Cases reported "Mandibular Diseases"

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1/15. Non-familial cherubism--a case report.

    A case of cherubism in a 9 year old boy with no familial history is presented. Clinical features, histologic appearance, radiographic findings, differential diagnosis and rationale for treatment is discussed.
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2/15. cherubism: case reports and literature review.

    Two new cherubism cases have been documented and reported. Both were relatively mild clinically, being a Grade I within Motamedi's I to V clinical grading system. Each presented with bilateral posterior mandibular lesions having a multiloculated radiolucent appearance. In each case, detection with eventual diagnosis evolved from a mandibular molar failing to erupt (tooth No. 19). The partial literature review documented the wide spectrum of clinical expression of this disease and the consequent wide variation in its treatment.
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3/15. cherubism in siblings: a case report.

    cherubism is a non-neoplastic bone disease characterized by clinically evident bilateral, painless enlargements of the jaws that are said to give the patient a cherubic appearance. cherubism may appear in solitary cases or in many members of the same family, often in multiple generations. On radiography, the lesions exhibit bilateral multilocular radiolucent areas. Histopathologic evaluation reveals proliferating fibrous connective tissue containing numerous multinucleated giant cells. Since the first description of this condition in 1933, almost 200 cases have been reported. We describe cherubism in 2 siblings and briefly review the literature on this subject.
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4/15. Aggressive behaviour of cherubism in a teenager: 4-years of clinical follow-up associated with radiographic and histological features.

    cherubism is a rare hereditary fibro-osseous childhood disease characterized by bone degradation and fibrous tissue replacement at the angles of the mandible and at the tuberosity areas of the maxilla that leads to prominence of the lower face and an appearance reminiscent of the cherub's portrayal in Renaissance art. This disease has an autosomal dominant hereditary characteristic. The purpose of this report is to analyse laboratory tests, clinicopathological and radiographic features of cherubism and its intraoral manifestations in a patient during 4-years of follow-up, correlating the features observed in this case with those of the literature. Also discussed is the atypical and aggressive behaviour of this case during puberty.
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5/15. The aggressive form of cherubism: report of two cases in unrelated families.

    cherubism is a benign lesion that causes painless symmetrical enlargement of the jaws, usually with a familial tendency. We describe in two Chinese families two cases of the aggressive form of cherubism with extensive swelling on both sides of the mandible, typical microscopic findings, and apparent familial history.
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6/15. A prosthetic treatment approach for a cherubism patient: A clinical report.

    cherubism is an early childhood disease that primarily involves the mandible and consists of painless mandibular enlargement with or without maxillary involvement and progresses rapidly over the course of several years. This clinical report describes the fabrication of maxillary fixed partial dentures and a mandibular overdenture for a 21-year-old man with cherubism.
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7/15. Fibrous dysplasia and cherubism as an hereditary familial disease. Follow-up of four generations.

    Five cases of dysplasia of the jaws in one family which has been under our observation since 1970 are reported. The disease appeared as a mixed display of jaw lesions, in some members as fibrous dysplasia and in others as cherubism. We were able to trace the disorder through an unbroken line of four generations, and thus to demonstrate autosomal dominant inheritance. cytogenetic analysis performed on three members of this family revealed a significantly increased rate of chromosomal breakage.
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8/15. The noonan syndrome/cherubism association.

    noonan syndrome is characterized by short stature, unusual facies, congenital heart disease, chest deformity, mild mental retardation, and cryptorchidism in males. It may be sporadic or inherited as an autosomal dominant trait and occurs between 1 in 1000 and 1 in 2500 live births. cherubism is a giant cell lesion of the jaws thought to be transmitted as an autosomal dominant trait. It is usually recognized by age 7 years, follows a variable course, and is not known to be related to other genetic disorders. We herein report on four patients with noonan syndrome, all of whom had cherubism. Two other probable cases are cited in the literature for a total of six known cases.
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9/15. Treatment of mandibular cherubism.

    A case of cherubism of the mandible with an atypical history and with the onset of the facial deformity delayed until after puberty has been reported. The patient was treated during a relatively inactive stage, which followed a year of rather active growth. The distinct facial deformities were treated by a combination of curettage and recontouring, which resulted in an improved facial appearance and elimination of the fibro-osseous lesions of the mandible.
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10/15. Enlarging soft tissue mass involving the mandibular left alveolar ridge.

    The central giant-cell granuloma is a benign, fairly uncommon lesion that can appear clinically and radiographically similar to many other lesions. When histopathological examination discloses a giant-cell lesion of bone, clinical, radiographic, and laboratory data must be obtained and carefully analyzed to rule out such conditions as hyperparathyroidism, Paget's disease, and cherubism, and to confirm the diagnosis of central giant-cell granuloma. Once the diagnosis is established, thorough curettage or surgical excision of the tumor is recommended to assure complete removal. Although recurrence is rare, periodic postoperative examination is also suggested.
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