Cases reported "miosis"

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1/22. horner syndrome associated with implantation of a vagus nerve stimulator.

    PURPOSE: To report a case of horner syndrome that occurred after implantation of a vagus nerve stimulator. methods: Case report. RESULTS: A 6-year-old female with cerebral dysgenesis and intractable partial seizures presented with horner syndrome after vagus nerve stimulator implantation. CONCLUSION: horner syndrome can occur as a result of the vagus nerve stimulator implant procedure and should be included as one of its possible surgical complications. ( info)

2/22. Horner's syndrome and dissection of the internal carotid artery after chiropractic manipulation of the neck.

    PURPOSE: To report a case of Horner's syndrome and dissection of the internal carotid artery after chiropractic manipulation of the neck. methods: Case report. A 44-year-old woman with no prior ocular or vascular history presented with severe right-sided head and neck pain, ptosis, and miosis following chiropractic treatment for a strained right shoulder muscle. RESULTS: magnetic resonance angiography of the neck and brain revealed a dissection of the right internal carotid artery as well as a suggestion of subtle dissection in the right vertebral artery. No significant brain abnormalities were noted on magnetic resonance imaging. Pharmacological testing was consistent with preganglionic oculosympathetic damage. CONCLUSION: Acute, painful Horner's syndrome as a manifestation of vascular dissection may be associated with chiropractic manipulation of the neck. ( info)

3/22. Bilateral neurotrophic keratopathy complicating Vidian neurectomy.

    PURPOSE: To present a case of bilateral neurotrophic keratopathy with miosis after bilateral Vidian neurectomy for vasomotor rhinitis. methods: Observational case report. RESULTS: A 68 year-old man presented with irritation and blurred vision in both eyes 2 weeks after bilateral Vidian neurectomy. Slit-lamp examination revealed a large epithelial defect, typical of neurotrophic keratopathy, in the inferior two-thirds of cornea in both eyes. Corneal sensitivity test with a Cochet-Bonnet anesthesiometer and electrical study of the blink reflex indicated bilateral trigeminal dysfunction. Both pupils were miotic, and tests with hydroxyamphetamine 1% and epinephrine 0.1% showed postganglionic sympathetic nerve damage. The corneal epithelial defects healed after 2 months of treatment with systemic prednisolone, vitamin B(12) and tarsorrhaphy. CONCLUSION: Neurotrophic keratopathy associated with dry eye syndrome may be a serious complication of Vidian neurectomy. ( info)

4/22. basilar artery aneurysm with autonomic features: an interesting pathophysiological problem.

    Unruptured cerebral aneurysms often present with neuro-ophthalmological symptoms but ocular autonomic involvement from an aneurysm of the posterior circulation has not previously been reported. A patient is described with a basilar artery aneurysm presenting with headache and unilateral autonomic symptoms. After angiographic coiling of the aneurysm there was a near complete resolution of these features. The relevant anatomy and proposed mechanism of autonomic involvement of what may be considered--from a pathophysiological perspective as a secondary trigeminal-autonomic cephalgia--is discussed ( info)

5/22. Atypical presentation of risperidone toxicity.

    A new class of antipsychotic medication, the "atypical antipsychotics" are gaining popularity. We report 2 cases of toxicity associated with 1 of these new antipsychotics, risperidone. Both patients presented with fatigue, orthostasis, dry mucous membranes, and the unusual finding of miosis. The pathophysiology and clinical presentation of overdose with this agent are discussed. ( info)

6/22. spasm of the near reflex associated with head injury.

    spasm of the near reflex is characterized by intermittent miosis, convergence spasm and pseudomyopia with blurred vision at distance. Usually, it is a functional disorder in young patients with underlying emotional problems. Only rarely is it caused by organic disorder. We report a patient who developed convergent spasm associated with miosis after head trauma at the age of 84 years. ( info)

7/22. Autosomal dominant congenital miosis with megalocornea.

    A family with AD congenital miosis is presented. The ocular symptoms were: megalocornea, iris translucency, microcoria with poor pupillary dilatation and goniodysgenesis with anterior insertion of the iris. This observation confirms that in congenital miosis abnormal development of the whole anterior eye segment may occur. The patients have an increased risk to develop glaucoma. If retinoscopy is impossible due to pin-point pupils, ultrasonic biometry to determine the axial length is recommended. An optical iridectomy could improve visual performance at low illumination; the complaints of photophobia, which are related to the iris translucency, persist. ( info)

8/22. Familial myopathy with tubular aggregates associated with abnormal pupils.

    The authors describe familial tubular aggregate myopathy associated with abnormal pupils. Four family members from two generations had myopathy and pupillary abnormalities. The myopathologic findings consisted of tubular aggregates in many fibers but predominantly type I fibers. ( info)

9/22. Small pupil phacoemulsification using iris sutures: a new method?

    A case is presented in which an alternative surgical method for managing poorly dilating pupils preoperatively or intraoperatively was employed. The technique uses iris sutures, is simple, safe, easily reproducible, and reversible, and does not require new devices for pupil enlargement. This new surgical method is appropriate for extracapsular cataract extraction and phacoemulsification or posterior segment surgeries, and maintains pupillary appearance and function postoperatively. Although other techniques are generally available, this technique may be useful in unique situations where other pupil devices or methods are not available. ( info)

10/22. Raeder's syndrome after embolization of a giant intracavernous carotid artery aneurysm: pathophysiological considerations.

    We present the case of a 47 years old woman submitted to an endovascular trapping of a left cavernous internal carotid artery aneurysm, in which the distal balloon was inflated, as usually done, within the cavernous segment of the internal carotid artery, different from the proximal one which was inflated inside the carotid canal due to technical problems. Consequently, a clinical picture of Raeder's paratrigeminal neuralgia took place. This is the first case report in the literature with theses characteristics. A review of the anatomic pathways and further considerations about the possible pathophysiological mechanisms involved are presented. ( info)
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