FAQ - Epidermodysplasia Verruciformis
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What year and where did epidermodysplasia verruciformis first appear?


It's genetic.  (+ info)

How many cases of Epidermodysplasia verruciformis are there in the world?

Or atleast an estimate of how many cases.
LOL. Sure - you can try again later. =D
No. I didn't "stumble" across a this. I've only ever heard it referred to as "tree disease" and have only also "seen" one particular person who suffers from it.

This is a very rare disease you know. If I had to choose, I would say that no more that 10.000 people have that, right now.  (+ info)

Need some info about epidermodysplasia verruciformis (treeman)?

I'm reading about the information on epidermodysplasia veruciformis and it says how its caused by receiving a rare autosomal recessive inherited disorder. And then it says something about the human papillomavirus. Which causes it? Is the genetic disorder leaving you vulnerable to the virus?

Your last sentence encapsulates the cause perfectly. Patients with EV have a defective cell-mediated immune response to HPV infection. I shall briefly discuss this. Epidermodysplasia verruciformis (EV) is a rare inherited disease. It is a lifelong, viral-mediated, autosomal recessive disorder affecting the skin. Genes come in pairs. Recessive inheritance means both genes in a pair must be defective to cause disease. People with only one defective gene in the pair are considered carriers. However, they can pass the abnormal gene to their children. Autosomal dominant inheritance patterns have also been reported - Dominant inheritance means an abnormal gene from one parent is capable of causing disease, even though the matching gene from the other parent is normal. The abnormal gene "dominates" the pair of genes. If just one parent has a dominant gene defect, each child has a 50% chance of inheriting the disorder. The disease is characterized by chronic infection with human papillomavirus (HPV). Widespread skin eruptions of flat-to-papillomatous, wartlike lesions and reddish-brown pigmented plaques on the trunk, the hands, the upper and lower extremities, and the face are typical. Many of the HPV types found in EV lesions are nonpathogenic to the general population. Cells with early signs of malignant transformation have been found closely connected with virus-infected epidermal regions. The exact mechanisms involved in the malignant transformation of keratinocytes in skin lesions of patients with EV are still unclear. Studies have shown that interactions occur between oncogenic HPVs and antioncogene proteins, such as p53 and pRb, in cell cycle regulation, DNA repair, and the execution of programmed cell death (apoptosis).


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Hope this helps
matador 89  (+ info)

Worst desease possible?

What do you think is the worst desease someone can get. to me i think it would be "epidermodysplasia verruciformus" which is caused by HPV but i would like to know your opinion.

HIV... Do I need to say more?????  (+ info)

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