FAQ - Leishmaniasis, Cutaneous
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what are cutaneous leukemic infiltrates?


What would this look like?
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It is when the cancer has invaded the skin. It is usually red, sometimes ulcerated, unhealing sores.  (+ info)

i recently went to houndourous and got 63 san fly bites what are the chances i will contract leishmaniasis?


  (+ info)

Dilated blood vessels increase perfusion and heatloss, while constricted vessels greatly reduce cutaneous bloo?


Dilated blood vessels increase perfusion and heatloss, while constricted vessels greatly reduce cutaneous blood flow and conserve heat.!!!


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explain please?!!!
it has something to do with skin cells and temperature regulation ..
i found it on wikipedia under FUNCTIONS :)
thankss

Future Mrs. Bieber
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Most of the body heat is produced in the liver, major muscles and brain. The blood transports this heat from these organs to the peripheral regions to be lost to the environment. If and when peripheral blood vessels become more dilated more warm blood is transported to the peripheral regions to lost to the environment. If the blood vessels were constricted less heat would be lost.

Conclusion: In hot weather peripheral blood vessels become dilated and the body loses more heat. Net effect: Body becomes cooler.
In cold weather there is peripheral vasoconstriction. This reduces the movement of warm blood to the periphery. So heat is conserved.  (+ info)

What is the cure for leishmaniasis?


Is it cureable, how do you find a compentent physician
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From wiki:
There are two common therapies containing antimony (known as pentavalent antimonials), meglumine antimoniate (Glucantim®) and sodium stibogluconate (Pentostam®). It is not completely understood how these drugs act against the parasite; they may disrupt its energy production or trypanothione metabolism. Unfortunately, in many parts of the world, the parasite has become resistant to antimony and for visceral or mucocutaneous leishmaniasis,[2] but the level of resistance varies according to species.[3] Amphotericin is now the treatment of choice[4]; failure of AmBisome® to treat visceral leishmaniasis (Leishmania donovani) has been reported in Sudan,[5] but this failure may be related to host factors such as co-infection with HIV or tuberculosis rather than parasite resistance.

Miltefosine (Impavido®), is a new drug for visceral and cutaneous leishmaniasis. The cure rate of miltefosine in phase III clinical trials is 95%; Studies in Ethiopia show that is also effective in Africa. In HIV immunosuppressed people who are coinfected with leishmaniasis it has shown that even in resistant cases 2/3 of the people responded to this new treatment. Clinical trials in Colombia showed a high efficacy for cutaneous leishmaniasis. In mucocutaneous cases caused by L.brasiliensis it has shown to be more effective than other drugs. Miltefosine received approval by the Indian regulatory authorities in 2002 and in Germany in 2004. In 2005 it received the first approval for cutaneous leishmaniasis in Colombia. Miltefosine is also currently being investigated as treatment for mucocutaneous leishmaniasis caused by L. braziliensis in Colombia,[2] and preliminary results are very promising. It is now registered in many countries and is the first orally administered breakthrough therapy for visceral and cutaneous leishmaniasis.[6](More, et al, 2003). In October 2006 it received orphan drug status from the US Food and Drug administration. The drug is generally better tolerated than other drugs. Main side effects are gastrointetinal disturbance in the 1-2 days of treatment which does not affect the efficacy. Because it is available as an oral formulation, the expense and inconvenience of hospitalisation is avoided, which makes it an attractive alternative.

More info here:
http://en.wikipedia.org/wiki/Leishmaniasis#Treatment  (+ info)

What is leishmaniasis? and how do i get checked? Can i die from it?


go to this website : http://www.cdc.gov/ncidod/dpd/parasites/leishmania/factsht_leishmania.htm

It has alot of info, and from what I've read you won't die from it. Just get it treated. Hope I helped a little!  (+ info)

Has anyone heard of Primary Cutaneous t-cell lymphoma?


if yes, would you know where I can find some info. I did a google search and very little info came up.
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You don’t often see this kind of lymphoma.
You are probably using too many words in your search try just “T-cell lymphoma”.
I have attached a link for information intended for patients and their families.


http://www.leukemia.org/hm_lls  (+ info)

Have you ever had cutaneous leishmaniosis? If so, how did you threat it and did it ever come back?


