Does anyone know what pulmonary infiltrates with eosinophilia is?
My 2 month old baby died 3 months ago. I got the autopsy report back and this is what they said she died of. She had a history of RSV, and they said that this could have caused it. I don't think that the doctor's did everything that they should have. She stopped breathing once when she had it, but we brought her back. She spent 6 days in the hospital and they sent her home. Well, it seemed like she stayed after that. I kept taking her to the doctor, and all they would say is that she just had a cold. They never would check anything on her. Any help you can give me would be great. Thanks
I am so, so, so sorry for your loss. I am not a doctor, just a Mom like you. My little girl has an interstitial lung disease of unknown origin (possibly post viral although no virus was identified) and suffered respiratory failure as a 2 week old baby. Thankfully, we have wonderful physicians who managed to save her when even they thought it was not likely. She is not perfect and is on oxygen at almost 10 months old, but she is still alive. There is absolutely no excuse for them not checking your daughter when you took her to them for help, and you definitely should see a lawyer. As far as what your baby may have had, is it possible for you to sit down and talk to a professional about what the results mean? If you can find one willing to talk to you, a pediatric pulmonologist would be an excellent resource and may help you find some answers. Also, I came across this article on eosinophilic pneumonia (the first link below) months ago. For some reason, it really caught my attention when I first saw it, and when I saw your question, I thought of it immediately. As I said, I'm not a doctor, so I may be way off, but to me, some of it seems to fit. The second link below is a link to the description of interstitial lung disease in children. Did any of those symptoms sound like your daughter after the RSV when she had the "cold?" Our kids are all too often dismissed as "just a cold" or "just asthma." My own daughter was "failure to thrive" until thankfully we found the right doctors. Again, I am so sorry. (+ info
what is the exact mechanism of tropical pulmonary eosinophilia?
what is the mechanism and causes of TPE illustrating with diagrams?
Some individuals with fertilized adult female worms produce allergic reactions directed against microfilaria, thereby clearing the released mf, but producing the clinical syndrome of Tropical Pulmonary Eosinophilia (TPE) without microfilariaemia (occult filariasis).
This is usually detected in areas endemic for bancroftian filariasis. Males are more commonly affected.
The commonest presentation is paroxysmal nocturnal cough with hyper eosinophilia, raised ESR, radiological findings of diffuse milary lesions or increased broncho-vascular markings with very high titres of antifilaial antibodies particularly against mf.
There in an impaired lung function in most cases. If untreated, TPE may progress to a condition of chronic pulmonary fibrosis.
So, basically, it's caused by an allergic reaction to microfilaria in the lungs. (+ info
Please i want to know role of antigen detection for diagnosis of tropical pulmonary eosinophilia?
Please i want to know role of antigen detection for diagnosis of tropical pulmonary eosinophilia in details
The disease is due to hypersensitivity to certain filarial parasites in the tropics specially South East Asia and China
Microfilariae are not found in the blood of these patients.0
But their blood will have high titres of antifilarial antibodies.
This test called FAT if found positive in a patient having the typical symptoms of Tropical pulmonary Eosinophilia then diagnosis is confirmed (+ info
What toxicant causes pulmonary eosinophilia?
differential study between simple and tropical pulmonary eosinophilia?
I hope these help!!! (+ info
tropical pulmonary eosinophilia causes and differential diagnosis?
causes and differential diagnosis of TPE IN DETAILS
tropical pulmonary eosinophilia causes mechanism diagnosis differential diagnosis and management in details and images
Tropical pulmonary eosinophilia (TPE) was first described in 1939 in a group of seven Javanese men. Over the next few years, other cases were reported, with lymph node biopsies showing eosinophilic granulomata surrounding microfilariae.
All the detailed information you seek is available at the website below. (+ info
what are the difference between sinusitis,eosinophilia and bronchitis?
what are the difference and symptoms between sinusitis,eosinophilia and bronchitis?is three are connected?
Sinusitis is in the sinuses..nasal area....Bronchitis ..is in the bronchia...lungs...not sure about the eosinophilla..hope this helps. (+ info
What effects could pneumonia and pulmonary congestion have on gas exchange?
Both pneumonia (infectious inflammation of the lungs) and pulmonary congestion (left heart failure) can lead to fluid accumulation in the lungs.
Pneumonia and pulmonary congestion cause a fundamental change in the way gas is exchanged within the lungs. Gas is exchanged through structures called alveoli. They are microscopic sacs of epithelial tissue. When these sacs become filled with fluid (referred to as consildation) gas cannot pass from the alveoli into the blood. Thus, gas exchange is impeded and the patient typically must work harder to breathe. (+ info
What is involved in a typical pulmonary function test?
I have to get a typical pulmonary function test tomorrow. What exactly does this entail? I looked online and didn't find anything that really explained it. I'm 15, if age makes any difference. Thanks!
In a spirometry test, you breathe into a mouthpiece that is connected to an instrument called a spirometer. The spirometer records the amount and the rate of air that you breathe in and out over a period of time. (+ info
How does pregnancy increase the risk of pulmonary and other emboli?
How is pregnancy associated with an increased risk in pulmonary embolism? Does this explain the association between long term use of oral contraceptives and thrombosis, which could possibly lead to embolism?
Yes and no. The hormones that are increased in pregnancy and contained in the contraceptive Pill aren't the only risk factors for blot clots forming during pregnancy. What the Pill and pregnancy both have in common is that they both produce hormones that increase the bloods clotting ability. Oestrogen increases the risk of DVTs by increasing the production of certain chemicals necessary for the blood to clot. It also increases platelet numbers + stickiness of platelets, which increases clot formation (to prepare for blood loss during child birth). Progestogen in the Pill causes the blood vessels to relax and widen, allowing the blood to pool in the veins, increasing risk of clot formation.
Blood clots in pregnancy are generally the result of increased venous stasis- blood pooling in the lower deep veins. As I said above, balance of proteins of the coagulation + fibrinolytic systems also play a part. DVT can of course lead to a pulmonary embolism. Clots in pregnancy and from the Pill are generally always the results of clots that have formed in the deep veins of the legs, broken away and then embolised in the lungs.
Women who do have clots in pregnancy and whilst taking the contraceptive Pill, generally also have certain other risk factors such as protein C and S deficiency and most commonly Factor V Leiden mutation. Over all health of the woman also plays a part- obesity, poor diet/unhealthy lifestyle and a smoking history also increases the risk as does age.
During pregnancy the uterus also expands, putting pressure on the lower blood vessels which can restrict blood flow from the legs and pelvis back to the heart. Physiological structural changes during pregnancy are risk factors alone. (+ info
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