Is there any way to treat lipodystrophy in diabetics?
I rotate spots often, but I have to take 4 shots a day. I usually only get these bumps on my arms where I am taking a high dose of insulin.
i don't know many ways to treat it. avoid the place in which lipodistrophy has developed,.
For the rapid acting insulin try the abdominal area (the absorbtion is better, faster, more predictable, and you have more place to rotate), for the intermediate or long acting the best place is the tigh.
A massage of the areas affected can be useful.
When you inject a high dose, the chance of developing lypodistrophy is higher. For doses higher than 30 units I would recommend splitting the dose in 2 and injecting it separately. (+ info
How soon after HIV infection can facial wasting/ lipodystrophy/lipoatrophy occur?
I am just wondering how long it would take for facial wasting (sunken in, hollow checks) to occur after HIV infection.
Can HIV on its own cause hollow cheeks/ after only a few months (2-6 months) of infection?
If not, what else could be the cause of hollow, sunken inn cheeks, that are continuing to become more hollow, be?
It depends on your immune system, how long it can cope until being fully suseptible to facial wasting. People live up to 10 years or more without any way of knowing they had it. (+ info
I have hiv.I have lipodystrophy,around my belly,but even worse around my neck and going into the back of my Hd?
What can i do about this,its getting worse,so worse people asked me about 4 times these week,what it was.
Does anyone know the condition lipodystrophy but not the aids related one?
Not the insulin injection one, thats secondary localized lipodystrophy. I mean congenital primary lipodystrophy.
is it the one related to injecting insulin or and other medication? (+ info
is lipodystrophy harmful to my stomach?
i alwaysget lipodystrophy when i inject insulin.
lipodistrophy can cause a bad absorbtion of insulin if insulin is injected there.
it can cause pain sometimes.
Some people develop it more frequently than others.
The general advice is to rotate the place of injection. When the dose is high the risk of lipodistrophy is higher. Doses higher than 30 UI should be splitted and administered separately.
Apply a gentle massage on the area, some anticelulitic creams may be useful too. If lipodistrophy has developed, avoid the area for a while.
take care (+ info
Congenital Generalized Lipodystrophy?
I am doing a project about a genetic disoder for Biology. I unfortunatley didn't realize that the genetic disorder I chose only had about 2 paragraphs about it on the whole internet (listed below). I would appreciate any links or info not included in the folowing information.
Here two more websites to check out. Look up Berardinelli-Seip congenital lipodystrophy, another name for congeital generalized lipodystrophy(CGL). (+ info
can lipomas cause lipodystrophy?
no. (+ info
I can't get fat... what's my condition?!?
Okay, I can't get fat. AT ALL. I eat tons of stuff, including junk food but I remain 119 lbs. I don't even look fat. I'm not super skinny, I look pretty normal. I know that i don't have Lipodystrophy, which is a medical condition for people who can't gain weight. But... the symptoms don't match up to me, so I know that is not my case. I haven't checked with a doctor because I do not want to pay. Is it that I have a high metabolism? Are there any other conditions like this?
It's called being young, LOL!
Until I was about 22 or 23 I was practically a stick insect (5'7" and about 105lbs) even though I ate loads and never had to watch what sort of food I ate, either.
Then gradually I started putting on weight because my body needed fewer calories and I was in the habit of over-eating.
I'm still not overweight, but at 25 (when I got married) I weighed 135lbs and had gone up several clothes sizes. I seem to have maintained that weight/size since (10 years later) with little effort, but there was a definite slow down of my metabolism in a relatively short space of time. (+ info
How to go about seeking HGH Therapy? / insurance coverage?
I'm wondering how I should go about seeking HGH Therapy,
I just turned 18 , am HIV+ , been since birth got it from my mom..
been taking meds ever since i can remember, been on a 10+ pill daily regime since I was very young...can't say exact time but I say 10+ years of my life..I suffer from lipodystrophy , really started bugging me when i got into my teenage years... + i have trouble sleeping severe,I feel so weak physically & mentally/emotionally..I'm convinced I have a low GH level, my life has been on a severe downward spiral for years now ... I think hgh therapy will help greatly,seems to be the last glimpse of hope for me @ this point ... is my hiv status sufficient to qualify me for hgh therapy ? I have amerihealth mercy insurance.. if not, what conditions are valid qualifications for insurance to cover treatment.. thanks 2 everyone
healthquotes.awardspace.info - here is my health insurance plan. As I remember they can provide such a service. (+ info
Why do so many people oversimplify obesity, which is extremely complex?
Why do people do this - people affiliated with " The Biggest Loser" for example, not to mention numerous media sources?
