lateral Tunnel Fontan because the child has to be big enough to handle a conduit that can handle the blood flow as the child grows.
There are pros and cons of each type of Fontan procedure, so make sure you discuss with your surgeon the different types of Fontan operations and what the risks and benefits are of each.
See our Resources Page for websites that contain more detailed explanations and diagrams of each of the surgeries (
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why is the valve in the right ventricle of the heart called the Tricuspid valve?
And also why is the valve in the left ventricle called the bicuspid valve
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the tricuspid valve has three "flaps" and the bicuspid valve has two. (
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My Dr. says the result of my echocardiogram shows my tricuspid valve with a mild to moderate leakage?
Can this be repaired? Would it be the cause of recent chest discomfort & shortness of breath? Thanks to anyone that can help.
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The chest pain and shortness of breath are unlikely to be related to your tricupsid regurgitation. The tricupsid valve is on the right side of the heart and does not produce symptoms related to the lung, such as shortness of breath or cough. Tricupsid regurgitation itself is not known to produce chest pain. Mild to moderate tricupsid regurgitation is unlikely to produce symptoms or need treatment. With severe disease you can develop lower extremity swelling, enlarged/painful liver and abdominal swelling.
Tricuspid regurgitation is most commonly functional, being caused by dilatation of the right ventricular and the tricuspid annulus. Right ventricular dilatation may result from any condition that directly involves the right ventricle or causes pulmonary hypertension and an elevation in right ventricular systolic pressure, leading to dilatation of the right ventricle and tricuspid annulus.The treatment for functional TR is to treat the underlying cause, such as primary pulmonary hypertension.
Most cases of tricupsid regurgitation do not require treatment. Treatment should be considered symptoms of right heart sided failure occur and are unresponsive to medical therapy. The tricupsid valve is virtually never replaced. It can be repaired with Tricuspid valve annuloplasty. The procedure is generally well tolerated. Anyways, discuss it with your doctor, because it is unlikely you even need it repaired. (
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Surgical treatment of tricuspid valve atresia?
I read about the sequence of surgeries: first the Blalock-Taussig shunt, then the Glenn operation, and then finally the Fontan procedure. Why is this sequence used? Why isn't the Glenn operation and the Fontan procedure done at the same time, and the Blalock-Taussig shunt skipped entirely? Thanks.
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I have Tricuspid Atresia, but my surgical treatment was different. I had the Glenn procedure at six months; the Modified Blalock-Taussig at 10 years; and the Fontan at 21. But I was born in 1966, and they were using a different form of the Glenn back then. Today it is known as a "Bidirectional Glenn Shunt", and sends blood to both lungs. My Glenn only goes to the right lung -- according to my Cardiologist, they changed the way the operation was performed in the mid 1980's.
A "by-the-book" Modified Blalock-Taussig dictates the use of a Gortex tube to create the shunt, but surgeons like to keep things as simple as possible. The less time the patient is out; and the fewer times that he/she needs to be operated on, the better. So if it is possible to use existing blood vessels to create the shunt, the surgeon will do it.
The sequence of operations is dictated by what various medical tests show the doctors is the best route to take. And after the first operation, of course, the patient has a completely different anatomy. That also dictates what will happen next. (
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Why endocarditis affect the mitral valve and not the tricuspid valve?
This is dealing with Cardiovascular disorders
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An important factor in development of endocarditis that is the high pressure which's more common on left side ( where mitral valve present) so blood will gush forcefuly leading to tear of the valve and also fix bacteria on it that causes the disease.
But on Rt side ( tricuspid valve) there is low pressure source. (
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