Cases reported "Oligohydramnios"

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21/35. autopsy findings in interstitial deletion 6q.

    autopsy findings from a child with interstitial deletion 6q [46,XX,del(6)(q13q21)] are reported. There was cervical scoliosis, an endocardial cushion defect, right ventricular hypertrophy, subependymal cysts, multicystic kidneys (Potter type IIB), and lung hypoplasia.
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22/35. The value of color Doppler sonography in the diagnosis of renal agenesis.

    Real-time ultrasonographic diagnosis of renal agenesis may be difficult because of severe oligohydramnios. Recent reports have suggested that the absence of renal arteries on color Doppler ultrasonography may assist in the identification of renal agenesis. Because the position of the renal arteries is variable owing to fetal size, this study was undertaken to determine the location of the renal arteries as a function of fetal growth. Using color Doppler ultrasonography, 200 normal fetuses were studied in which the length of the aortic segment from the bifurcation of the iliac arteries to the renal arteries was measured. Regression analysis was performed in which the measured aortic segment was the dependent variable and the femur length the independent variable. Four cases of severe oligohydramnios were studied. The regression equation for the length of the aortic segment (Y) was significantly (P < 0.000001; R = 0.921) related to the femur length (X) (Yaortic segment (mm) = 3.1950 0.3034Xfemur length (mm); 99% prediction interval /- 4.6 mm). In three cases of renal agenesis the renal arteries were not imaged with color Doppler sonography. color Doppler imaging may be useful to identify the location of the renal arteries as they originate from the aorta. Using the data from this study, the location of the renal arteries can be determined for the corresponding femur length. If the renal arteries are not identified within the expected range ( /- 99% prediction interval) in fetuses in whom the kidneys are not identified with real-time ultrasonography, renal agenesis must be considered.
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23/35. Fetal renal maldevelopment with oligohydramnios following maternal use of piroxicam.

    A female neonate, born by cesarean section at 37 weeks of gestation, presented with respiratory distress syndrome, right pneumothorax and anuria. A sonogram showed increased echogenicity, with neither hydronephrosis nor macroscopic cysts. peritoneal dialysis was started on the 14th day because of renal insufficiency, but the newborn died on the 33rd day. family history was unremarkable, except that the mother received piroxicam at about the 26th week of gestation. A sonogram at the 28th week showed oligohydramnios. Histopathological study of the kidneys revealed crowded glomeruli and only few differentiated proximal convoluted tubules in the inner cortex, abnormally differentiated microcystic tubules and microcystic glomeruli in the outer cortex. periodic acid-Schiff staining showed only traces of brush border in the dilated tubules of the outer cortex. Immunoperoxidase staining for epithelial membrane antigen was positive in the luminal border of all tubules. Electron microscopy confirmed the presence of brush border remnants and other proximal tubular characteristics in some segments. The renal abnormality bears some similarities to that found in familiar renal tubular dysgenesis, but it fits better with those described after maternal use of angiotensin converting enzyme inhibitors and nonsteroidal anti-inflammatory drugs. The lesion in this case appears to have resulted from fetal exposure to piroxicam. Recently, a second pregnancy ended in a completely normal female newborn.
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24/35. Unilateral renal hypoplasia and contralateral renal agenesis: a new association with 45,X/46,XY mosaicism.

    The association of urinary anomalies with Turner's syndrome is well established. This report describes an unusual antenatal presentation of this cytogenetic disorder and the first reported case of unilateral renal hypoplasia and contralateral renal agenesis in a 45,X/46,XY fetus. The fetus presented with severe third trimester oligohydramnios and symmetrical intrauterine growth retardation at 29 weeks and 2 days' gestation. Chorionic villus cultures revealed a 45,X karyotype. A phenotypically male infant weighing 1833 g was delivered at 35 weeks and 2 days. Chromosomal analysis of the newborn showed a 45,X/46,XY mosaicism, and surgical exploration revealed absence of the left kidney and a hypoplastic right kidney. The infant died at 11 months of age from renal failure and peritonitis. This case demonstrates that monosomy X may be encountered in fetuses with marked growth delay and oligohydramnios. The etiology of the oligohydramnios in this case was a fetal renal malformation not previously described in Turner's syndrome. Antenatal cytogenetic findings should be confirmed postnatally, with a search for mosaicism, when monosomy X is encountered.
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25/35. The early prenatal sonographic diagnosis of renal agenesis: techniques and possible pitfalls.

