1/74. Congestive heart failure treated by the upgrade from VVI to DDD pacing.The case is presented of an elderly woman with normal left ventricular (LV) systolic function and VVI pacing complicated by severe congestive heart failure. The symptoms and findings of congestive heart failure became refractory to medical treatment and resolved with the upgrade of the VVI to a DDD system. Right heart catheterization during VVI pacing showed increased mean pulmonary capillary wedge and right atrial pressures both being normalized under DDD pacing. This case report illustrates the need to consider permanent physiological pacing in elderly patients, even in presence of normal LV systolic function, to ensure AV synchrony when the atrium can be paced, since diastolic LV dysfunction is quite common in these subjects.- - - - - - - - - - ranking = 1keywords = dysfunction (Clic here for more details about this article) |
2/74. Autonomic effects of radiofrequency catheter ablation.Radiofrequency (RF) catheter ablation has become the treatment of choice for a variety of supraventricular tachycardias. Autonomic dysfunction may occur during application of RF current; these abnormalities resolve quickly when current delivery is terminated. We present a case of sinus node arrest and AV block in a 69-year-old woman induced by RF catheter ablation of an AV nodal slow pathway. The proposed mechanisms are a Bezold-Jarisch-like phenomenon or paradoxical activation of cardiac C fibres by direct neural sympathetic stimulation or RF-induced myocardial injury. Based on a review of previously published reports, autonomic effects of RF ablation are discussed.- - - - - - - - - - ranking = 1keywords = dysfunction (Clic here for more details about this article) |
3/74. Pacing-induced left ventricular dysfunction. Relationship with coronary perfusion.In a patient admitted with symptomatic complete heart block, a DDD pacemaker was implanted. Prior to implantation, echocardiography showed normal left ventricular function. Shortly after implantation, acute congestive heart failure developed with extensive regional hypo- and akinetic segments in the anteroseptal, anterolateral and apical region. Subsequent perfusion imaging with methoxyisobuticeisonitrite (MIBI) at rest demonstrated hypoperfusion in the same regions while coronary angiography showed normal epicardial coronary vessels. Thus, impaired regional coronary flow can be associated with cardiac stimulation, inducing marked deterioration of left ventricular function.- - - - - - - - - - ranking = 403.44806701018keywords = ventricular dysfunction, left ventricular dysfunction, dysfunction (Clic here for more details about this article) |
4/74. The Mount Sinai Hospital clinicalpathological conference: a 45-year-old man with Pompe's disease and dilated cardiomyopathy.This is an unusual case of a 45-year-old man, born in ecuador, with evidence of profound left ventricular dysfunction, dilated cardiomyopathy and marked myocardial hypertrophy. Preceding events were advanced atrioventricular block (necessitating pacemaker implantation) and atrial flutter. The diagnosis of Pompe's disease was established by endomyocardial biopsy and appropriate staining, which indicated abnormal glycogen storage.- - - - - - - - - - ranking = 100.86201675254keywords = ventricular dysfunction, left ventricular dysfunction, dysfunction (Clic here for more details about this article) |
5/74. Chronic binodal block with Wenckebach phenomenon.Coexistent sino-atrial and atrioventricular block with Wenckebach phenomenon is an extremely unusual cardiac arrhythmia. Observations on the natural history of sinus node dysfunction are rare. For over six years our elderly patient has been followed without symptoms but with increasing severity of atrio-ventricular block.- - - - - - - - - - ranking = 1keywords = dysfunction (Clic here for more details about this article) |
6/74. Use of an ultrasonic scalpel in the open-heart reoperation of a patient with pacemaker.Electrocautery has a potential risk of serious pacemaker dysfunction in patients with implanted pacemaker. Here we present the safe and efficient use of ultrasonic scalpel (Harmonic scalpel) for the first time in a patient with implanted pacemaker undergoing open-heart reoperation.- - - - - - - - - - ranking = 1keywords = dysfunction (Clic here for more details about this article) |
