1/7. Salivary gland cysts of the oral cavity: clinical observation and surgical management.Mucoceles are common cystic lesions of the oral mucosa. Extravasation mucoceles are mainly found in the lower lip of young patients, whereas retention mucoceles are usually located in the cheek or palate of older patients. The disparate site and age incidences of extravasation and retention mucoceles suggest that these two types are not related and have different pathogenesis. Treatment modalities for mucoceles are reviewed, with special attention to the cryosurgical technique for the treatment of oral ranulas.- - - - - - - - - - ranking = 1keywords = observation (Clic here for more details about this article) |
2/7. Pediatric recurrent sialectasis: case report.A 2 1/2-year-old female presented to the Department of Paediatrics with a fever (38.2 degrees C) and bilateral swelling of the parotid salivary glands. A month later, she presented with similar signs and symptoms. Three months later the patient had a third attack and was referred to the Paediatric Dental Department. A clinical examination and sialography showed a normal Stensen's duct. The parenchyma of the right parotid had a combination of punctate lesions (1-2 mm in diameter) and globular lesions (3 mm in diameter). In the left parotid there were punctate lesions approximately 1 mm in diameter. The patient was managed with antipyretics (acetaminophen) and penicillin. This condition should be referred to as sialadenitis pediatrica as it presents as a clinical entity with signs and symptoms of swelling and pyrexia in the absence of sialography and histological findings. It is referred to as sialectasis after histological and sialographic observations detect lesions affecting the parenchyma of the salivary glands.- - - - - - - - - - ranking = 0.25keywords = observation (Clic here for more details about this article) |
3/7. Necrotizing sialometaplasia: a sheep in wolf's clothing. Healing as a diagnostic test.Necrotizing sialometaplasia is a benign self-limited intraoral lesion that is easily confused both clinically and histologically with squamous cell carcinoma. It presents as a painless ulceration, frequently on the hard palate, that histologically shows necrosis, inflammation, squamous metaplasia, and granulation tissue. It is thought to be due to infarction of minor salivary glands and heals spontaneously in six to 12 weeks. A brief period of observation for evidence of healing can be an important diagnostic clue in distinguishing this entity from cancer, thus saving the patient unnecessary surgery or radiation therapy.- - - - - - - - - - ranking = 0.25keywords = observation (Clic here for more details about this article) |
4/7. Necrotizing sialometaplasia of the nasopharynx.Necrotizing sialometaplasia is an uncommon salivary gland disease originally described by Abrams et al in 1973. The disease may occur wherever salivary gland tissue is found. Theories on the etiology of this disorder have been advanced, but never definitely determined. Treatment consists of adequate biopsy and observation until healing occurs at six to eight weeks. While the disease is considered benign, its similarity to more aggressive neoplasms can be both disturbing and misleading to the patient and the clinician. Such a case of necrotizing sialometaplasia of the nasopharynx is presented to demonstrate the clinical and histological similarity of this disease to carcinoma. In this case, the patient first presented with a neck mass which could easily have been mistaken for a regional metastasis. The current literature is reviewed.- - - - - - - - - - ranking = 0.25keywords = observation (Clic here for more details about this article) |
5/7. Chronic sialadenitis of the submandibular gland (so-called Kuttner tumor).Two cases of chronic sialadenitis of the submandibular gland (so-called Kuttner tumor) are reported. Clinically, they are asymptomatic except for firm swellings which are similar to neoplasms. light microscopic observations revealed that the lesion is characterized by various degrees of atrophy and/or destruction of acini, infiltration of inflammatory cells, periductal fibrosis and lymph follicle formation. With the electron microscope, a marked increase of collagen fibers are observed throughout the gland parenchyma, and the secretory granules of acinal cells in the remaining acini remarkably decreased in number. We suggest that an autoimmune mechanism may play a role in case 1 and that an ascending infection through the duct system may be a main factor in case 2.- - - - - - - - - - ranking = 0.25keywords = observation (Clic here for more details about this article) |
6/7. Anterior salivary gland inclusion in the mandible: pathological entity or anatomical variant?A case of anterior salivary gland inclusion of the mandible is reported and the literature reviewed. It is suggested that these inclusions arise in the same way as the more common examples from the angle of the mandible, by progressive resorption of the lingual cortex in response to pressure exerted by the adjacent salivary glands. It is likely that these lesions become evident on radiographs only when the resorption is marked, and they may, therefore, be more widespread than reported figures indicate. Supportive evidence for this hypothesis is provided by the observations that the lesions usually present in middle age and, on histological examination, exhibit evidence of resorption.- - - - - - - - - - ranking = 0.25keywords = observation (Clic here for more details about this article) |
7/7. Salivary gland disorders and heredity.From personal observations, I review the genetic disorders of salivary gland development and function, including the lacrimo-auriculodentodigital (LADD) syndrome, autosomal dominant hypoplasia/agenesis of salivary and/or lacrimal glands, chronic recurrent sialadenitis, polycystic-dysgenetic disease of the parotids and salivary calculi.- - - - - - - - - - ranking = 0.25keywords = observation (Clic here for more details about this article) |