FAQ - demyelinating diseases
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What other diseases is associated with demyelinating disease besides MS?


I had a MRI done where they found demyelinating disease and thinks that it's multiple sclerosis I have several of the symptoms for MS I have to get a spinal tap done.
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Besides MS, there are several other demyelinating diseases, such as the leukodystrophies (Adrenoleukodystrophy, Canavan Disease, Krabbe Disease, Pelizaeus-Merzbacher Disease, Cockayne Syndrome, and Alexander's Disease), Marchiafava-Bignami Disease, Central pontine myelinolysis, and Progressive Multifocal Leukoencephalopathy (PML). Among other things, the spinal tap will help to rule out some of the other demyelinating diseases. Some of these are fairly unlikely (for example, Adrenoleukodystrophy occurs when more than 90% of the adrenal cortex is destroyed, and is a very rare condition, so you'd likely have far more symptoms without a functioning adrenal cortex).

The "white spots" visible on an MRI are generally pretty specific for MS; in other words, for certain white spot patterns, only MS produces such patterns on an MRI. That, along with the other tests (blood tests and lumbar puncture) can rule out the other causes of white spots on an MRI.

The referenced site has loads of (mostly medical) information about all of these diseases, but it may be helpful to you. You can look up each disease individually and find some information on them.

Good luck.  (+ info)

What autoimmune diseases are associated with demyelinating conditions?


Have Rheumatoid Arthritis and Fibromyalgia; am experi-
encing changes in ability to walk, which comes and goes,
also tremor, excessive weakness, difficulty with thought
processing which occurs with the weakness. Brain MRI
shows nonspecific pattern of demyelinization. Any clues?
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[Celiac disease in adults revealed by sensory-motor neuropathy]


Viader F, Chapon F, Dao T, Rivrain Y, Lechevalier B.

Service de Neurologie, CHRU de Caen.

Central or peripheral nervous system complications are occasionally observed in adult patients with celiac disease. Several mechanisms have been proposed including vitamin deficiency, vascular inflammation and a direct effect of gluten intolerance. Typical nerve fiber damage due to demyelinization has been suggested. We observed a 65-year old woman with a right peroneal nerve palsy superimposed on a diffuse peripheral neuropathy who was found to have folic acid deficiency which in turn led to the diagnosis of adult celiac disease. Electrophysiological and histological studies demonstrated a predominantly demyelinating peripheral neuropathy which responded first to parenteral folic acid supplementation and second to a gluten-free diet. The mechanisms of peripheral nerve damage in adult celiac disease are briefly discussed and the possible role of folic acid deficiency is emphasized.  (+ info)

Does marijuana help preserve the myelin sheath in demyelinating diseases?


No, I'm telling you this again because well, you will need to hear the answers to questions repeatedly if you smoke marijuana. It hinders your memory and hastens the myelin sheath. Sorry! Ohh yeah and...

No  (+ info)

Does marijuana preserve the myelin sheath in demyelinating diseases?


No, this is a actually a common misconception of people who have multiple sclerosis, but there is no scientific evidence to support these claims. But there is sufficient evidence that it does help decrease symptomatic pain associated with demylinating disorders.  (+ info)

i have chronic demyelinating brain disease confirmed by MRI what precautions should i take ?


so that chronic will not change into acute. neurologist said it is a autoimmune disease and prescribed vitamin B1,B12 and piracetam and exercise also said not to wet your head and do not be more excited.
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Only your neurologist knows your case and can advise you.
Sorry.  (+ info)

Will a demyelinating disease reoccur after several years?


Patient has been treated with steroids (via intravenous) after diagnosis.
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It depends on the disease and the individual. For example, diseases such as Multiple Sclerosis wreak their havoc on a body through demyelination. For some, the disease will progress fast, and for others slow. If steroids are the form of treatment chosen by the physician and patient, then hopefully it should slow down. But demyelination is a continual process.  (+ info)

Does anyone have Chronic Inflammatory Demyelinating Polyneuropathy?


IIt's also known as Chronic Guillian Barre syndrome.It's an autoimmune disease where your immune system mistakes your nerves as foreign bodies and attacks the protective coating of myelin around them. It causes a very wide range of symptoms because it deals with the nervous system and is hard to diagnose because it can only be done after eliminating everything else. Does anybody happen to have this rare disease?
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  (+ info)

What diseases would have the symptom of coughing up blood or blood in the phlegm?


This is not a symptom of my own so please don't tell me to go to the doctor, it is for a piece of work I have to do for College.

