Cases reported "toxoplasmosis, animal"

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1/7. The identification of a rabbit-transmitted cervical toxoplasmosis mimicking malignant lymphoma.

    A case is presented of acquired cervical toxoplasmosis occurring in a 43-year-old male, which clinically mimicked malignant lymphoma. The histopathology of this case was probable toxoplasmic lymphadenitis. serologic tests and the use of FITC-labeled antibodies revealed high levels of specific IgG antibodies in the serum and toxoplasmic antigens in paraffin sections of the patient, respectively. During survey of the infection route, it was learned that the patient's pet rabbit and three other rabbits of the same family line had cervicofacial lumps. The pet rabbit had high levels of toxoplasmic antibodies. Immunofluorescence tests on the infraorbital lump also revealed toxoplasma gondii. Therefore, it was concluded that in this case the rabbit had transmitted toxoplasma to the patient. The authors know of no other reports of toxoplasmosis transmitted by or through rabbit to human. ( info)

2/7. clindamycin for treatment of toxoplasma polymyositis in a dog.

    toxoplasma polymyositis was diagnosed in a dog given repeated injections of glucocorticoids for a skin condition. The dog developed generalized lower motor neuron paralysis, with generalized hyperesthesia, myoglobinuria, and high serum muscle enzyme activities. diagnosis of toxoplasmosis was confirmed by muscle biopsy and seroconversion. Despite supportive care, the dog's neurologic status deteriorated progressively until parenteral treatment with clindamycin was instituted. The dog made a gradual recovery, with complete return of locomotor function and muscle mass within 2 months. ( info)

3/7. Zoonotic diseases of cats.

    As veterinarians, our responsibilities do not end with the care of our patients. The welfare of our clients and their families depends on our ability to detect and control potentially zoonotic diseases in their pets. Because some of these zoonoses can have devastating effects on the development of the unborn fetus or on family health in general, discussion about these diseases between veterinarian and client is often emotionally charged. Under such circumstances, the offering of inaccurate and erroneous information by the veterinarian can have drastic consequences. It is likely that other zoonotic diseases of domestic pets will be identified in the future, especially those that can cause opportunistic infections in debilitated and immunodeficient persons. In the meantime, the potential hazards of cat ownership can be significantly reduced through an increased understanding of feline diseases and an improved level of health care for our feline patients. ( info)

4/7. An outbreak of toxoplasmosis on an illinois farm.

    Two siblings residing on a farm in illinois were concurrently hospitalized with cervical lymphadenitis. Clinical and serologic evidence indicated that the etiology of the lymphadenitis in each case was toxoplasmosis. The simultaneous occurrence of two cases of toxoplasmosis in a household prompted a prospective investigation of 11 other immediate family members for evidence of disease. During the next month seven of the other family members developed serologic evidence of acute toxoplasmosis. Four of these seven individuals were symptomatic. Investigation of the family's home environment failed to unequivocally identify a common source of infection, but epidemiologic evidence suggested that this outbreak was related to contact with sick cats. ( info)

5/7. Acquired toxoplasmosis in children.

    Nine cases of acquired toxoplasmosis, 8 from rural backgrounds, are described, illustrating the wide variety of clinical pictures. Evidence of familial infection was found whenever sought, and was also found in cattle in one case. The availability of an easily performed screening test should make diagnosis of this common disease more frequent. ( info)

6/7. Acquired toxoplasmosis.

    An outbreak of systemic toxoplasmosis occurred in October 1977 in Atlanta, georgia. Thirty-seven people became ill and/or had serologic evidence of acute infection. Epidemiologic study by the Center for disease Control (CDC) suggested that toxoplasma oocysts from infected cats in a riding stable were the source of infection. Aerolization of oocysts or hand-to-mouth contact were the presumptive means of transmission. All patients were examined and followed for over a year, without evidence of toxoplasma retinochoroiditis. In a four-year follow-up study, only one patient had shown evidence of ocular disease. This suggests that at least some sporadic cases are due to acquired toxoplasmosis. ( info)

7/7. Acquired toxoplasmic chorioretinitis.

    A patient developed an acquired toxoplasmic chorioretinitis 11 days after exposure to infected animals. As systemic manifestations of the disease, the patient had rhinitis, sore throat, muscular pains, fatigue, cervical lymphadenopathy, and cardiac symptoms, with highly elevated toxoplasma antibody titers. During treatment with prednisone, the chorioretinal lesion progressed to the macular area, but improvement was seen when pyrimethamine and sulfonamide therapy was started. The importance of this case relates to the unusual occurrence of acquired infection with systemic manifestations in toxoplasmic chorioretinitis and to the possibility of the transmission of toxoplasmosis by trophozoites in excretions. ( info)


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