A hereditary disease characterized by childhood onset hypertension, hypokalemic alkalosis, and low renin and aldosterone secretion. It results from a defect in the activity of the 11-beta-hydroxysteroid dehydrogenase type 2 enzyme which results in inadequate conversion of CORTISOL to cortisone. The build up of unprocessed cortisol to levels that stimulate MINERALOCORTICOID RECEPTORS creates the appearance of having excessive mineralocorticoids.

Leave a message about 'Mineralocorticoid Excess Syndrome, Apparent'

We do not evaluate or guarantee the accuracy of any content in this site. Click here for the full disclaimer.