7/74. Cardiac pacing as emergency care for serious bradyarrhythmia with circulatory shock.Three cases of bradyarrhythmia with serious illness of extracardiac organs are reported. Case 1 had sick sinus syndrome. He was admitted to our hospital complaining of syncope and developed apnea. Case 2 had complete atrioventricular block and serious hepatic failure in the hospital. Case 3 had paroxysmal atrioventricular block. He complained of syncope which followed convulsions. Their symptoms might be due to circulatory shock caused by a lazy lower pacemaker from the ventricle. Emergent temporary pacing successfully improved the extracardiac organ dysfunction. Although their bradyarrhythmias were transient, permanent pacemakers were implanted to inhibit the recurrence. A quick temporary pacing should be indicated in patients with critical bradyarrhythmia like our cases for survival.- - - - - - - - - - ranking = 1keywords = dysfunction (Clic here for more details about this article) |
8/74. atrioventricular block after reciprocating atrioventricular junctional tachycardia.Short runs of symptomatic atrioventricular (A-V) block occurred after spontaneous cessation of reciprocating A-V junctional tachycardia in a patient with right bundle branch block, normal H-V interval and sinus nodal dysfunction. These episodes were characterized by long (more than 1 sec) P-P intervals during which the A deflections were not followed by His bundle electrograms. Three possible explanations are: (1) a posttachycardia-induced period of abnormally prolonged A-V nodal refractoriness; (2) pseudo-A-V block produced by concealed A-V junctional tachycardia, or (3) bradycardia-dependent (phase 4) A-V block at the "upper" His bundle, above the site from which the H deflection was recorded.- - - - - - - - - - ranking = 1keywords = dysfunction (Clic here for more details about this article) |
9/74. Alarming atrioventricular block and mitral valve prolapse in the kearns-sayre syndrome.kearns-sayre syndrome (KSS) is a multisystem mitochondrial disorder characterized by the invariant triad: onset before 20, progressive external ophthalmoplegia and pigmentary retinal degeneration, plus at least one of the following: complete (or not) heart block, cereberal dysfunction and CSF protein above 100 mg/dl. Autopsies from patients with KSS revealed widespread tissue distribution mtDNA deletions. These deletions result in significantly lower activities of the enzymes of the respiratory chain. The same deletion of mitochondrial dna present in skeletal muscle is found in myocardial tissue. An 18-year-old girl diagnosed with the KSS was admitted to our hospital because of an upper respiratory tract infection and dysphagia. ECG showed cardiac conduction defects. The patient had no history of syncope. At her surface ECG there was a complete RBBB (QRS duration approximately 130 ms), a clockwise rotation with an axis of approximately 90 degrees and a slight QT prolongation (420 ms). echocardiography showed prolapse with thickening and degeneration of both mitral valve leaflets but without mitral regurgitation. The patient was started on a diet rich in potassium and pharmaceutical therapy with magnesium oxide (240 mg of elemental Mg p.o. per day), 1 g of calcium carbonate t.i.d., vitamin d (calcitriol 0.25 microg p.o. per day) and coenzyme Q(10) 100 mg daily and discharged 6 days later with slightly improved biochemical profile but apparent clinical improvement. Urgent pacemaker implantation was decided but unfortunately the patient died due to acute cardiac arrest 10 days later.- - - - - - - - - - ranking = 1keywords = dysfunction (Clic here for more details about this article) |
10/74. coronary artery disease obscuring giant cell myocarditis--a case report.A case in which the diagnosis of idiopathic giant cell myocarditis was obscured by the presence of severe coronary artery disease is described. A 47-year-old man presented with recurrent inferior myocardial infarction and complete heart block. cardiac catheterization confirmed severe 2-vessel disease and left ventricular dysfunction. Incessant ventricular arrhythmia rapidly ensued, which did not respond to anti-arrhythmic therapy and overdrive pacing despite complete surgical revascularization. He eventually died. autopsy revealed giant cell myocarditis superimposed on coronary artery disease. Acute myocarditis masquerading as myocardial infarction has been well known, but virtually all reported cases had normal coronary arteries. This case illustrated the fact that even in the presence of obvious coronary artery disease the remote possibility of myocarditis should not be entirely disregarded. Although giant cell myocarditis is a rare and frequently fatal disorder, recent studies suggest that combined immunosuppressive therapy may improve the prognosis.- - - - - - - - - - ranking = 100.86201675254keywords = ventricular dysfunction, left ventricular dysfunction, dysfunction (Clic here for more details about this article) |
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