Nope... Never had it. It's cutaneous leishmaniasis caused by a parasite under your skin.

An ointment comprising of 15% paromomycin sulphate (PR)
and 12% methylbenzethonium chloride (MBCl)
in soft white paraffin (SWP) was developed.
This ointment was proven to be highly effective against a variety of leishmanial
strains both in human and in several animal models.

It is presently manufactured by Teva Pharmaceutical Industries
(P.O.Box 1142, Jerusalem 91010, Israel. Tel: 972-2-5892811; Fax: 972-2-5814345)
under the trade name "Leshcutan"  (+ info)

malignant cutaneous adnexal tumor...it's rare, i got it. what is it and what's the treatment?


it's some kind of rare cancer but no one can tell me much about it. they say it's technically a skin cancer but it was a tumor the size of a golf ball they took out. i'm not sure what course to take. the doctors don't seem to agree on what treatment to use. help please.
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It looks like the treatment is removel of the tumor. Did they do a sentinel node biopsy?  (+ info)

I have a very ugly cutaneous sarcoidosis lesion on my face dermotolgist told me makeup i'm suicidal HELP?


I NEVER WANT TO LEAVE THE HOUSE ANYMORE BECAUSE OF THIS
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My mum used cicatrix cream on her sarcoidosis scars and whilst it didn't remove them completely it did reduce them considerably. She got it on prescription but that was many years ago, it could be available over the counter by now.  (+ info)

what is a lateral femoral cutaneous nerve?


and how can it be treated
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Meralgia paresthetica is the term that describes a painful mononeuropathy of the lateral femoral cutaneous nerve (LFCN).
It is an entrapment neuropathy (pinched nerve) that develops as the nerve passes through the inguinal ligament. It may be due to direct trauma, stretch injury, or loss of blood flow to the nerve. The clinical history and examination is usually enough to make the diagnosis. The LFCN is a purely sensory nerve and is responsible for sensation of the anterolateral thigh. The LFCN has no motor component.

The LFCN originates from the lumbar plexus (with root innervation from L2-3). The nerve runs through the pelvis along the lateral border of the psoas muscle to the lateral part of the inguinal ligament. Here, it passes to the thigh through a tunnel formed by the lateral attachment of the inguinal ligament and the anterior superior iliac spine. This is the most common site of entrapment.



When meralgia paresthetica develops, it causes tingling and numbness of the upper outer thigh area. The tingling can be quite uncomfortable.

Symptoms are usually on one side only. Walking or standing may aggravate the symptoms, while sitting tends to relieve them.

Examination reveals numbness of the anterolateral thigh in all or part of the area involved with the paresthesias. Sometimes, patients are very sensitive in this area.

Tapping over the upper and lateral aspects of the inguinal ligament or extending the thigh and stretching the nerve may reproduce or worsen the symptoms.

Muscle strength in the involved leg is normal.

Common causes are pregnancy, tight clothing, and obesity.

Meralgia paresthetica is more common in diabetics than in the general population.

Rarely, pinching of the LFCN by masses [eg, neoplasms, iliopsoas hematomas (clots)] in the retroperitoneal space before it reaches the inguinal ligament can cause the same symptoms.

A lumbar disk bulge or herniation should be excluded as part of the work-up.
Most patients with meralgia paresthetica will have mild symptoms that respond to conservative management.
The treatment involves removing the cause of compression, weight loss and wearing loose clothing, if these are suspected.
When the pain is severe, a nerve block can be done at the inguinal ligament with a combination of lidocaine and corticosteroid. This may relieve the symptoms for several days to weeks.
In rare and particularly painful cases that are unresponsive to nerve block, surgical decompression may be warranted.
The numbness and tingling typically resolves over time, but it may persist.
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