How do they explain lipodystrophy , a condition where extreme emaciation and obesity manifest itself on the same body? Is that person supposed to "eat less" for only one part of their body? These poeple need to understand that fat cell behavior is regulated in a HEALTHY person. Obesity is a DISEASED STATE OF FAT CELL DYSREGULATION.
Please........ The regulation of mammalian fat cells is NOT well understood by sciience currently.
The people on The Biggest Loser, with their misinformation and oversimplification of a complex diseased state DO NO ONE ANY FAVORS, ESPECIALLY THE OBESE
The science is clear- dieting ( moderate to significant caloric restriction) only RAISES your bodyfat setpoint. It is a FAILURE for the long term treatment of obesity Look into DR LINDA BACON Ph.D.
The majority of people gain excess fat accumulation due to a high carb way of eating. Long term diet of highly refined carbs "burn out" insulin receptors, causing most obese people to become insulin resistant, which makes all calories go directly to fat cell. It's a fairly simple answer though, control carbs, eliminate sugar & starches & accept that your body no longer can process carbs, that there is no returning to a high carb way of eating without refilling fat stores.
There is no better way to bring the body to the state of optimal health than with a low carb way of eating. Low carb doesn't cause high blood pressure, high blood sugar or high cholesterol, it cures it. It is actually dangerous to take meds that lower these levels and do low carb at the same time because the levels will become dangerously low. Carbohydrates trigger insulin. High insulin levels unbalance other hormones. Anything less that 9 grams of carbs per hour controls insulin and is considered low carb (up to 144 grams per day).
U.S. government guidelines were changed 35 years ago to suggest we lower our fat intake & increase our carb intake. American society followed these recommendations & lowered their fat intake by 11% & increased their carb consumption. In this same time frame obesity, diabetes, heart disease are all at epidemic levels. Through their direct effects on insulin & blood sugar, refined carbs are the dietary cause of coronary heart disease & diabetes.
Glucose is the bodies preferred fuel (if you want to get technical, it actually burns alcohol most efficiently, but that doesn't make it any healthier for the body than carbs), the body can convert 100% of carbs, 58% of protein & 10% of dietary fat into glucose. The body can also be fueled by fat (dietary fat & fat cells) but only in the absence of carbs. Your brain actually prefers* to be fueled by ketones (part of the fat burning process), it does require glucose also, but glucose can be easily converted from excess protein if needed or dietary fat.
Plaque build up in the arteries is more attributable to carb consumption than dietary fats, which seems to be the conclusion of the following study. Carb consumption raises triglycerides & VLDL (bad cholesterol). Fats raise the HDL (good cholesterol). High triglyceride levels & low HDL levels are an indicator of plaque & glycation - the precursors to a heart attack & heart disease.
study from the Oxford group examining the postprandial (after-eating) effects of a low-fat vs. low-carbohydrate diet. (Roberts R et al, 2008)
Postprandial lipoproteins, you'd think, would be plentiful after ingesting a large quantity of fat, since fat must be absorbed via chylomicrons into the bloodstream. But it's carbohydrates that figure most prominently in determining the pattern and magnitude of postprandial triglycerides and lipoproteins. Much of this effect develops by way of de novo lipogenesis, the generation of new lipoproteins like VLDL after carbohydrate ingestion.
Gary Taubes who wrote "Good Calories, Bad Calories" spent 7 years going through all the studies over the last century & dividing up the real science from the faulty science & concluded that low carb was the best way to control insulin levels which balances out other hormones & allows the body to function properly.
His main points are:
1. Dietary fat, whether saturated or not, is not a cause of obesity, heart disease or any other chronic disease.
2. The problem is refined carbs in diet, their effect on insulin secretion & the hormonal regulation of homeostasis.
3. Sugars - sucrose, high-fructose corn syrup specifically - are particularly harmful, the combination of fructose & glucose simultaneously elevates insulin levels & overload liver with carbs.
4. Through their direct effects on insulin & blood sugar, refined carbs, starches, sugars are the dietary cause of coronary heart disease & diabetes. They are likely dietary causes of cancer, Alzheimer's & other diseases.
5. Obesity is a disorder of excess fat accumulation, not overeating.
6. Consuming excess calories does not cause us to grow fatter.
7. Fattening & obesity are caused by an imbalance in the hormonal regulation of adipose tissue & fat metabolism. Fat synthesis & storage exceed the mobilization of fat from adipose tissue & its subsequent oxidation.
8. Insulin is the primary regulator of fat storage. When insulin levels fall, we release fat from fat tissue.
9. By stimulating insulin secretion, carbs make us fat.
10. By driving fat accumulation, carbs also increase hunger & decrease the amount of energy we expend in metabolism & physical activity. (+ info
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