    Out of 13,252 cases in which fetal bilateral echogenic kidneys were detected by transvaginal sonography between 12 and 18 weeks' gestation, there were nine fetuses where oval hypoechogenic masses were detected in the renal bed. In five fetuses where hypoechogenic masses in the renal bed were sonographically visualized, postabortal examination was compatible with renal agenesis and the hypoechogenic masses proved to be enlarged adrenals. In three additional cases, unilateral renal agenesis was accompanied by unilateral enlarged adrenals, radiologically confirmed postnatally. In one case, a false-positive sonographic diagnosis of Potter syndrome was made because of bilateral hypoechogenic masses in the renal bed. Postabortal examination detected hypoplastic kidneys, but of normal histology, in a dyskaryotic fetus with trisomy 22. In four cases of renal agenesis, the amniotic fluid was of normal volume until the 17th week. In two of the five cases of Potter syndrome, a cystic structure, compatible with the urinary bladder, was detected in the pelvis at 14 weeks. The diagnostic criteria for renal agenesis in the early fetus differ from those used in the second half of gestation.
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26/35. Diagnostic transabdominal amnioinfusion in a case of anhydramnion and fetal kidney dysplasia.

    Diagnostic transabdominal amnioinfusion was performed at 32 weeks of gestation in a case of anhydramnion. Before saline infusion, no exact fetal structures could be identified by sonography. Also, there was a total diastolic block in the fetal aorta (FA) and umbilical artery (UA). After infusion of 270ml of saline, the diastolic blocks in the FA and UA disappeared, and, after another infusion of 410ml of saline, severe fetal congenital kidney abnormalities were identified. Transabdominal amnioinfusion is a practical aid to fetal sonography in severe oligohydramnion and anhydramnion.
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keywords = kidney
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27/35. Potter sequence and consanguinity--a case report.

    The first child of first-cousin parents had Potter sequence, including rudimentary, dysplastic kidneys and pulmonary hypoplasia. The girl died after 5 h. During the next pregnancy, early fetal ultrasound was normal. In the 33rd week, however, ultrasound revealed oligohydramniosis and reduced renal size. After 40 weeks of gestation a healthy girl of normal weight without any stigmata was born. Her pulmonary function and X-ray were normal. Renal ultrasound demonstrated small kidneys with high echogenicity. There was a transient renal insufficiency with a peak serum creatinine of 160 mumol/l. At 5 months of age the infant still has an increased serum creatinine concentration of 57 mumol/l and an inulin clearance of 29 ml/min per 1.73 m2. In this case there is a possibility of an autosomal recessive inheritance.
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28/35. Bilateral renal agenesis and cytomegalovirus infection in a case of fraser syndrome.

    A case of fraser syndrome diagnosed prenatally is presented. Detection of oligohydramnios, hydrops fetalis and bilateral absence of the kidneys were the initial findings leading to further study. Specific IgM for cytomegalovirus in maternal serum and confirmed infection by fetal blood sampling was an associated finding. The importance of an etiologic diagnosis of nonimmune hydrops and the relevant aspects of genetic counselling are emphasized. The association of the fraser syndrome with cytomegalovirus infection has not been previously reported.
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29/35. prenatal diagnosis of fetal pelvic kidney. A case report.

    Published experience on prenatal diagnosis of pelvic kidney is limited. We present the ultrasonographic characteristics of a case of ectopic pelvic kidney and discuss the significance of prenatal diagnosis.
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keywords = kidney
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30/35. polyhydramnios-oligohydramnios in a twin pregnancy complicated by fetal glomerulocystic kidney disease.

    polyhydramnios and oligohydramnios in twin gestation is most often caused by twin-twin transfusion syndrome. Presented is a monozygotic twin pair with polyhydramnios and oligohydramnios, in which both twins had glomerulocystic kidney disease of differing severity. The more severely affected donor twin died of renal failure in the neonatal period. The surviving twin is well following unilateral nephrectomy. This case illustrates the varied spectrum of pathology in glomerulocystic kidney disease.
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ranking = 54.571749802637
keywords = kidney disease, kidney
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