is there a link between cholelithiasis and vitamin b12 deficiency?
Just wondering if there is a link between cholelithiasis (gallbladder disease) and vitamin B12 deficiency?
I had my gallbladder out almost 12 months ago and have now found out i am deficient in b12. Just wondering if there is a link!
If there is a link do they do some kind of test to confirm or do they just assume?
My gp has referred me to a gastroenterologist and I am just curious about how things might go
B12 is also important in maintaining the nervous system. Nerves are surrounded by an insulating fatty sheath comprised of a complex protein called myelin. B12 plays a vital role in the metabolism of fatty acids essential for the maintainence of myelin. Prolonged B12 deficiency can lead to nerve degeneration and irreversible neurological damage.
When deficiency occurs, it is more commonly linked to a failure to effectively absorb B12 from the intestine rather than a dietary deficiency. Absorption of B12 requires the secretion from the cells lining the stomach of a glycoprotein, known as intrinsic factor. The B12-intrinsic factor complex is then absorbed in the ileum (part of the small intestine) in the presence of calcium. Certain people are unable to produce intrinsic factor and the subsequent pernicious anaemia is treated with injections of B12.
Vitamin B12 can be stored in small amounts by the body. Total body store is 2-5mg in adults. Around 80% of this is stored in the liver.
Vitamin B12 is excreted in the bile and is effectively reabsorbed. This is known as enterohepatic circulation. The amount of B12 excreted in the bile can vary from 1 to 10ug (micrograms) a day. People on diets low in B12, including vegans and some vegetarians, may be obtaining more B12 from reabsorption than from dietary sources. Reabsorption is the reason it can take over 20 years for deficiency disease to develop in people changing to diets absent in B12. In comparison, if B12 deficiency is due to a failure in absorption it can take only 3 years for deficiency disease to occur. (+ info
What % of people with rapid weight loss develop cholecyctitis or cholelithiasis?
If I remember it around 32 %. (+ info
Please help me making nursing care plane for a client who has cholelithiasis?
nursing diagnosis- Subj. pain, nausea, pyrosis
1. alteration in comfort secondary to blockage of bile ducts-
Medicate for pain and nausea
2. alteration in fluids secondary to vomiting or nausea, or pain meds medicate with anti nausea drug replace fluids
3 alteration in nutrition secondary to nausea -control nausea
4. alteration in body temp secondary to inflammation of gallbladder-give antipyrexia meds
Evaluate to see if patients expected comfort level is maintained. (+ info
My radiologist diagnosed me with cholelithiasis, but my gall bladder was removed 4 years ago? How can it be?
The gall bladder is just a storage device for bile. If it is removed, the bile flows directly into your duodenum. Bile is continuously excreted from your liver and flows through a number of ducts. These ducts can become affected by inflammation or very small gall stones can be formed and block the flow of bile in that duct. Therefore, these stones will show during examination by x-rays. Normally this can be remedied by a special diet and some specific medication without the need for another surgical procedure. However, you should discuss this with your physician/radiologist. (+ info
Why might cholelithiasis develop after rapid weight loss?
like if yuou have gastic bypass and loose a lot of weight
wow,i googled this,and a lot of articles came up:gallstones develop after rapid weight loss!
It does not have to be gastric bypass,any diet causing 10-30lbs or more weight loss is likely to result in development of gallstones.
there are a couple theories,either the bile is sitting in the gallbladder or the concentration changes?
the result is,gallstones. (+ info
what are the nursing diagnosis for patients suffering from cholelithiasis?
the diagnosis cholelithiasis is a doctor's diagnosis...
nursing diagnosis describes the symptoms or things that can happen secondary to the actual diagnonsis...
some examples are:
nausea related to cholelithiasis
pain secondary to cholelithiasis
imbalanced nutrition related to cholelithiasis (+ info
what are risk factors of cholelithiasis and why they acquire it? what are the signs and syptoms and why?
it is about the gallstones
The gallbladder is a small pouch that sits on top of the liver where excess bile is stored. Bile is a substance that the body uses to help digest fatty foods.