If you could tell me the name of a disease/ diseases that cause this, preferably not consumption or TB, a little about it and other symptoms it would be a massive help!
If you could also include treatment options and how serious a disease it is that would also be fantastic!



Hope you can help! Thanks!
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First: spitting up blood is clinically known as: HEMOPTYSIS (bloody sputum, spit)
Yes, pneumonia is the most likely, but......
The following is from my medical e-book (I'm a nursing student)

"Blood in the sputum (hemoptysis) is most often seen in clients with chronic bronchitis or lung cancer. Clients with tuberculosis, pulmonary infarction, bronchial adenoma, or lung abscess may have grossly bloody sputum."
Also the end stage of cycstic fibrosis will present with hemoptysis.

a biggie in the hospital is:
PULMONARY EMBOLISM
PATHOPHYSIOLOGY
A pulmonary embolism (PE) is a collection of particulate matter (solids, liquids, or gaseous substances) that enters venous circulation and lodges in the pulmonary vessels. Large emboli obstruct pulmonary blood flow, leading to decreased systemic oxygenation, pulmonary tissue hypoxia, and potential death. Any substance can cause an embolism, but a blood clot is the most common.

Pulmonary embolism is the most common acute pulmonary disease (90%) among hospitalized clients. In most people with PE, a blood clot from a deep vein thrombosis (DVT) breaks loose from one of the veins in the legs or the pelvis. The thrombus breaks off, travels through the vena cava and right side of the heart, and then lodges in a smaller blood vessel in the lung. Platelets collect with the embolus, triggering the release of substances that cause blood vessel constriction. Widespread pulmonary vessel constriction and pulmonary hypertension impair gas exchange. Deoxygenated blood shunts into the arterial circulation, causing hypoxemia. About 12% of clients with PE do not have hypoxemia.

Pulmonary embolism affects at least 500,000 people a year in the United States, about 10% of whom die. Many die within 1 hour of the onset of symptoms or before the diagnosis has even been suspected.

For clients with a known risk for PE, small doses of prophylactic subcutaneous heparin may be prescribed every 8 to 12 hours. Heparin prevents excessive coagulation in clients immobilized for a prolonged period, after trauma or surgery, or when restricted to bedrest. Occasionally, a drug to reduce platelet aggregation, such as clopidogrel (Plavix), is used in place of heparin.



A smaller one that popped up in the book:
GOODPASTURE'S SYNDROME
PATHOPHYSIOLOGY
Goodpasture's syndrome is an autoimmune disorder in which autoantibodies are made against the glomerular basement membrane and neutrophils. The two organs with the most damage are the lungs and the kidney. Lung damage is manifested as pulmonary hemorrhage. Kidney damage manifests as glomerulonephritis that may rapidly progress to complete renal failure (see Chapters 74 and 75). Unlike other autoimmune disorders, Goodpasture's syndrome occurs most often in adolescent or young adult men. The exact cause or triggering agent is unknown.

COLLABORATIVE MANAGEMENT
Goodpasture's syndrome usually is not diagnosed until serious lung and/or kidney problems are present. Manifestations include shortness of breath, hemoptysis (bloody sputum), decreased urine output, weight gain, generalized nondependent edema, hypertension, and tachycardia. Chest x-rays show areas of consolidation. The most common cause of death is uremia as a result of renal failure.

Spontaneous resolution of Goodpasture's syndrome has occurred but is rare. Interventions focus on reducing the immune-mediated damage and performing some type of renal supportive therapy.

  (+ info)

What diseases can you get from cutting yourself with a rusty knife?


This is a question from a growing nurse. I've always wondered if you really can get a disease from a rusty knife or any rust and what kind of diseases you can get.
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staph aureus and staph epidermidis are commonly found on the skin and are responsible for
most infected wounds. methicillin resistant staph aureus (MRSA) is becoming a serious
problem. tetanus (clostridium tetani) is also a possibility but is usually not a problem with superficial
cuts that bleed a lot. infected wounds not treated properly can become gangrenous (clostridium
perfringens). clostridium bacteria are anaerobic which means that require a lack of oxygen to
grow. poor circulation or elevating an infected foot may lead to gangrene due to the lack of oxygen
in the infected area. if a person touches the cut with unclean hands, e. coli could infect the wound.  (+ info)

How accurate is MRI of the brain without contrast for diagnosing inflammatory demyelinating lesions?


Help I have a vague aquaintance with brain problems asking me this.
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Its the best we have & quite sensitive  (+ info)

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