FAQ - pleural effusion, malignant
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what is the pathophysiology of pleural effusion secondary to parapneumonic process?

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anyone here with details regarding Pleural effusion in your fetus or newborn? ?

how did it get diagnosed, in which week, how did it get cured ? in which week?

did u have a fetal surgery? or did it get resolved by itself?

this is quite rare and i am sorry but you may not get many answers  (+ info)

Question about Pleural Effusion?

I had pleural effusion and was stabbed to removed the excess liquid from the lungs. It turned out there were no diseases, making it just an infection only.

But it has been two months and i still have not recovered to the max. I am more tired than before the pleural effusion started, and still, whenever i yawn, i can feel my lungs tightening or some sort of effect i cannot explain, not really painful though. Recovery is progressing so slow, i told my doctors about this, they did not find anything.

So, does recovery take a lot of time after surgery or could it be another problem?

Pleural effusion results from various reasons. You have not mentioned what caused you the pleural effusion.
Even though, pleural tap(drawing the fluid) is effective for some individuals it may not be effective for some. They may eventually develop another effusion after few months. It is based on the primary causes. I think, you need to undergo a thorough investigation if you feel that is recurring.
Your doctor may suggest a CT scan-chest and abdomen to rule out any other underlying causes.
If you develop any pain,fever, tiredness, and breathlessness please do seek medical attention ASAP.
Hope, this helps.  (+ info)

how to properly auscultate the lung with pleural effusion?

how to do it and,where part can you exactly hear the dullness and flatness sound. please use medical terms to describe the location. if u have any other suggestions to make feel free. thanks a lot...(",)

p.s. pleural effusion is on the left lung, secondary to pulmonary tuberculosis

You're med/paramed ? If yes, it should be pretty easy for you.
1. You better check you patient chest by doing percussion first. Patient must be sit, naked (bare skin). Percussion your patient, hear the different between dry part of lung and drown part. Mark them with pen. Pretty easy to differ.

You can also use coin to differ them. Tell someone to put a coin in your patient chest or posterior lung. Click the coin with another coin (make clicking sound). Listen with stet, in the opposit side, clicking sound will gone/decrease in drown lung.
Doing this from apex to basis pulmo, from near back bone or sternal part of lung, to outer part of lung.

2. When you differ them, you simply put your stetoscope and hear : decreased / absent of breathing sound.

Aw, it's pretty hard to explain than to do it myself, ha ha ha. I'm not good tutor. Check this site, maybe can help you better.
(they have picture on it).
Good luck.  (+ info)

why does pleural effusion occur in Congestive Cardiac Failure?

why must the Left lung be affected first (the effusion starts from the left lung) then the Right??

There was a debate on the above and a lot of answers were delivered on the above subject " you are correct".
Some called it BEWARE OF THE LEFT PLEURAL EFFUSION others used a conservative approach.
This will give you an insight view of one of the respondents:
Heart failure (CHF) is the first cause of pleural effusion in the western world. Mrs Dempsey’s pre-test probability of CHF calculated with the Boston criteria is very high. Coronary heart disease is the first cause of CHF in the western world and, despite the absence of ECG findings or chest pain, should not be excluded. The presence of a holosystolic murmur could suggest a mitral rigurgitation or an interventricular septal defect due to a silent ischemic injury, even though the absence of a third heart sound seems unlikely in this clinical presentation. An infective endocarditis is unlikely, because of the normal WBC and the absence of renal failure, spleen enlargment or systemic findings. It is not known, however, if the patient had fever. The rupture of a tendinae chordae would be also possible, but the insidious onset of breathlessness argues against this possibility. A dilatative cardiomyopathy, either primitive, ischemic, valvular or due to hypertension cannot be excluded and the worsening of the mitral regurgitation could have led to this clinical presentation.