Gallstones occur when the bile in the gallbladder becomes over concentrated (they precipitate out like the salt crystal lab experiments done by children in grade school) and form "stones" or crystals in the gallbladder or bile ducts.
Gallstones are predominantly of two types: 1) cholesterol (80%) and 2) Calcium bilirubinate (20%). When the crystals are located in the bladder itself, the condition is called cholelithiasis; when they are located in the bile ducts, the condition is called choledocholithiasis.
Pain in right upper abdomen
Common in forties (can occur at any age)
Sickle Cell Anemia
Inflammatory Bowel Disease
Short gut syndrome
Prosthetic heart valves
If there are no symptoms, treatment is unnecessary, except if the patient is diabetic. Diabetics with gallstones usually need them removed because they are at risk for sudden, severe infections and perforations of the gallbladder wall.
If you have symptoms of recurrent severe pain despite diet changes, and diagnosis has been confirmed through X-Ray or ultrasound:
o Laparoscopic cholecystectomy may be performed in which the gallbladder is removed via a special technique using a camera scope inserted through an incision in the abdominal wall; or via open laparotomy (open surgical procedure).
Ursodeoxycholic acid-a medication that dissolves some cholesterol stones. It usually works very slowly (two years or longer with side effects, and therefore is rarely used).
Chenodial and other medications are also being used to dissolve the stones.
Sonic Shock waves
Cholecystitis/cholangitis-gallstones cause inflammation and infection of the gallbladder or ducts leading to the gallbladder. (See cholecystitis/cholangitis section.)
Pancreatitis-gallstones may pass though ducts into pancreas, causing blockage and inflammation. (+ info
pathophysiology of cholelithiasis?
can anybody help me with the correct and complete pathophysiology of cholelithiasis?? thanks.. i really need it..
what to avoid when you have cholelithiasis?
Treatment for simple cholelithiasis is symptomatic.
One option for nonsurgical management of gallstone disease is the use of ursodeoxycholic acid. One study demonstrated a 56% reduction in biliary pain after 3 months of therapy and a mean dissolution of gallstones in 59% of cases after 12 months of treatment with 10 mg/kg/d of ursodeoxycholic acid. The primary disadvantage with this approach is the incidence of recurrent gallstones, approximately 25% within 5 years. The nonsurgical option is currently only indicated for patients either unfit or unwilling to undergo surgical intervention and has not been recommended in the pediatric population.
The number of pediatric cholecystectomies in symptomatic patients has increased.
Removal of a stone in an asymptomatic patient is not standard practice.
Admission may be required for observation with nasogastric tube placement.
The number of gallbladders removed in patients with symptomatic stones is increasing.
Removal of the gallbladder in those who are asymptomatic is not the standard treatment.
Laparoscopic cholecystectomies are now being routinely performed in children and are accepted as the criterion standard for the treatment of symptomatic cholelithiasis. Laparoscopic cholecystectomy with intraoperative cholangiography has demonstrated promise as an alternative to ERCP in patients with obstructive common bile duct stones (choledocholithiasis).
Endoscopic cholecystotomy has been demonstrated to be ameliorating.
Consultation with a general surgeon is appropriate in symptomatic patients with stones and evidence of cholecystitis.
A decrease in the consumption of fatty foods and controlled reduction in weight may be effective in some patients.
Leitzmann et al have demonstrated in a prospective cohort study that symptomatic gallstones in men were reduced by approximately 20% with increased exercise. This reduction may be extrapolated to the pediatric population. (+ info
what activities should i do when im diagnosed w/ cholelithiasis?exercise?
Once you have stones, exercise will not get rid of them. Sugery is called for. (+ info
We do not evaluate or guarantee the accuracy of any content in this site. Click here for the full disclaimer.