Cases reported "Arbovirus Infections"

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1/11. Epidemic polyarthritis: a cytological, virological and immunochemical study.

    A patient with epidemic polyarthritis was studied within 48 hours of onset when specific serum antibodies were still low. The synovial fluid showed a fall in hyaluronic acid level and rise in protein levels though the immune globulins, and especially IgM, rose to a lesser degree and remained well below the levels in the serum. Attempts to grow virus from synovial fluid or blood lymphocytes failed despite the use of several new techniques. The complement components C'3 and C'4 were not depleted in serum or synovial fluid. The synovial fluid was devoid of neutrophil leucocytes, and contained predominantly monocytes and macrophages which were remarkable for mitotic and for enhanced and indiscriminate phagocytic activity. From this and other evidence, an explanation is proposed for the cytological response and difficulties in recovering infective virions in virus-induced arthritis. No virus antigen was detected in the supernatant synovial fluid and electron microscopy showed virus-like particles only in cytolysosomes. ( info)

2/11. Arbovirus studies in two towns in western state of nigeria.

    Three hundred and fifty-one persons were tested for HI antibody to arbovirus Groups A, B and Ingwavuma viruses in Ilesha and Oshogbo, two towns in western nigeria. Chikungunya accouted for most Group A infections (39%). antibodies to Group B virus were distributed as follows: dengue 22%, yellow fever 25%, West Nile 28% and Wesselsbron 30%. Few sera 5% were positive to Ingwavuma. No virus was isolated from 188 blood specimens processed for virus isolation. ( info)

3/11. Acute Barmah Forest polyarthritis.

    More than 60 arboviruses have been isolated in australia with the human pathogenicity of the majority yet to be determined. Recent reports outline extensive clinical and sub-clinical infection occurring in Eastern australia by such agents as Ross River and Barmah Forest viruses. We describe the clinical features of two patients with an acute polyarthritis associated with serological evidence of acute Barmah Forest virus infection to alert physicians to this form of viral polyarthritis and highlight the need to consider this agent when confronted with patients with an acute 'viral-type' polyarthritis in whom serology for ross river virus is negative. ( info)

4/11. semliki forest virus: cause of a fatal case of human encephalitis.

    A fatal case of human encephalitis has been observed for which our results indicate that semliki forest virus (SFV) was the etiologic agent. This is surprising in view of the fact that this virus, which has been widely studied, was believed to be one of the arboviruses nonpathogenic for man. Described are the clinical course, the virological examinations performed, and the histopathological findings in the central nervous system. ( info)

5/11. Acute anterior myelitis complicating west nile fever.

    A healthy young man developed acute anterior myelitis resembling the "polio syndrome." He had visited an area (the Gulf of Suez) in which west nile fever is endemic prior to his illness. The course of his febrile illness, the spinal fluid findings, and complement fixation antibody titers established the diagnosis of west nile fever. meningoencephalitis is a serious, and previously the only known, complication of this usually benign febrile illness. This is the first report, to our knowledge, of another neurological complication, namely acute anterior myelitis. ( info)

6/11. Pancreatic islet-cell damage in children with fatal viral infections.

    The pancreases from 250 children with fatal infections caused by at least fourteen different viruses were examined for lesions in the islets of langerhans. Viral cytopathology was found in 4 of 7 cases of Coxsackievirus B infection, 20 of 45 cases of cytomegalovirus infection, 2 of 14 cases of varicella-zoster infection, and 2 of 45 cases of congenital rubella. Destruction of beta cells and acute and chronic inflammatory infiltrates were found in islets from cases with Coxsackievirus B infections. Characteristic inclusion bodies were observed in islets from cases with cytomegalovirus and varicella-zoster infections. This survey provides further evidence that some viruses can infect and damage human beta cells. ( info)

7/11. arbovirus infections among laboratory personnel in Ibadan, nigeria.

    Laboratory-acquired infections encountered between 1963 and 1977 among personnel of the Virus research Laboratory, Ibadan, nigeria, are reported. Two cases of chikungunya infection occurred and one each with Dugbe, Wesselsbron, and dengue viruses. In each case, virus was isolated or development of antibody demonstrated. Among virus and two each to chikungunya and rift valley fever viruses, without experiencing any clinically recognized disease. ( info)

8/11. Epidemic polyarthritis (Ross River) virus infection in the Cook islands.

    An epidemic of ross river virus infection occurred in the Cook islands early in 1980 and affected the majority of the inhabitants of Rarotonga, the most populated island in the group. This represents the easternmost extension of the virus which, until 1979, was believed limited to australia, new guinea, and the Solomon islands. The clinical manifestations of Ross River disease, predominantly polyarthritis, did not differ significantly from those observed previously in australia. However, unlike the experience in australia, where ross river virus has never been isolated from a patient with polyarthritis, the agent was recovered from the serum of one-half of approximately 100 such patients with serologically proven infections. It is not known if this latter observation is the result of a change in the virus, the different virus isolation technique employed, or other factors. It was found that the incubation period of the disease could be as short as 3 days--much less than previously suspected. ross river virus was isolated from six pools of aedes polynesiensis mosquitoes collected in nature and it appeared that this species was the most probable vector on Rarotonga. In view of the widespread distribution of Ae. polynesiensis on islands, in the eastern Pacific it would not be surprising if ross river virus occurs in other previously unaffected areas in the future. ( info)

9/11. Epidemic polyarthritis acquired in fiji.

    Four cases of epidemic polyarthritis which was acquired in fiji between April and July of 1979 are described. The manifestations of the illness were similar to those of the disease in australia. knowledge of the geographic distribution of this disease can be valuable in diagnosis, but it is likely that its range is not yet fully known. ( info)

10/11. Arboviral disease--united states, 1994.

    arboviruses are mosquitoborne and tickborne agents that persist in nature in complex cycles involving birds and mammals, including humans. Characteristics of arboviral infection include fever, headache, encephalitis, and sometimes death. In 1994, health departments in 20 states reported 100 presumptive or confirmed human cases of arboviral disease to CDC. Of these, 76 were california (CAL) serogroup encephalitis; 20, St. Louis encephalitis (SLE); two, western equine encephalomyelitis (WEE); one, eastern equine encephalomyelitis (EEE); and one, Powassan encephalitis (POW). This report summarizes information about arboviral disease in the united states during 1994. ( info)
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