In Respiratory acidosis is there hypoventilation or hyperventilation? what is the rationale?
The major sign of hypoventilation is?
b. increased airway resistance
I may be looking to far into this question. The best indication of ventilation (with out a doubt) is PaCO2 or carbon dioxide in the arterial blood. To much is hypercapnia or low ventilation. This is a blood test result.
Signs are what someone can SEE and symptoms are what someone can FEEL.
If you consider a blood test as a sign, then C is your answer.
If you do not consider a blood test a sign then A, cyanosis is your answer.
I hope I didn't confuse you. (+ info
Racing / abnormal heartbeat - What does this sound like to you? hypoventilation?
I have been using alcohol too much for the past couple years and about three weeks ago went on a spree of taking xanax (I think....pill said mylan) and also aderol...I have since stopped but am experincing a racing heartbeat....sometimes it feels like my heart stops or beats real slow and suddenly beats real hard and fast....I catch myself breathing real shallow...I get dizzy when it happens. The closest I can find is something called 'hypoventilation'.....is this something that will go away or do I need to see a doc?
Hypoventilation is when you are breathing too slow and your body is taking in less oxygen. Now to answer your other question, you should never mix heart medications with xanax without speaking with your doctor first. Or you could just be in withdrawals. You are experiencing a high level of anxiety from not taking the xanax. You should not stop taking xanax abruptly because it will make you feel nervous, anxiety, and an increase in blood pressure. If you have a problem quitting them, you may want to seek treatment. (+ info
How does CNS depression cause respiratory alkalosis?
Now as far as I knew, CNS depression caused a decreased respiratory rate and would therefore cause hypoventilation and would therefore cause respiratory acidosis, however my sources say that CNS depression causes hyperventilation and therefore is a cause of respiratory alkalosis. Can anyone explain the pathophysiology of this to me so I can understand it?
Yes, quite simply. Your sources are wrong. (+ info
Otology: Complexing question for any medical practicioners and/or medical researchers.?
Is it possible for this string of reactions to occur in the human body over a long period of time (3 years) :
Eustachian tube dysfunction -> Middle ear Inflammation -> Hypoventilation -> Hypercapnia -> Acidosis
middle ear infllammation doesnt lead to hypoventilation,unless severe septicaemia and another complication arises (+ info
Respiratory acidosis or alkalosis?
Would hypoventilation lead to respiratory acidosis or respiratory alkalosis?
Normally it would lead to respiratory alkalosis,
but remember blood gases are tricky if the Base Excess is off or the HCO3 is off then the metabolic side may outweigh anything the respiratory system can do (+ info
question for RRT's?
what would your decision be if a COPD patient suffered from smoke inhalation? i know that for a regular person, you would put the patient on a 100% non-rebreather (if there is no hyperbaric chamber around). but in my head, im still debating on what to prioritize when it comes to COPD. if we put the patient on 100%, this can trigger hypoventilation because of air-trapping, but if we don't put them on 100%, he can suffer from carbon monoxide poisoning.
what would your decision be? in school, i was taught that ventilation comes first before oxygenation. i just thought this topic is worth debating about... it's acute versus chronic.
RRemember that COPD encompasses several chronic conditions such as asthma, bronchitis and emphysema. The only sub-group of COPD'ers that this would really be an issue with is that group of patient's that are near end stage the CO2 retainers. Air trapping would only be an issue after approx. 48 hours due to nitrogen washout resulting in atelectasis.
Because of the CO2 affinity for the hemoglobin providing a high concentration of O2 is necessary in cases of carbon monoxide/carbon dioxide poisoning associated with smoke inhalation (other issues heat trauma, toxic fumes, etc.). So a high concentration of O2 over rules the possibility of hypoventilation. If the patient isn't oxygenated you can pump all the air into the lungs you want but without freeing up the hemoglobin through some method (hyperbaric, positive press. ventilation or high O2 concentration) just plain ventilation isn't going to do the patient any good. (+ info
(5). Carbon dioxide can be carried in the blood:
A. Bound to hemoglobin as a compound called “carbaminohemoglobin”.
B. As a dissolved gas in the blood plasma.
C. As the bicarbonate ion.
D. All of the above.
(6). What would be the consequence of an increase in the level of carbon dioxide in the
A. The rate and depth of breathing would increase.
D. All of the above.
(7). The neurons that control rhythmic breathing are found in ___________
A. The hypothalamus.
B. The hippocampus.
C. The cerebral cortex.
D. The medulla oblongata.
(8). Blood in the systemic arteries has high levels of ___________ and low levels of
_____________ compared to blood in the systemic veins.
A. Carbon dioxide. Oxygen.
B. Oxygen. Carbon dioxide.
C. Nitric oxide. Oxygen.
D. Carbon monoxide. Oxygen.
6. A The main mechanism for your body to get rid of carbon dioxide is to breath it out. If there is an excess in the blood stream, your brain causes you to breath faster to get rid of it.
7. A The hypothalamus is responsible for regulating most of your body's automatic functions.
8. B The arteries job is to deliver oxygen from the lungs to the body tissues. Muscle and other cells use oxygen from the blood, and pass carbon dioxide into the blood. Veins are oxygen poor, with much more carbon dioxide than the arteries. (+ info
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