The predominance of the effusion on the left lung is strange, because more tipically, when the effusion due to CHF is in both lungs, it is of the same size or bigger on the right side. Constrictive pericarditis (as restrictive cardiomyopathy) could be an interesting hypothesis due to the presence of a raised central venous pressure, a tricuspid murmur and dispnoea on exertion. It’s not known if she had a history of TBC exposure, even though there’s not anemia, lymphocytosis, cough, weight loss or systemic illness. A neoplastic constrictive pericarditis in another interesting option and pleural effusion could be a consequence of this as well as a concomitant process. The primitive and secondary lung cancer rarely cause such a cardiac failure unless there are cardiac metastases or there’s an involvment of pulmonary arteries. I would consider in particular lymphoma, lung cancer (even though there’s not smoking history), breast, ovarian, renal cancer and melanoma. Nonetheless weight loss, anorexia or other systemic symptoms are not known and the plasma viscosity and WBC are normal. However, a cardio-pulmonary neoplastic syndrome should not be excluded and the greatest effusion on the left could also suggest a retraction of the lung induced by cancer. The association between hypertension and constrictive pericarditis has been moreover described. Some connective tissue disorders as some vasculitis can present with a pleuro-pericardial involvment, but the apparent lack of systemic finding, the normal WBC and plasma viscosity make this diagnoses unlikely. Furthermore these elements, as the absence of the cough, argue against a parapneumonic effusion. Pulmonary embolism looks like an attractive hypothesis, mainly multiple micro-embolization altough there are any risk factor known for deep venous thrombosis. The bilateral ankle oedema could suggest thrombosis of the inferior vena cava. Systemic hypertension and orthopnoea lower the probability of pulmonary embolism as well as the absence of chest pain or cough, but do not rule out it.

In conclusion, CHF remains the leading hypothesis and the cause of this syndrome needs to be rapidly discovered. Even constrictive pericarditis, particulary neoplastic, and pulmonary embolism should be carefully considered. With regard to the concomitant hypertension, I would keep in mind other less probable etiologies, such as renal artery stenosis, a carcinoid syndrome or an atrial septal defect.

I would continue the diuretic therapy, increasing the dose (intravenous) and monitoring the patient for two-three days. I think that we still don’t need to make a thoracentesis, unless the patient develops rest dispnoea. I would not give her an ACE inhibitor before a renal artery stenosis has been excluded. I would make thoracentesis if there has been any improvement after two days.

I would ask echocardiography at this instant and the next steps are largely dependent on his results.

I would dose glicemia, lipids, amilase, lipase, DE-dimers, serum globulins level and ask for an abdominal-pelvic ecography. I would ask more about her past history and make an accurate objective examination.

If these investigations would not be available, the dosage of BNP could be an idea, but the pre-test probability of CHF is very high and only a very low level of BNP could significantly lower this value. I would esplain to Mrs Dempsey that her dispnoea is due to an overload of fluids in her lungs an that this condition can be due to many causes, most of which are resolvable. I would also tell her that in the next days it could be necessary to make a thoracentesis in order to discover the cause of her illness and give her relief.  (+ info)

bilateral pneumonia complicated by pleural effusion: fatality rate?

What is the fatality rate for someone wheelchair bound (currently in the ICU) and with a weakened immune system?

I'm afraid the news isn't good. There's nothing you mentioned that can't be treated but taken together and the fact they are in ICU now means that there is a danger of sepsis (blood infection). The sequelae to that is ARDS and multiple system failures. I'm not saying lose all hope but the stats are against it. I've seen people survive worse situations but it's an uphill battle.
God bless you and the person you're talking about. I'll pray for you.  (+ info)

"Mild light Pleural Effusion/thickening with right apical pleural thickenining and fribrotic scarring.?

What is this suppose to mean.. this was a report for a chest x-ray...

"Mild light Pleural Effusion/thickening with right apical pleural thickenining and fribrotic scarring. Sequelae to old kochs etiology"

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is massive pleural effusion in one lung is high risk for mastectomy for breast cancer?

Pleural effusion will decrease the area of respiratory exchange, increasing the chance of having complication with general anasthesia. However, I would want to ask one question.

Why doesn't the doctor tap the effusion out and make sure the patient is well prepared before proceed to mastectomy?

Also, I bet the anasthesiologist will have fully evaluate the patient before the surgery, so if he says it is okay, probably he is right.

Hopefully, that helps. Good luck  (+ info)

Difference between Pleural effusion and thoracentesis?

What is the Difference between Pleural effusion and thoracentesis???

Pleural effusion is defined as an abnormal accumulation of fluid in the pleural space. Excess fluid results from the disruption of the equilibrium that exists across pleural membranes.

Thoracentesis is chest wall puncture for aspiration of pleural fluid. It is used to determine the etiology of a pleural effusion (diagnostic thoracentesis), and to relieve dyspnea caused by pleural fluid (therapeutic thoracentesis).  (+ info)

pleural effusion. How to make it get better and disappear?Would breathing exercises or heat help?

Back is sore on the side above the waist.

actually, yes breathing excercises can help!! True most go away on their own. my daughter at age six had a pleural effusion. considering how small it was we did nothing for it. some people how ever need to have a thoracentesis or in laymans terms, have it tapped and drained. A good breathing excercise that helps push fluid from the lung called ez-pap. its a device that causes the person to exhale towards a positive pressure. this is very well known in the respiratory world. although it is not done at home, mostly in acute care facilities

ps...dont decrease your fluid intake...  (+ info)

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